Kai M. Schmidt‐Ott

9.6k total citations · 3 hit papers
106 papers, 5.8k citations indexed

About

Kai M. Schmidt‐Ott is a scholar working on Molecular Biology, Nephrology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Kai M. Schmidt‐Ott has authored 106 papers receiving a total of 5.8k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Molecular Biology, 36 papers in Nephrology and 19 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Kai M. Schmidt‐Ott's work include Renal and related cancers (30 papers), Acute Kidney Injury Research (27 papers) and Chronic Kidney Disease and Diabetes (12 papers). Kai M. Schmidt‐Ott is often cited by papers focused on Renal and related cancers (30 papers), Acute Kidney Injury Research (27 papers) and Chronic Kidney Disease and Diabetes (12 papers). Kai M. Schmidt‐Ott collaborates with scholars based in Germany, United States and Japan. Kai M. Schmidt‐Ott's co-authors include Jonathan Barasch, Kiyoshi Mori, Prasad Devarajan, Avtandil Kalandadze, Neal Paragas, David J. Cohen, Klemens Budde, Vivette D. D’Agati, Shuntaro Kagiyama and Timm H. Westhoff and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Clinical Investigation.

In The Last Decade

Kai M. Schmidt‐Ott

95 papers receiving 5.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Endocytic delivery of lipocalin-siderophore-iron complex rescues the kidney from ischemia-reperfusion injury

2005 746 citations Kiyoshi Mori, Kai M. Schmidt‐Ott et al. profile →
Dual Action of Neutrophil Gelatinase–Associated Lipocalin

2007 619 citations Kai M. Schmidt‐Ott, Kiyoshi Mori et al. Journal of the American Society of Nephrology profile →
Kidney physiology and susceptibility to acute kidney injury: implications for renoprotection

2021 197 citations Holger Scholz, Felix Boivin et al. Nature Reviews Nephrology profile →

Peers

Countries citing papers authored by Kai M. Schmidt‐Ott

Since Specialization

Citations

This map shows the geographic impact of Kai M. Schmidt‐Ott's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kai M. Schmidt‐Ott with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kai M. Schmidt‐Ott more than expected).

Fields of papers citing papers by Kai M. Schmidt‐Ott

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kai M. Schmidt‐Ott. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kai M. Schmidt‐Ott. The network helps show where Kai M. Schmidt‐Ott may publish in the future.

