Ka Ka Ting

1.0k total citations
20 papers, 765 citations indexed

About

Ka Ka Ting is a scholar working on Molecular Biology, Immunology and Physiology. According to data from OpenAlex, Ka Ka Ting has authored 20 papers receiving a total of 765 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Immunology and 5 papers in Physiology. Recurrent topics in Ka Ka Ting's work include Cell Adhesion Molecules Research (5 papers), Angiogenesis and VEGF in Cancer (4 papers) and Alzheimer's disease research and treatments (3 papers). Ka Ka Ting is often cited by papers focused on Cell Adhesion Molecules Research (5 papers), Angiogenesis and VEGF in Cancer (4 papers) and Alzheimer's disease research and treatments (3 papers). Ka Ka Ting collaborates with scholars based in Australia, United States and China. Ka Ka Ting's co-authors include Gilles J. Guillemin, Bruce J. Brew, Jennifer R. Gamble, Mathew A. Vadas, Paul Coleman, Yang Zhao, Roger S. Chung, Seray Adams, Jia Li and Jinbiao Chen and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Circulation Research.

In The Last Decade

Ka Ka Ting

20 papers receiving 759 citations

Peers

Ka Ka Ting
Zhenzhong Ma United States
Ammar Kutiyanawalla United States
Silvia Caioli Italy
L.-W. Fan United States
Li‐Jin Chew United States
Kelly Ceyzériat Switzerland
Delphine Stephan France
Sabrina Etteri Italy
Elissa J. Donzis United States
Alerie Guzman de la Fuente United Kingdom
Zhenzhong Ma United States
Ka Ka Ting
Citations per year, relative to Ka Ka Ting Ka Ka Ting (= 1×) peers Zhenzhong Ma

Countries citing papers authored by Ka Ka Ting

Since Specialization
Citations

This map shows the geographic impact of Ka Ka Ting's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ka Ka Ting with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ka Ka Ting more than expected).

Fields of papers citing papers by Ka Ka Ting

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ka Ka Ting. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ka Ka Ting. The network helps show where Ka Ka Ting may publish in the future.

