K. Kleibl

540 total citations
18 papers, 461 citations indexed

About

K. Kleibl is a scholar working on Molecular Biology, Genetics and Cancer Research. According to data from OpenAlex, K. Kleibl has authored 18 papers receiving a total of 461 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 8 papers in Genetics and 6 papers in Cancer Research. Recurrent topics in K. Kleibl's work include DNA Repair Mechanisms (15 papers), Bacterial Genetics and Biotechnology (7 papers) and Carcinogens and Genotoxicity Assessment (6 papers). K. Kleibl is often cited by papers focused on DNA Repair Mechanisms (15 papers), Bacterial Genetics and Biotechnology (7 papers) and Carcinogens and Genotoxicity Assessment (6 papers). K. Kleibl collaborates with scholars based in Slovakia, United Kingdom and Poland. K. Kleibl's co-authors include Jacques Laval, Murat Saparbaev, G.P. Margison, Françoise Laval, Jaroslav Jelı́nek, Lynn Cawkwell, Philip M. Potter, T. M. Dexter, Jela Brozmanová and Milan Škorvaga and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Biophysical Journal.

In The Last Decade

K. Kleibl

18 papers receiving 453 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
K. Kleibl Slovakia	10	394	129	96	34	31	18	461
Gagan A. Pandya United States	8	266 0.7×	107 0.8×	51 0.5×	30 0.9×	33 1.1×	12	389
Anu Voho Finland	10	505 1.3×	142 1.1×	104 1.1×	24 0.7×	21 0.7×	15	622
Lioudmila V. Loukachevitch United States	13	553 1.4×	137 1.1×	71 0.7×	20 0.6×	24 0.8×	20	660
Ramiro Dip Switzerland	11	263 0.7×	71 0.6×	74 0.8×	55 1.6×	41 1.3×	18	429
Albena Kozekova United States	12	536 1.4×	180 1.4×	39 0.4×	51 1.5×	25 0.8×	19	603
O S Bhanot United States	13	423 1.1×	154 1.2×	66 0.7×	28 0.8×	61 2.0×	26	494
Shrey Jain India	8	229 0.6×	92 0.7×	32 0.3×	26 0.8×	77 2.5×	10	376
R Boorstein United States	8	234 0.6×	106 0.8×	31 0.3×	71 2.1×	33 1.1×	12	426
A. Margot France	12	433 1.1×	190 1.5×	24 0.3×	75 2.2×	49 1.6×	15	480
Anton J.L. de Groot Netherlands	7	410 1.0×	113 0.9×	40 0.4×	147 4.3×	26 0.8×	9	490

Countries citing papers authored by K. Kleibl

Since Specialization

Citations

This map shows the geographic impact of K. Kleibl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K. Kleibl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K. Kleibl more than expected).

Fields of papers citing papers by K. Kleibl

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K. Kleibl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K. Kleibl. The network helps show where K. Kleibl may publish in the future.

Co-authorship network of co-authors of K. Kleibl

This figure shows the co-authorship network connecting the top 25 collaborators of K. Kleibl. A scholar is included among the top collaborators of K. Kleibl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K. Kleibl. K. Kleibl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Gurský, Ján, et al.. (2009). Newly identified CHO ERCC3/XPB mutations and phenotype characterization. Mutagenesis. 25(2). 179–185. 8 indexed citations

Chovanec, Miroslav, et al.. (2004). Fourth DNA repair workshop on dna damage and repair: mechanisms and biological consequences. Smolenice Castle, 2-5 May 2004. DNA repair. 3(12). 1639–1659. 1 indexed citations

Slameňová, Darina, Eva Horváthová, Alena Gábelová, et al.. (2003). Molecular and cellular influences of butylated hydroxyanisole on Chinese hamster V79 cells treated with N‐methyl‐N′‐nitro‐N‐nitrosoguanidine: Antimutagenicity of butylated hydroxyanisole. Environmental and Molecular Mutagenesis. 41(1). 28–36. 12 indexed citations

