K Wonigeit

5.0k total citations
174 papers, 4.0k citations indexed

About

K Wonigeit is a scholar working on Immunology, Transplantation and Surgery. According to data from OpenAlex, K Wonigeit has authored 174 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 62 papers in Immunology, 60 papers in Transplantation and 48 papers in Surgery. Recurrent topics in K Wonigeit's work include Renal Transplantation Outcomes and Treatments (56 papers), T-cell and B-cell Immunology (45 papers) and Immune Cell Function and Interaction (42 papers). K Wonigeit is often cited by papers focused on Renal Transplantation Outcomes and Treatments (56 papers), T-cell and B-cell Immunology (45 papers) and Immune Cell Function and Interaction (42 papers). K Wonigeit collaborates with scholars based in Germany, United States and Japan. K Wonigeit's co-authors include R. Pichlmayr, Reinhard Schwinzer, Gustav Steinhoff, Hans J. Schlitt, J. Hundrieser, B. Ringe, Björn Nashan, Gereon Raddatz, Matthias Behrend and Hidde L. Ploegh and has published in prestigious journals such as Science, The Lancet and Nature Medicine.

In The Last Decade

K Wonigeit

174 papers receiving 3.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
K Wonigeit Germany 34 1.7k 1.0k 1.0k 867 571 174 4.0k
Thomas Wekerle Austria 36 2.5k 1.5× 1.6k 1.6× 2.1k 2.0× 558 0.6× 138 0.2× 176 5.5k
Ruchuang Ding United States 28 1.0k 0.6× 1.4k 1.3× 1.8k 1.7× 955 1.1× 77 0.1× 47 3.9k
J. R. Batchelor United Kingdom 37 2.6k 1.5× 556 0.5× 424 0.4× 465 0.5× 68 0.1× 97 4.9k
T. Mohanakumar United States 31 815 0.5× 1.5k 1.5× 716 0.7× 435 0.5× 97 0.2× 106 2.7k
Nader Najafian United States 40 3.3k 2.0× 942 0.9× 1.2k 1.1× 699 0.8× 58 0.1× 96 5.3k
Wuding Zhou United Kingdom 39 3.2k 1.9× 757 0.7× 1.1k 1.1× 499 0.6× 79 0.1× 93 4.4k
Behdad Afzali United Kingdom 33 2.4k 1.5× 424 0.4× 446 0.4× 663 0.8× 93 0.2× 85 4.2k
Michael Eikmans Netherlands 30 988 0.6× 546 0.5× 674 0.7× 641 0.7× 261 0.5× 108 2.8k
Zoltán Lászik United States 27 1.0k 0.6× 493 0.5× 363 0.4× 998 1.2× 205 0.4× 87 3.9k
Jean‐Luc Taupin France 37 2.3k 1.4× 1.1k 1.0× 1.5k 1.4× 890 1.0× 55 0.1× 167 4.6k

Countries citing papers authored by K Wonigeit

Since Specialization
Citations

This map shows the geographic impact of K Wonigeit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K Wonigeit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K Wonigeit more than expected).

Fields of papers citing papers by K Wonigeit

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K Wonigeit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K Wonigeit. The network helps show where K Wonigeit may publish in the future.

