Karen R. Pratt

1.6k citations
28 papers · 1.4k indexed · h-index 19

Karen R. Pratt

28 papers receiving 1.2k citations

Peers

Karen R. Pratt
Comparison fields: 5 of 87
  • Immunology 654
  • Virology 51
  • Microbiology 65
  • Behavioral Neuroscience 37
  • Radiology, Nuclear Medicine and Imaging 218
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G J Stanton United States
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Citations per year

Countries citing papers authored by Karen R. Pratt

Since Specialization
Citations

This map shows the geographic impact of Karen R. Pratt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Karen R. Pratt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Karen R. Pratt more than expected).

Fields of papers citing papers by Karen R. Pratt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Karen R. Pratt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Karen R. Pratt. The network helps show where Karen R. Pratt may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Karen R. Pratt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Karen R. Pratt Line = papers co-authored together Karen R. Pratt links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 200041
2 19941
3 199318
4 199018
5 199047
6 199035
7 198846
8 198549
9 198310
10 19831
11 198251
12 198122
13 1978153
14
Human bone marrow lymphocytes. III. Polyclonal activation of B lymphocytes in human bone marrow measured by a direct plaque-forming cell assay.
197716
15 197749
16
Activation of human B lymphocytes. IV. Regulatory effects of corticosteroids on the triggering signal in the plaque-forming cell response of human peripheral blood B lymphocytes to polyclonal activation.
197798
17 1976167
18 197684
19 197626
20 197658

About Karen R. Pratt

Karen R. Pratt is a scholar working on Immunology, Virology, Toxicology, Molecular Biology and Cancer Research, having authored 28 papers that have together received 1.4k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (7 papers), T-cell and B-cell Immunology (6 papers), Epigenetics and DNA Methylation (5 papers), Immune Response and Inflammation (4 papers), Glycosylation and Glycoproteins Research (3 papers), Molecular Biology Techniques and Applications (3 papers), RNA modifications and cancer (3 papers) and Animal Genetics and Reproduction (3 papers). The work is most often cited by research in Immunology (654 citations), Virology (51 citations), Microbiology (65 citations), Behavioral Neuroscience (37 citations) and Radiology, Nuclear Medicine and Imaging (218 citations). Karen R. Pratt has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include Anthony S. Fauci, Gail Whalen, Stanley Hattman, Sharon Germana, David H. Sachs, Kenth Gustafsson, François Hirsch, Claire Kenny, Leszek Berger and Christian LeGuern. Their work appears in journals such as The Journal of Immunology, Immunogenetics, Journal of Bacteriology, Nucleic Acids Research and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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