Škorvaga, Milan, Miroslav Chovanec, Danuša Vlasáková, et al.. (2003). Effect of expression of theEscherichia coli nthgene inSaccharomyces cerevisiaeon the toxicity of ionizing radiation and hydrogen peroxide. International Journal of Radiation Biology. 79(9). 747–755. 3 indexed citations

Kleibl, K.. (2002). Molecular mechanisms of adaptive response to alkylating agents in Escherichia coli and some remarks on O6-methylguanine DNA-methyltransferase in other organisms. Mutation Research/Reviews in Mutation Research. 512(1). 67–84. 43 indexed citations

Kleibl, K., et al.. (1998). Increasing DNA repair capacity in bone marrow by gene transfer as a prospective tool in cancer therapy.. PubMed. 45(4). 181–6. 10 indexed citations

Saparbaev, Murat, K. Kleibl, & Jacques Laval. (1995). Escherichia coil,Saccharomyces cerevisiae, rat and human 3-methyladenine DNA glycosylases repair 1,N⁶-ethenoadenine when present in DNA. Nucleic Acids Research. 23(18). 3750–3755. 187 indexed citations

Kleibl, K., et al.. (1994). Repair of O6-methylguanine and O4-methylthymine by the human and rat O6-methylguanine-DNA methyltransferases.. Journal of Biological Chemistry. 269(1). 730–733. 59 indexed citations

Kleibl, K., et al.. (1993). Enhancement of the uvrA gene dosage reduces pyrimidine dimer excision in UV-irradiated Escherichia coli. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 290(2). 249–254. 1 indexed citations

10.

Fridrichová, Ivana, et al.. (1993). Inhibition of dimer excision in repeatedly UV-irradiated Escherichia coli: Its requirement for RecA protein and de novo protein synthesis. Journal of Photochemistry and Photobiology B Biology. 18(2-3). 205–210. 4 indexed citations

11.

Angelis, Karel J., et al.. (1992). Increased resistance to the toxic effects of alkylating agents in tobacco expressing the E. coli DNA repair gene ada. Mutation Research/DNA Repair. 273(3). 271–280. 12 indexed citations

12.

Fridrichová, Ivana, et al.. (1991). Expression of the Escherichia coli recA gene in the yeast Saccharomyces cerevisiae. Biochimie. 73(2-3). 285–288. 5 indexed citations

13.

Brozmanová, Jela, et al.. (1990). Expression of the E.coli ada gene in yeast protects against the toxic and mutagenic effects of N-methyl-N′ -nitro- N -nitrosoguanidine. Nucleic Acids Research. 18(2). 331–335. 13 indexed citations

14.

Kleibl, K., et al.. (1989). In UV-irradiatedEscherichia coli PQ35 overproducing the RecA protein, expression of thesfiA gene and dimer excision are alleviated. Molecular and General Genetics MGG. 217(2-3). 427–429. 2 indexed citations

15.

Potter, Philip M., K. Kleibl, Lynn Cawkwell, & G.P. Margison. (1989). Expression of theogtgene in wild-type andadamutants ofE.coli. Nucleic Acids Research. 17(20). 8047–8060. 37 indexed citations

16.

Jelı́nek, Jaroslav, K. Kleibl, T. M. Dexter, & G.P. Margison. (1988). Transfection of murine multi-potent haemopoietic stem cells with an E.coli DNA alkyltransferase gene confers resistance to the toxic effects of alkylating agents. Carcinogenesis. 9(1). 81–87. 37 indexed citations

17.

Kleibl, K., et al.. (1987). Inhibition of pyrimidine dimer excision in ultraviolet-irradiated Escherichia coli overproducing RecA protein. Mutation Research Letters. 191(1). 13–16. 8 indexed citations

18.

Brozmanová, Jela, et al.. (1981). Evidence that dimers remaining in preinduced Escherichia coli B/r Hcr+ become insensitive after DNA replication to the extract from Micrococcus luteus. Biophysical Journal. 36(2). 429–441. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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