K. Ayed

1.6k total citations
91 papers, 1.3k citations indexed

About

K. Ayed is a scholar working on Immunology, Rheumatology and Epidemiology. According to data from OpenAlex, K. Ayed has authored 91 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Immunology, 27 papers in Rheumatology and 22 papers in Epidemiology. Recurrent topics in K. Ayed's work include Systemic Lupus Erythematosus Research (23 papers), T-cell and B-cell Immunology (15 papers) and Hepatitis B Virus Studies (14 papers). K. Ayed is often cited by papers focused on Systemic Lupus Erythematosus Research (23 papers), T-cell and B-cell Immunology (15 papers) and Hepatitis B Virus Studies (14 papers). K. Ayed collaborates with scholars based in Tunisia, France and Poland. K. Ayed's co-authors include Kamel Hamzaoui, A. Hamzaoui, M'Hamed Hamza, Yousr Gorgi, Fethi Guémira, Imen Sfar, M Hamza, T. Ben Abdallah, R. Bardi and J.L. Touraine and has published in prestigious journals such as SHILAP Revista de lepidopterología, The Journal of Immunology and World Journal of Gastroenterology.

In The Last Decade

K. Ayed

88 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
K. Ayed Tunisia 19 426 415 392 256 241 91 1.3k
Tammy M. Martin United States 28 598 1.4× 643 1.5× 529 1.3× 533 2.1× 299 1.2× 68 1.9k
Maxime Touzot France 13 782 1.8× 188 0.5× 163 0.4× 255 1.0× 104 0.4× 41 1.5k
P. T. Klouda United Kingdom 22 647 1.5× 273 0.7× 58 0.1× 130 0.5× 110 0.5× 57 1.5k
María Francisca González‐Escribano Spain 25 727 1.7× 498 1.2× 37 0.1× 260 1.0× 343 1.4× 64 1.6k
Sergio Peralta Italy 21 529 1.2× 571 1.4× 31 0.1× 205 0.8× 474 2.0× 49 1.6k
Evelyne T. Edwards United States 5 606 1.4× 885 2.1× 74 0.2× 91 0.4× 389 1.6× 6 1.9k
S. Trad France 14 555 1.3× 409 1.0× 412 1.1× 136 0.5× 177 0.7× 42 1.6k
Ibtissem Ghédira Tunisia 18 251 0.6× 276 0.7× 35 0.1× 92 0.4× 241 1.0× 70 885
Satoshi Motoya Japan 21 367 0.9× 132 0.3× 74 0.2× 147 0.6× 732 3.0× 65 1.4k
Sungnack Lee South Korea 19 124 0.3× 372 0.9× 621 1.6× 166 0.6× 201 0.8× 60 1.1k

Countries citing papers authored by K. Ayed

Since Specialization
Citations

This map shows the geographic impact of K. Ayed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K. Ayed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K. Ayed more than expected).

Fields of papers citing papers by K. Ayed

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K. Ayed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K. Ayed. The network helps show where K. Ayed may publish in the future.

