Kaimi Li
Impact in
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- Cancer-related molecular mechanisms research
- Cancer, Hypoxia, and Metabolism
- Cancer, Lipids, and Metabolism
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- Cancer Immunotherapy and Biomarkers
Papers in
- Oncology 11
- Cancer Cells and Metastasis 3
- Cancer-related Molecular Pathways 3
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- Cancer, Hypoxia, and Metabolism 3
- Cancer Genomics and Diagnostics 2
- Journals
- Frontiers in Oncology (2 papers)Neoplasia (1 paper)Modern Pathology (1 paper)Cell Death and Disease (1 paper)Biomedicine & Pharmacotherapy (1 paper)
- Partner nations
- ChinaThailandUnited States
In The Last Decade
Kaimi Li
15 papers receiving 393 citations
Peers
Comparison fields: 5 of 57
- Cancer Research 132
- Oncology 138
- Immunology 79
- Pulmonary and Respiratory Medicine 102
- Molecular Biology 211
Countries citing papers authored by Kaimi Li
This map shows the geographic impact of Kaimi Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kaimi Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kaimi Li more than expected).
Fields of papers citing papers by Kaimi Li
This network shows the impact of papers produced by Kaimi Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kaimi Li. The network helps show where Kaimi Li may publish in the future.
Co-authors
The 25 scholars most cited alongside Kaimi Li, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 2 | |
| 2 | 2023 | 3 | |
| 3 | 2023 | 12 | |
| 4 | 2021 | 11 | |
| 5 | 2021 | 9 | |
| 6 | 2020 | 56 | |
| 7 | 2020 | 13 | |
| 8 | 2019 | 77 | |
| 9 | 2019 | 46 | |
| 10 | 2019 | 55 | |
| 11 | 2019 | 12 | |
| 12 | 2019 | 20 | |
| 13 | Dysregulated ENPP1 increases the malignancy of human lung cancer by inducing epithelial-mesenchymal transition phenotypes and stem cell features. | 2019 | 32 |
| 14 | Stabilization of PTGES by deubiquitinase USP9X promotes metastatic features of lung cancer via PGE2 signaling. | 2019 | 28 |
| 15 | 2018 | 21 |
About Kaimi Li
Kaimi Li is a scholar working on Oncology, Cancer Research, Immunology, Molecular Biology and Pathology and Forensic Medicine, having authored 15 papers that have together received 397 indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (3 papers), Cancer Cells and Metastasis (3 papers), RNA modifications and cancer (3 papers), Cancer-related Molecular Pathways (3 papers), Epigenetics and DNA Methylation (2 papers), Genetic factors in colorectal cancer (2 papers), Immune cells in cancer (2 papers) and Cancer Genomics and Diagnostics (2 papers). The work is most often cited by research in Cancer Research (132 citations), Oncology (138 citations), Immunology (79 citations), Pulmonary and Respiratory Medicine (102 citations) and Molecular Biology (211 citations). Kaimi Li has collaborated with scholars based in China, Thailand and United States. Frequent co-authors include Wenzheng Guo, Hongyong Song, Bo Jing, Yueling Liao, Tong Wang, Yanbin Kuang, Ling Jing, Jiong Deng, Beibei Sun and Feng Yao. Their work appears in journals such as Frontiers in Oncology, Neoplasia, Modern Pathology, Cell Death and Disease and Biomedicine & Pharmacotherapy.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.