Jung-Won Lee

593 total citations
24 papers, 405 citations indexed

About

Jung-Won Lee is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Jung-Won Lee has authored 24 papers receiving a total of 405 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 9 papers in Oncology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Jung-Won Lee's work include Ubiquitin and proteasome pathways (5 papers), Cancer-related Molecular Pathways (5 papers) and Epigenetics and DNA Methylation (3 papers). Jung-Won Lee is often cited by papers focused on Ubiquitin and proteasome pathways (5 papers), Cancer-related Molecular Pathways (5 papers) and Epigenetics and DNA Methylation (3 papers). Jung-Won Lee collaborates with scholars based in South Korea, Singapore and United States. Jung-Won Lee's co-authors include Suk‐Chul Bae, Xin‐Zi Chi, Yoshiaki Ito, Juwon Jang, André J. van Wijnen, Wun‐Jae Kim, Dohun Kim, Minkyu Kim, Beom‐Gi Kim and Mi‐Jeong Jeong and has published in prestigious journals such as Nature Communications, SHILAP Revista de lepidopterología and Cancer Cell.

In The Last Decade

Jung-Won Lee

21 papers receiving 401 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jung-Won Lee South Korea 9 265 123 81 56 43 24 405
Enshun Xu United States 13 312 1.2× 72 0.6× 92 1.1× 91 1.6× 23 0.5× 15 424
Jian Ni China 10 197 0.7× 68 0.6× 24 0.3× 83 1.5× 35 0.8× 29 378
Camille Jacques France 12 380 1.4× 105 0.9× 31 0.4× 165 2.9× 67 1.6× 24 545
Fanjun Cheng China 10 172 0.6× 98 0.8× 14 0.2× 66 1.2× 25 0.6× 27 338
Tulasigeri M. Totiger United States 10 164 0.6× 87 0.7× 39 0.5× 28 0.5× 35 0.8× 22 318
Panpan An China 8 493 1.9× 99 0.8× 72 0.9× 290 5.2× 29 0.7× 9 583
Anh Hua United States 11 117 0.4× 81 0.7× 47 0.6× 29 0.5× 25 0.6× 15 355
Shalu Gupta India 8 264 1.0× 92 0.7× 64 0.8× 37 0.7× 13 0.3× 10 368
Yiting Li China 11 136 0.5× 71 0.6× 23 0.3× 33 0.6× 63 1.5× 31 302

Countries citing papers authored by Jung-Won Lee

Since Specialization
Citations

This map shows the geographic impact of Jung-Won Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jung-Won Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jung-Won Lee more than expected).

Fields of papers citing papers by Jung-Won Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jung-Won Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jung-Won Lee. The network helps show where Jung-Won Lee may publish in the future.