Co-authorship network of co-authors of K. Ayed

This figure shows the co-authorship network connecting the top 25 collaborators of K. Ayed. A scholar is included among the top collaborators of K. Ayed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K. Ayed. K. Ayed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sfar, Imen, et al.. (2015). Natural evolution of hepatitis C virus infection in hemodialysis Tunisian patients and CTLA-4 SNP's. World Journal of Gastroenterology. 21(35). 10150–10158. 1 indexed citations
2.
Gorgi, Yousr, Lamjed Bouslama, Imen Sfar, et al.. (2013). Phylogenetic Analysis of Isolated HCV Strains from Tunisian Hemodialysis Patients. Viral Immunology. 26(1). 40–48. 9 indexed citations
3.
Sfar, Imen, R. Bardi, T. Ben Abdallah, et al.. (2011). CD86 +1057G>A polymorphism and susceptibility to acute kidney allograft rejection.. PubMed. 5(3). 187–93. 11 indexed citations
4.
Sfar, Imen, M. Makhlouf, R. Bardi, et al.. (2011). Allograft renal rejection and chemokine polymorphism.. PubMed. 22(1). 18–23. 4 indexed citations
6.
Gorgi, Yousr, Imen Sfar, R. Goucha, et al.. (2010). [IL1/IL1 Ra, CTLA-4 and Apo1/Fas genes polymorphisms and susceptibility to IgA nephropathy in Tunisian patients].. PubMed. 88(11). 789–93. 8 indexed citations
7.
Sfar, Imen, Hassen Hadj Kacem, R. Bardi, et al.. (2010). (AT) Repeat in the 3′ Untranslated Region of the CTLA-4 Gene and Susceptibility to Acute Allograft Rejection in Tunisian Renal Transplantation. Transplantation Proceedings. 42(10). 4314–4317. 7 indexed citations
8.
Sfar, Imen, Samira Matri, Azza Filali, et al.. (2009). Polymorphism in ICAM-1, PECAM-1, E-selectin, and L-selectin genes in Tunisian patients with inflammatory bowel disease. European Journal of Gastroenterology & Hepatology. 21(2). 167–175. 19 indexed citations
9.
Sfar, Imen, Yousr Gorgi, R. Bardi, et al.. (2009). The PTPN22 C1858T (R620W) Functional Polymorphism in Kidney Transplantation. Transplantation Proceedings. 41(2). 657–659. 8 indexed citations
10.
Sfar, Imen, et al.. (2007). Clearance and Persistence of Hepatitis C Virus in a Tunisian Population: Association with HLA Class I and Class II. Viral Immunology. 20(2). 312–319. 27 indexed citations
11.
Sfar, Imen, et al.. (2007). Role of immune system, apoptosis and angiogenesis in pathogenesis of rheumatoid arthritis and joint destruction, a systematic review.. PubMed. 85(12). 991–8. 24 indexed citations
12.
Gorgi, Yousr, Imen Sfar, T. Ben Abdallah, et al.. (2006). CTLA-4 Exon 1 (+49) and Promoter (−318) Gene Polymorphisms in Kidney Transplantation. Transplantation Proceedings. 38(7). 2303–2305. 26 indexed citations
13.
Hamzaoui, Kamel, H. Houman, Fayçal Hentati, et al.. (2006). Discrepancies of NKT cells expression in peripheral blood and in cerebrospinal fluid from Behçet's disease. Journal of Neuroimmunology. 175(1-2). 160–168. 14 indexed citations
14.
Ayed, K., et al.. (2006). Hepatitis B virus genotypes and precore/core-promoter mutations in Tunisian patients with chronic hepatitis B virus infection. Journal of Infection. 54(3). 291–297. 24 indexed citations
15.
Goucha, R., et al.. (2002). Autoantibodies to ribosomal P proteins in systemic lupus erythematosus. Joint Bone Spine. 69(2). 173–176. 10 indexed citations
16.
Hamzaoui, A., A Kahán, K. Ayed, & Kamel Hamzaoui. (2002). T cells expressing the γδ receptor are essential for Th2‐mediated inflammation in patients with acute exacerbation of asthma. Mediators of Inflammation. 11(2). 113–119. 18 indexed citations
17.
Hamza, M'Hamed, K. Ayed, R. Bardi, et al.. (1990). HLA-antigens in a Tunisian familial chondrocalcinosis.. PubMed. 8(3). 109–12. 5 indexed citations
18.
Hamzaoui, Kamel, et al.. (1990). Natural killer cell activity, interferon-gamma and antibodies to herpes viruses in patients with Behçet's disease. Clinical & Experimental Immunology. 79(1). 28–34. 58 indexed citations
19.
Bellon, B, J Kuhn, K. Ayed, Jean Girard, & P Druet. (1979). Experimental immune glomerulonephritis induced in the rabbit with streptococcal vaccine.. PubMed Central. 37(2). 239–46. 4 indexed citations
20.
Druet, P, K. Ayed, J Bariéty, et al.. (1979). Experimental immune glomerulonephritis induced in the rat by mercuric chloride.. PubMed. 8. 321–42. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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