Co-authorship network of co-authors of Ka Ka Ting

This figure shows the co-authorship network connecting the top 25 collaborators of Ka Ka Ting. A scholar is included among the top collaborators of Ka Ka Ting based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ka Ka Ting. Ka Ka Ting is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Coleman, Paul, Ka Ka Ting, Yanfei Qi, Mathew A. Vadas, & Jennifer R. Gamble. (2023). Endothelial cell senescence — understanding aging and disease. Current Opinion in Physiology. 35. 100702–100702. 1 indexed citations
2.
Ting, Ka Ka, Paul Coleman, Hani Jieun Kim, et al.. (2023). Vascular senescence and leak are features of the early breakdown of the blood–brain barrier in Alzheimer’s disease models. GeroScience. 45(6). 3307–3331. 42 indexed citations
3.
Liu, Ken, Jinbiao Chen, Yang Zhao, et al.. (2023). Novel miRNA-based drug CD5-2 reduces liver tumor growth in diethylnitrosamine-treated mice by normalizing tumor vasculature and altering immune infiltrate. Frontiers in Immunology. 14. 1245708–1245708. 6 indexed citations
4.
Zhao, Yang, Ka Ka Ting, Paul Coleman, et al.. (2021). The Tumour Vasculature as a Target to Modulate Leucocyte Trafficking. Cancers. 13(7). 1724–1724. 16 indexed citations
5.
Ting, Ka Ka, Paul Coleman, Yang Zhao, Mathew A. Vadas, & Jennifer R. Gamble. (2021). The aging endothelium. SHILAP Revista de lepidopterología. 3(1). R35–R47. 22 indexed citations
6.
Zhao, Yang, Jia Li, Ka Ka Ting, et al.. (2020). The VE-Cadherin/β-catenin signalling axis regulates immune cell infiltration into tumours. Cancer Letters. 496. 1–15. 23 indexed citations
7.
Li, Jia, Yang Zhao, Jaesung P. Choi, et al.. (2020). Targeting miR-27a/VE-cadherin interactions rescues cerebral cavernous malformations in mice. PLoS Biology. 18(6). e3000734–e3000734. 36 indexed citations
8.
Coleman, Paul, Angelina J. Lay, Ka Ka Ting, et al.. (2020). YAP and the RhoC regulator ARHGAP18, are required to mediate flow-dependent endothelial cell alignment. Cell Communication and Signaling. 18(1). 18–18. 17 indexed citations
9.
Li, Jia, Yang Zhao, Paul Coleman, et al.. (2019). Low fluid shear stress conditions contribute to activation of cerebral cavernous malformation signalling pathways. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865(11). 165519–165519. 31 indexed citations
10.
Lay, Angelina J., Paul Coleman, Ka Ka Ting, et al.. (2019). ARHGAP18: A Flow‐Responsive Gene That Regulates Endothelial Cell Alignment and Protects Against Atherosclerosis. Journal of the American Heart Association. 8(2). e010057–e010057. 15 indexed citations
11.
Choi, Jaesung P., Rui Wang, Xi Yang, et al.. (2018). Ponatinib (AP24534) inhibits MEKK3-KLF signaling and prevents formation and progression of cerebral cavernous malformations. Science Advances. 4(11). eaau0731–eaau0731. 39 indexed citations
12.
Ting, Ka Ka, Yang Zhao, Weiyong Shen, et al.. (2018). Therapeutic regulation of VE-cadherin with a novel oligonucleotide drug for diabetic eye complications using retinopathy mouse models. Diabetologia. 62(2). 322–334. 30 indexed citations
13.
Zhao, Yang, Ka Ka Ting, Jia Li, et al.. (2017). Targeting Vascular Endothelial-Cadherin in Tumor-Associated Blood Vessels Promotes T-cell–Mediated Immunotherapy. Cancer Research. 77(16). 4434–4447. 50 indexed citations
14.
Liu, Renjing, Lisa Lo, Angelina J. Lay, et al.. (2017). ARHGAP18 Protects Against Thoracic Aortic Aneurysm Formation by Mitigating the Synthetic and Proinflammatory Smooth Muscle Cell Phenotype. Circulation Research. 121(5). 512–524. 47 indexed citations
15.
Lay, Angelina J., Ka Ka Ting, Yang Zhao, et al.. (2014). ARHGAP18: an endogenous inhibitor of angiogenesis, limiting tip formation and stabilizing junctions. Small GTPases. 5(3). e975002–e975002. 59 indexed citations
16.
Ting, Ka Ka, et al.. (2010). Characterisation of the Expression of NMDA Receptors in Human Astrocytes. PLoS ONE. 5(11). e14123–e14123. 143 indexed citations
17.
Guillemin, Gilles J., Abdur Rahman, Ka Ka Ting, et al.. (2010). P4‐002: Involvement of the Kynurenine Pathway in Alzheimer's Disease. Alzheimer s & Dementia. 6(4S_Part_20). 1 indexed citations
18.
Ting, Ka Ka, Bruce J. Brew, & Gilles J. Guillemin. (2009). Effect of quinolinic acid on human astrocytes morphology and functions: implications in Alzheimer's disease. Journal of Neuroinflammation. 6(1). 36–36. 135 indexed citations
19.
Ting, Ka Ka, Bruce J. Brew, & Gilles J. Guillemin. (2007). Effect of quinolinic acid on gene expression in human astrocytes: Implications for Alzheimer's disease. International Congress Series. 1304. 384–388. 10 indexed citations
20.
Ting, Ka Ka, Bruce J. Brew, & Gilles J. Guillemin. (2007). The involvement of astrocytes and kynurenine pathway in Alzheimer’s disease. Neurotoxicity Research. 12(4). 247–262. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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