Co-authorship network of co-authors of Jung-Won Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Jung-Won Lee. A scholar is included among the top collaborators of Jung-Won Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jung-Won Lee. Jung-Won Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bae, Suk‐Chul, In‐Jae Oh, Cheol‐Kyu Park, et al.. (2024). Nicotinamide in Combination with EGFR-TKIs for the Treatment of Stage IV Lung Adenocarcinoma with EGFR Mutations: A Randomized Double-Blind (Phase IIb) Trial. Clinical Cancer Research. 30(8). 1478–1487. 3 indexed citations
2.
Matsuo, Junichi, Aashiq Hussain, Jung-Won Lee, et al.. (2024). IQGAP3 signalling mediates intratumoral functional heterogeneity to enhance malignant growth. Gut. 74(3). 364–386. 3 indexed citations
3.
Kim, Min‐Kyu, Xin‐Zi Chi, Eung‐Gook Kim, et al.. (2023). The TGFβ→TAK1→LATS→YAP1 Pathway Regulates the Spatiotemporal Dynamics of YAP1. Molecules and Cells. 46(10). 592–610. 2 indexed citations
4.
Lee, Jung-Won, et al.. (2023). Role of RUNX3 in Restriction Point Regulation. Cells. 12(5). 708–708. 3 indexed citations
5.
Lee, Jung-Won, Ok-Jun Lee, Dohun Kim, et al.. (2023). Runx3 Restoration Regresses K-Ras-Activated Mouse Lung Cancers and Inhibits Recurrence. Cells. 12(20). 2438–2438. 2 indexed citations
6.
Lee, Jung-Won, et al.. (2020). A Study on Performance Improvement of Hyperledger Fabric Through Batched Chaincode. 62.
7.
Lee, Jung-Won, et al.. (2020). Breeding of Lettuce ‘Sambokhacheong’ Tolerant to Tipburn and with Good Yield. Korean Journal of Breeding Science. 52(2). 200–205. 2 indexed citations
8.
Lee, Jung-Won, et al.. (2020). K-Ras-Activated Cells Can Develop into Lung Tumors When Runx3-Mediated Tumor Suppressor Pathways Are Abrogated.. PubMed. 43(10). 889–897. 5 indexed citations
9.
Lee, Jung-Won & Suk‐Chul Bae. (2020). Role of RUNX Family Members in G1 Restriction-Point Regulation.. PubMed. 43(2). 182–187. 3 indexed citations
10.
Lee, Jung-Won, et al.. (2019). Involvement of RUNX and BRD Family Members in Restriction Point.. PubMed. 42(12). 836–839. 7 indexed citations
11.
Lee, Jung-Won, Juwon Jang, Xin‐Zi Chi, et al.. (2019). RUNX3 regulates cell cycle-dependent chromatin dynamics by functioning as a pioneer factor of the restriction-point. Nature Communications. 10(1). 1897–1897. 41 indexed citations
12.
Lee, Jung-Won, André J. van Wijnen, & Suk-Chul Bae. (2017). RUNX3 and p53: How Two Tumor Suppressors Cooperate Against Oncogenic Ras?. Advances in experimental medicine and biology. 962. 321–332. 14 indexed citations
13.
Lee, Jung-Won, et al.. (2017). Comparison of the Thermal Degradation of Heavily Nb-Doped and Normal PZT Thin Films. IEEE Transactions on Ultrasonics Ferroelectrics and Frequency Control. 64(3). 617–622. 11 indexed citations
14.
Lee, Jung-Won, Juwon Jang, Xin‐Zi Chi, et al.. (2013). Runx3 Inactivation Is a Crucial Early Event in the Development of Lung Adenocarcinoma. Cancer Cell. 24(5). 603–616. 102 indexed citations
15.
Lee, Nam‐Jin, Jung-Won Lee, Gi‐Sun Im, et al.. (2011). In vitro and in vivo genotoxic effects of somatic cell nuclear transfer cloned cattle meat. Food and Chemical Toxicology. 49(9). 2273–2278. 4 indexed citations
16.
Lee, Seong-Kon, Beom‐Gi Kim, Taek-Ryoun Kwon, et al.. (2011). Overexpression of the mitogen-activated protein kinase gene OsMAPK33 enhances sensitivity to salt stress in rice (Oryza sativa L.). Journal of Biosciences. 36(1). 139–151. 79 indexed citations
17.
Chi, Xin‐Zi, Jiyeon Kim, Yong‐Hee Lee, et al.. (2009). Runt-Related Transcription Factor RUNX3 Is a Target of MDM2-Mediated Ubiquitination. Cancer Research. 69(20). 8111–8119. 47 indexed citations
18.
Park, Hyunsoo, et al.. (2007). Reconfigurable Front-End System for BD/DVD/CD Recorder. 44. 1–2.
19.
Lee, Eunsook, Jung-Won Lee, Jong Kook Park, et al.. (2006). Clinical pharmacokinetics of oxaliplatin and 5-fluorouracil administered in combination with leucovorin in Korean patients with advanced colorectal cancer. Journal of Cancer Research and Clinical Oncology. 132(5). 320–326. 4 indexed citations
20.
Lee, Jung-Won, et al.. (2006). Anti-tumor activity of heptaplatin in combination with 5-fluorouracil or paclitaxel against human head and neck cancer cells in vitro. Anti-Cancer Drugs. 17(4). 377–384. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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