Kae Shimazaki

652 total citations
18 papers, 510 citations indexed

About

Kae Shimazaki is a scholar working on Pathology and Forensic Medicine, Immunology and Oncology. According to data from OpenAlex, Kae Shimazaki has authored 18 papers receiving a total of 510 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Pathology and Forensic Medicine, 6 papers in Immunology and 5 papers in Oncology. Recurrent topics in Kae Shimazaki's work include Lymphoma Diagnosis and Treatment (8 papers), T-cell and Retrovirus Studies (4 papers) and Viral-associated cancers and disorders (4 papers). Kae Shimazaki is often cited by papers focused on Lymphoma Diagnosis and Treatment (8 papers), T-cell and Retrovirus Studies (4 papers) and Viral-associated cancers and disorders (4 papers). Kae Shimazaki collaborates with scholars based in Japan. Kae Shimazaki's co-authors include Junji Suzumiya, Masahiro Kikuchi, Koichi Ohshima, M Kanda, Chika Kawasaki, Motonobu Kanda, Midori Sugihara, Seiji Haraoka, Toshihiro Tanaka and Kensaku Sato and has published in prestigious journals such as British Journal of Haematology, Histopathology and Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin.

In The Last Decade

Kae Shimazaki

18 papers receiving 496 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kae Shimazaki Japan 13 298 185 131 130 85 18 510
K L Chang United States 7 302 1.0× 362 2.0× 107 0.8× 86 0.7× 42 0.5× 8 620
Dennis B. Cornfield United States 14 175 0.6× 195 1.1× 58 0.4× 69 0.5× 69 0.8× 29 479
Sumie Tabata Japan 13 141 0.5× 137 0.7× 50 0.4× 113 0.9× 96 1.1× 49 449
Yoshihito Hayami Japan 13 197 0.7× 154 0.8× 49 0.4× 161 1.2× 169 2.0× 26 583
Marina Narbaitz Argentina 15 423 1.4× 411 2.2× 81 0.6× 126 1.0× 63 0.7× 55 663
Keisuke Kawamoto Japan 14 279 0.9× 245 1.3× 44 0.3× 215 1.7× 60 0.7× 48 646
A. Neubauer Germany 7 396 1.3× 187 1.0× 479 3.7× 202 1.6× 87 1.0× 22 921
D. Rontogianni Greece 13 108 0.4× 138 0.7× 74 0.6× 58 0.4× 89 1.0× 32 427
Anne–Catherine Baglin France 10 210 0.7× 239 1.3× 151 1.2× 118 0.9× 49 0.6× 28 539
Mitsutoshi Kurosawa Japan 13 140 0.5× 209 1.1× 108 0.8× 108 0.8× 119 1.4× 49 610

Countries citing papers authored by Kae Shimazaki

Since Specialization
Citations

This map shows the geographic impact of Kae Shimazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kae Shimazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kae Shimazaki more than expected).

Fields of papers citing papers by Kae Shimazaki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kae Shimazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kae Shimazaki. The network helps show where Kae Shimazaki may publish in the future.

