Kanehiro Hayashi

1.8k total citations
31 papers, 1.4k citations indexed

About

Kanehiro Hayashi is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, Kanehiro Hayashi has authored 31 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cellular and Molecular Neuroscience, 13 papers in Molecular Biology and 13 papers in Developmental Neuroscience. Recurrent topics in Kanehiro Hayashi's work include Neurogenesis and neuroplasticity mechanisms (13 papers), Neuroscience and Neuropharmacology Research (12 papers) and Genetics and Neurodevelopmental Disorders (5 papers). Kanehiro Hayashi is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (13 papers), Neuroscience and Neuropharmacology Research (12 papers) and Genetics and Neurodevelopmental Disorders (5 papers). Kanehiro Hayashi collaborates with scholars based in Japan, United States and France. Kanehiro Hayashi's co-authors include James Bibb, Kazunori Nakajima, Toshio Matsushima, Satoshi Suzuki, Katsunori Fujii, Takuya Inoue, Janice W. Kansy, Ken‐ichiro Kubo, Paul Greengard and David R. Benavides and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Kanehiro Hayashi

31 papers receiving 1.4k citations

Peers

Kanehiro Hayashi
Kanehiro Hayashi
Citations per year, relative to Kanehiro Hayashi Kanehiro Hayashi (= 1×) peers Ikuko Mizuta

Countries citing papers authored by Kanehiro Hayashi

Since Specialization
Citations

This map shows the geographic impact of Kanehiro Hayashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kanehiro Hayashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kanehiro Hayashi more than expected).

Fields of papers citing papers by Kanehiro Hayashi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kanehiro Hayashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kanehiro Hayashi. The network helps show where Kanehiro Hayashi may publish in the future.

Co-authorship network of co-authors of Kanehiro Hayashi

This figure shows the co-authorship network connecting the top 25 collaborators of Kanehiro Hayashi. A scholar is included among the top collaborators of Kanehiro Hayashi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kanehiro Hayashi. Kanehiro Hayashi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tabata, Hidenori, Megumi Sasaki, Masakazu Agetsuma, et al.. (2022). Erratic and blood vessel-guided migration of astrocyte progenitors in the cerebral cortex. Nature Communications. 13(1). 6571–6571. 14 indexed citations
2.
Hayashi, Kanehiro, Yusuke Seto, Mototsugu Eiraku, et al.. (2022). Development of intensiometric indicators for visualizing N-cadherin interaction across cells. Communications Biology. 5(1). 1065–1065. 4 indexed citations
3.
Kitazawa, Ayako, Satoshi Yoshinaga, Kanehiro Hayashi, et al.. (2019). Both excitatory and inhibitory neurons transiently form clusters at the outermost region of the developing mammalian cerebral neocortex. The Journal of Comparative Neurology. 527(10). 1577–1597. 7 indexed citations
4.
Hayashi, Kanehiro, Kyota Fujita, Kazuhiko Tagawa, et al.. (2018). Drebrin-like (Dbnl) Controls Neuronal Migration via Regulating N-Cadherin Expression in the Developing Cerebral Cortex. Journal of Neuroscience. 39(4). 678–691. 18 indexed citations
5.
Okuno, Hironobu, Shigeki Ohta, Kimiko Fukuda, et al.. (2017). CHARGE syndrome modeling using patient-iPSCs reveals defective migration of neural crest cells harboring CHD7 mutations. eLife. 6. 44 indexed citations
6.
Ito, Daisuke, Itaru Arai, Yuki Kobayashi, et al.. (2017). Dendritic Homeostasis Disruption in a Novel Frontotemporal Dementia Mouse Model Expressing Cytoplasmic Fused in Sarcoma. EBioMedicine. 24. 102–115. 17 indexed citations
7.
Hayashi, Kanehiro, Asako Furuya, Yuriko Sakamaki, et al.. (2017). The brain-specific RasGEF very-KIND is required for normal dendritic growth in cerebellar granule cells and proper motor coordination. PLoS ONE. 12(3). e0173175–e0173175. 9 indexed citations
8.
Hara, Yoshinobu, Masahiro Fukaya, Kanehiro Hayashi, et al.. (2016). ADP Ribosylation Factor 6 Regulates Neuronal Migration in the Developing Cerebral Cortex through FIP3/Arfophilin-1-dependent Endosomal Trafficking of N-cadherin. eNeuro. 3(4). ENEURO.0148–16.2016. 35 indexed citations
9.
Hayashi, Kanehiro, Ken‐ichiro Kubo, Ayako Kitazawa, & Kazunori Nakajima. (2015). Cellular dynamics of neuronal migration in the hippocampus. Frontiers in Neuroscience. 9. 135–135. 54 indexed citations
10.
Plattner, Florian, Kanehiro Hayashi, Adán Hernández, et al.. (2015). The role of ventral striatal cAMP signaling in stress-induced behaviors. Nature Neuroscience. 18(8). 1094–1100. 72 indexed citations
11.
Hayashi, Kanehiro, Jumpei Sasabe, Tomohiro Chiba, Sadakazu Aiso, & Naoko Utsunomiya‐Tate. (2011). d-Ser-containing humanin shows promotion of fibril formation. Amino Acids. 42(6). 2293–2297. 6 indexed citations
13.
Yoshikawa, Fumio, Yoshiko Banno, Yoshihide Yamaguchi, et al.. (2010). Phospholipase D Family Member 4, a Transmembrane Glycoprotein with No Phospholipase D Activity, Expression in Spleen and Early Postnatal Microglia. PLoS ONE. 5(11). e13932–e13932. 46 indexed citations
14.
Drerup, Justin, Kanehiro Hayashi, Huxing Cui, et al.. (2010). Attention-Deficit/Hyperactivity Phenotype in Mice Lacking the Cyclin-Dependent Kinase 5 Cofactor p35. Biological Psychiatry. 68(12). 1163–1171. 48 indexed citations
15.
Furutama, Daisuke, Noriyuki Morita, Riya Takano, et al.. (2010). Expression of the IP3R1 promoter‐driven nls‐lacZ transgene in Purkinje cell parasagittal arrays of developing mouse cerebellum. Journal of Neuroscience Research. 88(13). 2810–2825. 23 indexed citations
16.
Hawasli, Ammar H., Della Koovakkattu, Kanehiro Hayashi, et al.. (2009). Regulation of Hippocampal and Behavioral Excitability by Cyclin-Dependent Kinase 5. PLoS ONE. 4(6). e5808–e5808. 26 indexed citations
17.
Nguyen, Chan, Akinori Nishi, Janice W. Kansy, et al.. (2007). Regulation of Protein Phosphatase Inhibitor-1 by Cyclin-dependent Kinase 5. Journal of Biological Chemistry. 282(22). 16511–16520. 22 indexed citations
18.
Hawasli, Ammar H., David R. Benavides, Chan Nguyen, et al.. (2007). Cyclin-dependent kinase 5 governs learning and synaptic plasticity via control of NMDAR degradation. Nature Neuroscience. 10(7). 880–886. 250 indexed citations
19.
Hayashi, Kanehiro, Yong Pan, Hongjun Shu, et al.. (2006). Phosphorylation of the tubulin‐binding protein, stathmin, by Cdk5 and MAP kinases in the brain. Journal of Neurochemistry. 99(1). 237–250. 46 indexed citations
20.
Ohshima, Toshio, Hiroo Ogura, Kazuhito Tomizawa, et al.. (2005). Impairment of hippocampal long‐term depression and defective spatial learning and memory in p35–/– mice. Journal of Neurochemistry. 94(4). 917–925. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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