Kali Janes

1.2k total citations
18 papers, 958 citations indexed

About

Kali Janes is a scholar working on Physiology, Molecular Biology and Physiology. According to data from OpenAlex, Kali Janes has authored 18 papers receiving a total of 958 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 11 papers in Molecular Biology and 11 papers in Physiology. Recurrent topics in Kali Janes's work include Pain Mechanisms and Treatments (15 papers), Adenosine and Purinergic Signaling (11 papers) and Pharmacological Receptor Mechanisms and Effects (8 papers). Kali Janes is often cited by papers focused on Pain Mechanisms and Treatments (15 papers), Adenosine and Purinergic Signaling (11 papers) and Pharmacological Receptor Mechanisms and Effects (8 papers). Kali Janes collaborates with scholars based in United States, Italy and Russia. Kali Janes's co-authors include Daniela Salvemini, Timothy M. Doyle, Kenneth A. Jacobson, Joshua W. Little, Salvatore Cuzzocrea, Zhoumou Chen, Emanuela Esposito, Leesa Bryant, William L. Neumann and Daniela Salvemini and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Experimental Medicine and Journal of Neuroscience.

In The Last Decade

Kali Janes

18 papers receiving 952 citations

Peers

Kali Janes
Sooyeon Jo United States
Zhongfang Weng United States
Richard P. Hulse United Kingdom
Deborah Holstein United States
Svetlana Drndarski United Kingdom
Kali Janes
Citations per year, relative to Kali Janes Kali Janes (= 1×) peers Xueming Hu

Countries citing papers authored by Kali Janes

Since Specialization
Citations

This map shows the geographic impact of Kali Janes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kali Janes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kali Janes more than expected).

Fields of papers citing papers by Kali Janes

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kali Janes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kali Janes. The network helps show where Kali Janes may publish in the future.

Co-authorship network of co-authors of Kali Janes

This figure shows the co-authorship network connecting the top 25 collaborators of Kali Janes. A scholar is included among the top collaborators of Kali Janes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kali Janes. Kali Janes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Doyle, Timothy M., Kali Janes, Wen Xiao, et al.. (2024). Mitochondrial A3Adenosine Receptor as a Mechanism for the Protective Effects of A3AR Agonists on Chemotherapy-Induced Neuropathic Pain. Journal of Neuroscience. 45(3). e1268242024–e1268242024. 3 indexed citations
2.
Doyle, Timothy M., Mark R. Hutchinson, Kali Janes, et al.. (2020). Sphingosine-1-phosphate receptor subtype 1 activation in the central nervous system contributes to morphine withdrawal in rodents. Journal of Neuroinflammation. 17(1). 314–314. 8 indexed citations
4.
Doyle, Timothy M., Joshua W. Little, Livio Luongo, et al.. (2018). Chemotherapy-induced pain is promoted by enhanced spinal adenosine kinase levels via astrocyte-dependent mechanisms. Journal of Pain. 19(3). S74–S74. 4 indexed citations
5.
Doyle, Timothy M., Xisheng Yan, Zhoumou Chen, et al.. (2018). Dysregulation of sphingolipid metabolism contributes to bortezomib-induced neuropathic pain. The Journal of Experimental Medicine. 215(5). 1301–1313. 105 indexed citations
6.
Doyle, Timothy M., Joshua W. Little, Livio Luongo, et al.. (2018). Chemotherapy-induced pain is promoted by enhanced spinal adenosine kinase levels through astrocyte-dependent mechanisms. Pain. 159(6). 1025–1034. 76 indexed citations
7.
Janes, Kali, et al.. (2016). Identification of A3adenosine receptor agonists as novel non‐narcotic analgesics. British Journal of Pharmacology. 173(8). 1253–1267. 50 indexed citations
8.
9.
Janes, Kali, Emanuela Esposito, Timothy M. Doyle, et al.. (2014). A3 adenosine receptor agonist prevents the development of paclitaxel-induced neuropathic pain by modulating spinal glial-restricted redox-dependent signaling pathways. Pain. 155(12). 2560–2567. 90 indexed citations
10.
Little, Joshua W., Amanda Ford, Ashley M. Symons-Liguori, et al.. (2014). Endogenous adenosine A3 receptor activation selectively alleviates persistent pain states. Brain. 138(1). 28–35. 120 indexed citations
11.
Little, Joshua W., Amanda Ford, Kali Janes, et al.. (2014). (297) Central adenosine A3 receptor (A3AR) activation reverses neuropathic pain. Journal of Pain. 15(4). S50–S50. 3 indexed citations
13.
Janes, Kali, Joshua W. Little, Chao Li, et al.. (2014). The Development and Maintenance of Paclitaxel-induced Neuropathic Pain Require Activation of the Sphingosine 1-Phosphate Receptor Subtype 1. Journal of Biological Chemistry. 289(30). 21082–21097. 123 indexed citations
16.
Salvemini, Daniela, Kali Janes, Timothy M. Doyle, et al.. (2013). Adenosine receptor subtype 3 (A3AR) agonists as novel analgesics in chronic neuropathic pain. The FASEB Journal. 27(S1). 2 indexed citations
17.
Chen, Zhoumou, Kali Janes, Tim Doyle, et al.. (2012). Controlling murine and rat chronic pain through A 3 adenosine receptor activation. The FASEB Journal. 26(5). 1855–1865. 91 indexed citations
18.
Janes, Kali, William L. Neumann, & Daniela Salvemini. (2011). Anti-superoxide and anti-peroxynitrite strategies in pain suppression. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1822(5). 815–821. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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