Kamil S. Sitarz

641 citations
9 papers · 225 indexed · h-index 7
Topics
Mitochondrial Function and Pathology (5 papers)ATP Synthase and ATPases Research (4 papers)Metabolism and Genetic Disorders (4 papers)

In The Last Decade

Kamil S. Sitarz

9 papers receiving 221 citations

Peers

Kamil S. Sitarz
Comparison fields: 5 of 54
  • Molecular Biology 176
  • Clinical Biochemistry 79
  • Cellular and Molecular Neuroscience 26
  • Genetics 18
  • Psychiatry and Mental health 16
Replace Arturo Solís Herrera with:
Arturo Solís Herrera United States
Asif Paker United States
Jane Hübertz Frederiksen Denmark
Simonetta Sangiorgi Italy
Karine Mention-Mulliez France
S. Romano France
Hitoshi Matsuo Japan
Daniela Buhaş Canada
Yair Anikster Israel
Maura Bailie United Kingdom
Kamil S. Sitarz relative to Arturo Solís Herrera United States Arturo Solís Herrera's profile →
Citations per field
00.5×4.5×
Arturo Solís Herrera · 1×
Citations per year

Countries citing papers authored by Kamil S. Sitarz

Since Specialization
Citations

This map shows the geographic impact of Kamil S. Sitarz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kamil S. Sitarz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kamil S. Sitarz more than expected).

Fields of papers citing papers by Kamil S. Sitarz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kamil S. Sitarz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kamil S. Sitarz. The network helps show where Kamil S. Sitarz may publish in the future.

Co-authorship network of co-authors of Kamil S. Sitarz

This figure shows the co-authorship network connecting the top 25 collaborators of Kamil S. Sitarz. A scholar is included among the top collaborators of Kamil S. Sitarz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kamil S. Sitarz. Kamil S. Sitarz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
#WorkIndexed citations
1 15
2 2
3 33
4 41
5 22
6 11
7 1
8 26
9 74

About Kamil S. Sitarz

Kamil S. Sitarz is a scholar working on Clinical Biochemistry, Cancer Research and Molecular Biology, having authored 9 papers that have together received 225 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (5 papers), ATP Synthase and ATPases Research (4 papers) and Metabolism and Genetic Disorders (4 papers). The work is most often cited by research in Clinical Biochemistry (79 citations), Molecular Biology (176 citations) and Neurology (13 citations). Kamil S. Sitarz has collaborated with scholars based in United Kingdom, Germany and United States. Frequent co-authors include Patrick F. Chinnery, Rita Horváth, Patrick Yu‐Wai‐Man, David C. Samuels, Philip G. Griffiths, Amy K. Reeve, Laurence A. Bindoff, Hannah R. Elliott, Caroline L. Relton and Robert W. Taylor. Their work appears in journals such as Neurology, Cancer Research and Human Molecular Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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