Co-authorship network of co-authors of Kai M. Schmidt‐Ott

This figure shows the co-authorship network connecting the top 25 collaborators of Kai M. Schmidt‐Ott. A scholar is included among the top collaborators of Kai M. Schmidt‐Ott based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kai M. Schmidt‐Ott. Kai M. Schmidt‐Ott is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Progressionshemmung bei chronischer Nierenerkrankung: Stellenwert von Mineralokortikoidrezeptorantagonisten 2024 ·Robert Greite, Kai M. Schmidt‐Ott	0
2	Beyond hemoglobin: Critical role of 2,3‐bisphosphoglycerate mutase in kidney function and injury 2024 ·Acta Physiologica ·(unknown), (unknown), (unknown), Mumtaz Kasim, Susanne Mathia, (unknown), Nikolaus Berndt, (unknown), Gohar Ter‐Avetisyan, Friedrich C. Luft, Philipp Enghard, Christian Hinze, Jan Klocke, Kai‐Uwe Eckardt, Kai M. Schmidt‐Ott, Pontus B. Persson, Christian Rosenberger, Michael Fähling	0
3	Empagliflozin in patients with autosomal dominant polycystic kidney disease (EMPA-PKD): study protocol for a randomised controlled trial 2024 ·BMJ Open ·(unknown), (unknown), Stefanie D. Krämer, (unknown), (unknown), Jessica Kaufeld, Armin Koch, Dagmar Hartung, Bernhard M. W. Schmidt, Kai M. Schmidt‐Ott, Roland Schmitt	5
4	CSF-1 and Notch signaling cooperate in macrophage instruction and tissue repair during peripheral limb ischemia 2023 ·Frontiers in Immunology ·Tamar Kapanadze, Jaba Gamrekelashvili, (unknown), (unknown), Hermann Haller, Kai M. Schmidt‐Ott, Florian P. Limbourg	3
5	Blood pressure dynamics during home blood pressure monitoring with a digital blood pressure coach—a prospective analysis of individual user data 2023 ·Frontiers in Cardiovascular Medicine ·(unknown), Dominik Rüegger, (unknown), Steffen Wagner, Herrmann Haller, Kai M. Schmidt‐Ott, (unknown), Florian P. Limbourg	4
6	#5924 LATE DIAGNOSIS OF IGA NEPHROPATHY IN LOWER SAXONY (GERMANY) – CLINICAL AND HISTOPATHOLOGICAL DATA FROM 246 PATIENTS AT TIME OF INITIAL RENAL BIOPSY 2023 ·Nephrology Dialysis Transplantation ·(unknown), Torsten M. Meyer, Gabriele Eden, Bernhard M. W. Schmidt, Kai M. Schmidt‐Ott, Jan Hinrich Bräsen, Jan T. Kielstein	1
7	Prevalence of pericardial effusion in autosomal dominant polycystic kidney disease 2023 ·Clinical Kidney Journal ·(unknown), (unknown), Bernd Auber, Johannes Beller, Kai M. Schmidt‐Ott, Roland Schmitt, (unknown)	1
8	Plasma NGAL levels in stable kidney transplant recipients and the risk of allograft loss 2023 ·Nephrology Dialysis Transplantation ·(unknown), (unknown), (unknown), Eugenia Singer, Friederike Bachmann, Fabian Halleck, Susanne Kron, Marcel G. Naik, Danilo Schmidt, Martin Obermeier, (unknown), Geraldine Rauch, Siegfried Kropf, Michael Haase, Klemens Budde, Kai‐Uwe Eckardt, Timm H. Westhoff, Kai M. Schmidt‐Ott	2
9	Was ist gesichert in der Therapie der chronischen Nierenerkrankung? 2022 ·Die Innere Medizin ·Robert Greite, Kai M. Schmidt‐Ott	1
10	Kidney physiology and susceptibility to acute kidney injury: implications for renoprotection breakdown → 2021 ·Nature Reviews Nephrology ·Holger Scholz, Felix Boivin, Kai M. Schmidt‐Ott, Sebastian Bachmann, Kai‐Uwe Eckardt, Ute I. Scholl, Pontus B. Persson	197
11	Limited utility of qPCR-based detection of tumor-specific circulating mRNAs in whole blood from clear cell renal cell carcinoma patients 2020 ·BMC Urology ·(unknown), Christian Hinze, Kai M. Schmidt‐Ott, Jonas Busch, Monika Jung, Klaus Jung, Anja Rabien	5
12	Fluconazole Increases Osmotic Water Transport in Renal Collecting Duct through Effects on Aquaporin-2 Trafficking 2019 ·Journal of the American Society of Nephrology ·(unknown), Christian Hinze, (unknown), Nina Himmerkus, Catarina Quintanova, Kerstin Zühlke, Friederike Compton, (unknown), Alessandro Dema, Jenny Eichhorst, Burkhard Wiesner, Markus Bleich, Kai M. Schmidt‐Ott, Enno Klußmann	18
13	The IgCAM CLMP regulates expression of Connexin43 and Connexin45 in intestinal and ureteral smooth muscle contraction in mice 2018 ·Disease Models & Mechanisms ·(unknown), René Jüttner, (unknown), (unknown), (unknown), Bettina Purfürst, Kai M. Schmidt‐Ott, Andreas Friebe, Fritz G. Rathjen	22
14	The basal chorionic trophoblast cell layer: An emerging coordinator of placenta development 2016 ·BioEssays ·Katharina Walentin, Christian Hinze, Kai M. Schmidt‐Ott	30
15	Redox Regulation of Cell Contacts by Tricellulin and Occludin: Redox-Sensitive Cysteine Sites in Tricellulin Regulate Both Tri- and Bicellular Junctions in Tissue Barriers as Shown in Hypoxia and Ischemia 2015 ·Antioxidants and Redox Signaling ·Jimmi Cording, (unknown), Emilia Vigolo, (unknown), Lars Winkler, (unknown), Dorothea Lorenz, Reiner F. Haseloff, Kai M. Schmidt‐Ott, Hartwig Wolburg, Ingolf E. Blasig	29
16	WNT/β-catenin signaling in nephron progenitors and their epithelial progeny 2008 ·Kidney International ·Kai M. Schmidt‐Ott, Jonathan Barasch	101
17	Localization of a Gene for Nonsyndromic Renal Hypodysplasia to Chromosome 1p32-33 2007 ·The American Journal of Human Genetics ·Simone Sanna‐Cherchi, Gianluca Caridi, Patricia L. Weng, Monica Dagnino, Marco Seri, (unknown), Danio Somenzi, Alba Carrea, Claudia Izzi, D. Casu, Landino Allegri, Kai M. Schmidt‐Ott, Jonathan Barasch, Francesco Scolari, Roberto Ravazzolo, Gian Marco Ghiggeri, Ali G. Gharavi	23
18	c-kit delineates a distinct domain of progenitors in the developing kidney 2006 ·Developmental Biology ·Kai M. Schmidt‐Ott, Xia Chen, Neal Paragas, Randy Levinson, Cathy Mendelsohn, Jonathan Barasch	47
19	Detection of intracellular iron by its regulatory effect 2004 ·American Journal of Physiology-Cell Physiology ·Jau‐Yi Li, (unknown), Katherine M. Gast, Xia Chen, (unknown), Kiyoshi Mori, Kai M. Schmidt‐Ott, Jianjun Wang, (unknown), Cathy Savage‐Dunn, Michael D. Garrick, Jonathan Barasch	44
20	Single-Cell Characterization of Endothelin System Gene Expression in the Cerebellum In Situ 1998 ·Journal of Cardiovascular Pharmacology ·Kai M. Schmidt‐Ott, (unknown), Frank Kirchhoff, Alexei Verkhratsky, Lutz Liefeldt, Helmut Kettenmann, Martin Paul	18

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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