Co-authorship network of co-authors of K Wonigeit

This figure shows the co-authorship network connecting the top 25 collaborators of K Wonigeit. A scholar is included among the top collaborators of K Wonigeit based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K Wonigeit. K Wonigeit is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wallemacq, Pierre, et al.. (2008). Analytical multi-site evaluation of the architect cyclosporine assay. American Journal of Transplantation. 8. 516–516. 2 indexed citations
2.
Baars, Wiebke, et al.. (2001). Target cells expressing CD95L are protected from lysis mediated by alloactivated T cells but are killed by resting NK cells. Transplantation Proceedings. 33(1-2). 262–263. 3 indexed citations
3.
Lambracht‐Washington, Doris, K F Lindahl, & K Wonigeit. (2000). Promoter structures suggest independent translocations of ancestral rat RT1.A and mouse H2-K class I genes. Immunogenetics. 51(10). 873–877. 7 indexed citations
4.
Deiwick, Andrea, et al.. (1999). Functional relevance of donor-derived hematopoietic microchimerism only for induction but not for maintenance of allograft acceptance. Transplantation Proceedings. 31(1-2). 920–921. 5 indexed citations
5.
Kiuchi, Tetsuya, K Oldhafer, Björn Nashan, et al.. (1998). BACKGROUND AND PROGNOSTIC IMPLICATIONS OF PERIREPERFUSION TISSUE INJURIES IN HUMAN LIVER TRANSPLANTS. Transplantation. 66(6). 737–747. 29 indexed citations
6.
Ueda, Mikiko, J. Hundrieser, Michiyoshi Hisanaga, et al.. (1997). Development of microchimerism in pediatric patients after living‐related liver transplantation. Clinical Transplantation. 11(3). 193–199. 28 indexed citations
7.
Vaage, John Torgils, et al.. (1997). Positive and negative MHC class I recognition by rat NK cells. Immunological Reviews. 155(1). 91–104. 48 indexed citations
8.
Vaage, John Torgils, Christian Naper, Annegret Rehm, et al.. (1994). Control of rat natural killer cell-mediated allorecognition by a major histocompatibility complex region encoding nonclassical class I antigens.. The Journal of Experimental Medicine. 180(2). 641–651. 72 indexed citations
9.
Winkler, M., B Ringe, Burkhard Rodeck, et al.. (1994). The use of plasma levels for FK 506 dosing in liver-grafted patients. Transplant International. 7(5). 329–333. 16 indexed citations
10.
Schumann, G., et al.. (1993). Monitoring Cyclosporin A (Ciclosporin, INN) Concentrations in Whole Blood: Evaluation of the EMIT™ Assay in Comparison with HPLC and RIA. Clinical Chemistry and Laboratory Medicine (CCLM). 31(6). 381–8. 11 indexed citations
11.
Hedrich, Hans J., et al.. (1993). Deletion of major histocompatibility complex class I genes in a new mutant RT1 haplotype (lm3) defined in BN.1LM3/Han rats.. PubMed. 25(5). 2756–8. 5 indexed citations
12.
Nashan, Björn, et al.. (1992). INTRAGRAFT IMMUNE EVENTS AFTER HUMAN LIVER TRANSPLANTATION. Transplantation. 54(2). 273–277. 20 indexed citations
13.
Christians, Uwe, K. Kohlhaw, J. S. Bleck, et al.. (1991). Ciclosporin metabolite pattern in blood and urine of liver graft recipients. European Journal of Clinical Pharmacology. 41(4). 291–296. 11 indexed citations
14.
Schlitt, Hans J., Uwe Christians, J. S. Bleck, et al.. (1991). Contribution of cyclosporin metabolites to immunosuppression in liver-transplanted patients with severe graft dysfunction. Transplant International. 4(1). 38–44. 6 indexed citations
15.
Schlitt, Hans J., B Ringe, Christian Wittekind, et al.. (1990). Die klinische Bedeutung der Transplantatbiopsie für die Behandlung lebertransplantierter Patienten. Toxicologic Pathology. 28(1). 137–41. 1 indexed citations
16.
Wurst, Wolfgang, K Wonigeit, & E. Günther. (1989). A mutant rat major histocompatibility haplotype showing a large deletion of class I sequences. Immunogenetics. 30(4). 237–242. 16 indexed citations
17.
Nashan, Björn, et al.. (1989). Patterns of immune activation during the first four weeks in liver transplant patients.. PubMed. 21(4). 3623–4. 1 indexed citations
18.
Pichlmayr, R., et al.. (1985). Experience with cyclosporin/prednisolone therapy in kidney transplantation, with special reference to nephrotoxicity, ATN and kidney preservation.. PubMed. 28(6). 268–71. 1 indexed citations
19.
Günther, E., Wolfgang Wurst, K Wonigeit, & Jörg T. Epplen. (1985). Analysis of the rat major histocompatibility system by Southern blot hybridization.. The Journal of Immunology. 134(2). 1257–1261. 35 indexed citations
20.
Wonigeit, K & R. Pichlmayr. (1978). Posttransplant monitoring of donor-specific T-cell reactivity at the precursor cell level.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 10(3). 563–7. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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