Co-authorship network of co-authors of Kae Shimazaki

This figure shows the co-authorship network connecting the top 25 collaborators of Kae Shimazaki. A scholar is included among the top collaborators of Kae Shimazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kae Shimazaki. Kae Shimazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Kato, Shinichi, et al.. (2005). Herpes simplex esophagitis in the immunocompetent host. Diseases of the Esophagus. 18(5). 340–344. 34 indexed citations
2.
Yamamoto, Ryuichi, Shingo Kato, Kae Shimazaki, et al.. (2005). A case of primary rectal mucosa‐associated lymphoid tissue lymphoma treated by endoscopic mucosal resection. Digestive Endoscopy. 17(2). 172–174. 2 indexed citations
3.
Ohshima, Koichi, Kennosuke Karube, Kae Shimazaki, et al.. (2003). Imbalance Between Apoptosis and Telomerase Activity in Myelodysplastic Syndromes: Possible Role in Ineffective Hemopoiesis. Leukemia & lymphoma. 44(8). 1339–1346. 21 indexed citations
4.
Shimazaki, Kae, K Ohshima, Junji Suzumiya, Chika Kawasaki, & Masahiro Kikuchi. (2002). Apoptosis and Prognostic Factors in Myelodysplastic Syndromes. Leukemia & lymphoma. 43(2). 257–260. 9 indexed citations
5.
6.
Shimazaki, Kae, Kohichi Ohshima, Seiji Haraoka, et al.. (2002). Accessory cell tumour: a clinicopathological study of 16 aggressive tumours containing EBV‐positive Hodgkin and Reed–Sternberg‐like giant cells. Histopathology. 40(1). 12–21. 10 indexed citations
7.
Kono, Suminori, Hiroshi Abe, Hiroyuki Eguchi, et al.. (2001). Glucose Intolerance, Plasma Insulin Levels, and Colon Adenomas in Japanese Men. Japanese Journal of Cancer Research. 92(8). 836–840. 44 indexed citations
8.
Ohshima, Koichi, Midori Sugihara, Seiji Haraoka, et al.. (2001). Possible Immortalization of Hodgkin and Reed-Sternberg Cells: Telomerase Expression, Lengthening of Telomere, and Inhibition of Apoptosis by NF-KappaB Expression. Leukemia & lymphoma. 41(3-4). 367–376. 11 indexed citations
9.
Katoh, Akiko, Koichi Ohshima, Motonobu Kanda, et al.. (2000). Gastrointestinal T Cell Lymphoma: Predominant Cytotoxic Phenotypes, Including Alpha/Beta, Gamma/Delta T Cell and Natural Killer Cells. Leukemia & lymphoma. 39(1-2). 97–111. 45 indexed citations
10.
Shimazaki, Kae, Koichi Ohshima, Junji Suzumiya, Chika Kawasaki, & Masahiro Kikuchi. (2000). Evaluation of apoptosis as a prognostic factor in myelodysplastic syndromes. British Journal of Haematology. 110(3). 584–590. 33 indexed citations
11.
Ohshima, Koichi, Kae Shimazaki, Midori Sugihara, et al.. (1999). Clinicopathological findings of virus‐associated hemophagocytic syndrome in bone marrow: Association with Epstein‐Barr virus and apoptosis. Pathology International. 49(6). 533–540. 16 indexed citations
12.
Ohshima, Koichi, et al.. (1999). Clinical, immunohistochemical and phenotypic features of aggressive nodal cytotoxic lymphomas, including α/β, γ/δ T-cell and natural killer cell types. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 435(2). 92–100. 21 indexed citations
13.
Ohshima, Koichi, Kensaku Sato, Motonobu Kanda, et al.. (1999). Prognostic clinicopathologic factors, including immunologic expression in diffuse large B‐cell lymphomas. Pathology International. 49(12). 1043–1052. 62 indexed citations
14.
Ohshima, Koichi, Seiji Haraoka, Junji Suzumiya, et al.. (1999). Absence of cytotoxic molecules in CD8- and/or CD56-positive adult T-cell leukaemia/lymphoma. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 435(2). 101–104. 14 indexed citations
15.
Sugihara, Midori, et al.. (1999). Basic Fibroblast Growth Factor and Fibrosis in Hodgkin's Disease. Pathology - Research and Practice. 195(3). 149–155. 21 indexed citations
16.
Ohshima, Koichi, et al.. (1998). Perforin and Fas pathways of cytotoxic T‐cells in histiocytic necrotizing lymphadenitis. Histopathology. 33(5). 471–478. 70 indexed citations
17.
Ohshima, Koichi, et al.. (1998). Apoptosis of cytotoxic T-cells in histiocytic necrotizing lymphadenitis. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 433(2). 131–134. 30 indexed citations
18.
Ohshima, Koichi, Junji Suzumiya, Kae Shimazaki, et al.. (1997). Nasal T/NK cell lymphomas commonly express perforin and Fas ligand: important mediators of tissue damage. Histopathology. 31(5). 444–450. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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