Jun‐Qiang Si

1.2k total citations
85 papers, 967 citations indexed

About

Jun‐Qiang Si is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jun‐Qiang Si has authored 85 papers receiving a total of 967 indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Molecular Biology, 36 papers in Physiology and 23 papers in Cellular and Molecular Neuroscience. Recurrent topics in Jun‐Qiang Si's work include Pain Mechanisms and Treatments (19 papers), Neuroscience and Neuropharmacology Research (15 papers) and Connexins and lens biology (15 papers). Jun‐Qiang Si is often cited by papers focused on Pain Mechanisms and Treatments (19 papers), Neuroscience and Neuropharmacology Research (15 papers) and Connexins and lens biology (15 papers). Jun‐Qiang Si collaborates with scholars based in China, United States and Pakistan. Jun‐Qiang Si's co-authors include Ketao Ma, Xinzhi Li, Li Li, Jiangwen Yin, Hongzhen Hu, Lei Zhao, Zhigang Dai, Li Li, Sheng Wang and Liping Xie and has published in prestigious journals such as PLoS ONE, The Journal of Physiology and Brain Research.

In The Last Decade

Jun‐Qiang Si

81 papers receiving 957 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jun‐Qiang Si China 19 464 292 201 94 80 85 967
Ketao Ma China 19 521 1.1× 243 0.8× 167 0.8× 99 1.1× 97 1.2× 128 1.1k
Tao Song China 22 438 0.9× 309 1.1× 122 0.6× 72 0.8× 82 1.0× 71 1.1k
Nanchang Xie China 23 595 1.3× 454 1.6× 247 1.2× 71 0.8× 168 2.1× 69 1.6k
Haiyan Zhou China 23 473 1.0× 235 0.8× 362 1.8× 71 0.8× 213 2.7× 104 1.6k
Chang‐Yue Gao China 22 361 0.8× 409 1.4× 196 1.0× 123 1.3× 246 3.1× 62 1.4k
Tahar Aboulkassim Canada 18 450 1.0× 320 1.1× 168 0.8× 75 0.8× 200 2.5× 27 1.1k
Regino Perez‐Polo United States 19 361 0.8× 202 0.7× 281 1.4× 149 1.6× 103 1.3× 29 1.1k
Anna A. Zmijewska United States 15 768 1.7× 295 1.0× 233 1.2× 35 0.4× 86 1.1× 24 1.5k
Hiroki Yokoo Japan 23 735 1.6× 205 0.7× 302 1.5× 101 1.1× 102 1.3× 56 1.4k
Ling Chen China 18 455 1.0× 449 1.5× 117 0.6× 166 1.8× 108 1.4× 51 1.2k

Countries citing papers authored by Jun‐Qiang Si

Since Specialization
Citations

This map shows the geographic impact of Jun‐Qiang Si's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun‐Qiang Si with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun‐Qiang Si more than expected).

Fields of papers citing papers by Jun‐Qiang Si

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun‐Qiang Si. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun‐Qiang Si. The network helps show where Jun‐Qiang Si may publish in the future.

