K Itoh
- Molecular Biology
- Genetics
- Surgery
- Cellular and Molecular Neuroscience
- Endocrinology, Diabetes and Metabolism
- Co-authors
- Fujio UmeharaMasanobu NakataToshihiko YadaGenshu TateNaoki YamaguchiTsutomu DouchiToshiyuki MitsuyaMitsuhiro Osame
- Topics
- Hedgehog Signaling Pathway Studies (4 papers)Sexual Differentiation and Disorders (3 papers)T-cell and Retrovirus Studies (2 papers)
- Cited by
- UrologyGeneticsReproductive Medicine
- Partner nations
- JapanUnited KingdomArmenia
In The Last Decade
K Itoh
13 papers receiving 446 citations
Peers
Comparison fields: 5 of 55
- Molecular Biology 267
- Genetics 156
- Surgery 97
- Cellular and Molecular Neuroscience 83
- Endocrinology, Diabetes and Metabolism 69
Countries citing papers authored by K Itoh
This map shows the geographic impact of K Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K Itoh more than expected).
Fields of papers citing papers by K Itoh
This network shows the impact of papers produced by K Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K Itoh. The network helps show where K Itoh may publish in the future.
Co-authorship network of co-authors of K Itoh
This figure shows the co-authorship network connecting the top 25 collaborators of K Itoh. A scholar is included among the top collaborators of K Itoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K Itoh. K Itoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 30 | |
| 3 | 13 | |
| 4 | 27 | |
| 5 | Minifascicular neuropathy: a new concept of the human disease caused by desert hedgehog gene mutation. | 9 |
| 6 | 1 | |
| 7 | 138 | |
| 8 | 49 | |
| 9 | 77 | |
| 10 | [A case of myasthenia gravis associated with systemic lupus erythematosus and pemphigus erythematosus]. | 1 |
| 11 | [Multifunctional protective protein and its genetic deficiency 'galactosialidosis']. | 1 |
| 12 | 105 | |
| 13 | [Study on argyrophilic inclusions of multisystem atrophy (Oppenheimer)]. | 6 |
About K Itoh
K Itoh is a scholar working on Developmental Biology, Cellular and Molecular Neuroscience and Clinical Biochemistry, having authored 13 papers that have together received 458 indexed citations. Recurring topics across this work include Hedgehog Signaling Pathway Studies (4 papers), Sexual Differentiation and Disorders (3 papers) and T-cell and Retrovirus Studies (2 papers). The work is most often cited by research in Urology (45 citations), Genetics (156 citations) and Reproductive Medicine (45 citations). K Itoh has collaborated with scholars based in Japan, United Kingdom and Armenia. Frequent co-authors include Fujio Umehara, Masanobu Nakata, Toshihiko Yada, Genshu Tate, Naoki Yamaguchi, Tsutomu Douchi, Toshiyuki Mitsuya, Mitsuhiro Osame, M Osame and Mio Yamane. Their work appears in journals such as Endocrinology, The American Journal of Human Genetics and Acta Neuropathologica.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.