Co-authorship network of co-authors of Jun‐Qiang Si

This figure shows the co-authorship network connecting the top 25 collaborators of Jun‐Qiang Si. A scholar is included among the top collaborators of Jun‐Qiang Si based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun‐Qiang Si. Jun‐Qiang Si is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Wang, Ruixue, Lei Jia, Ketao Ma, et al.. (2023). Methyl Ferulic Acid Alleviates Neuropathic Pain by Inhibiting Nox4-induced Ferroptosis in Dorsal Root Ganglia Neurons in Rats. Molecular Neurobiology. 60(6). 3175–3189. 19 indexed citations
4.
Yan, Meijuan, Rui Yang, Ling Chen, et al.. (2021). Adiponectin Attenuates Lipopolysaccharide-induced Apoptosis by Regulating the Cx43/PI3K/AKT Pathway. Frontiers in Pharmacology. 12. 644225–644225. 52 indexed citations
5.
Xiao, Jingjie, Yingying Zhang, Li Li, et al.. (2020). Protective Effects of Adiponectin against Cobalt Chloride‐Induced Apoptosis of Smooth Muscle Cells via cAMP/PKA Pathway. BioMed Research International. 2020(1). 7169348–7169348. 4 indexed citations
6.
Zhang, Meng, et al.. (2019). Type of excitability of DRG neurons in rats with neuropathic pain. 29(11). 1–7. 1 indexed citations
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Li, Peng, Jiangwen Yin, Sheng Wang, et al.. (2019). Isoflurane Post-conditioning Ameliorates Cerebral Ischemia/Reperfusion Injury by Enhancing Angiogenesis Through Activating the Shh/Gli Signaling Pathway in Rats. Frontiers in Neuroscience. 13. 321–321. 32 indexed citations
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Xue, Wenjing, Wei Ji, Yang Wang, et al.. (2018). The Protective Effect of Propofol Against Ischemia–Reperfusion Injury in the Interlobar Arteries: Reduction of Abnormal Cx43 Expression as a Possible Mechanism. Kidney & Blood Pressure Research. 43(5). 1607–1622. 8 indexed citations
11.
Wang, Lijie, Wenwen Zhang, Liang Zhang, et al.. (2017). Association of connexin gene polymorphism with essential hypertension in Kazak and Han Chinese in Xinjiang, China. Journal of Huazhong University of Science and Technology [Medical Sciences]. 37(2). 197–203. 6 indexed citations
12.
Ni, Xin, Liang Zhang, Haichao Zhang, et al.. (2017). Up-regulation of gap junction in peripheral blood T lymphocytes contributes to the inflammatory response in essential hypertension. PLoS ONE. 12(9). e0184773–e0184773. 21 indexed citations
13.
Yin, Jiangwen, Sheng Wang, Di Cui, et al.. (2016). Effects of activin A and its downstream ERK1/2 in oxygen and glucose deprivation after isoflurane-induced postconditioning. Biomedicine & Pharmacotherapy. 84. 535–543. 10 indexed citations
14.
Wang, Sheng, Jiangwen Yin, Zhigang Dai, et al.. (2016). Transforming growth-beta 1 contributes to isoflurane postconditioning against cerebral ischemia–reperfusion injury by regulating the c-Jun N-terminal kinase signaling pathway. Biomedicine & Pharmacotherapy. 78. 280–290. 46 indexed citations
15.
Li, Li, Rui Wang, Ketao Ma, et al.. (2014). Differential effect of calcium-activated potassium and chloride channels on rat basilar artery vasomotion. Journal of Huazhong University of Science and Technology [Medical Sciences]. 34(4). 482–490. 7 indexed citations
16.
Cheng, Hongju, Ketao Ma, Li Li, et al.. (2014). Differential expression of alpha-adrenoceptor subtypes in rat dorsal root ganglion after chronic constriction injury. Journal of Huazhong University of Science and Technology [Medical Sciences]. 34(3). 322–329. 11 indexed citations
17.
Wei, Xiaofei, Lulu Zhang, Shujun Xu, et al.. (2013). CCK-8S increases the firing frequency of CCK-positive neurons and facilitates excitatory synaptic transmission in primary rat hippocampal neurons. Neuroscience Letters. 549. 34–39. 5 indexed citations
18.
Wei, Xiaofei, et al.. (2008). CCK-8S inhibited the NMDA-activated current of cultured hippocampal neuron under normal and ethanol exposure conditions. Neuroscience Letters. 449(1). 34–37. 5 indexed citations
19.
Ma, Ketao, Jun‐Qiang Si, Zhiqin Zhang, et al.. (2006). Modulatory effect of CCK-8S on GABA-induced depolarization from rat dorsal root ganglion. Brain Research. 1121(1). 66–75. 24 indexed citations
20.
Hu, Hongzhen, et al.. (1997). Evidence for the existence of substance P autoreceptor in the membrane of rat dorsal root ganglion neurons. Neuroscience. 77(2). 535–541. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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