Julio C. Tapia
- Molecular Biology top 10%
- Oncology top 10%
- Cell Biology top 5%
- Cancer Research top 10%
- Genetics
- Co-authors
- Andrew F. G. QuestLisette LeytonDiego A. RodríguezVicente A. TorresEduardo Silva-PavezIgnacio NiechiRicardo ArmisénMarcelo Antonelli
- Topics
- Protein Kinase Regulation and GTPase Signaling (8 papers)Wnt/β-catenin signaling in development and cancer (7 papers)MicroRNA in disease regulation (5 papers)
- Journals
- Proceedings of the National Academy of SciencesSHILAP Revista de lepidopterologíaMolecular and Cellular Biology
- Partner nations
- ChileUnited StatesArgentina
In The Last Decade
Julio C. Tapia
57 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 98
- Molecular Biology 1.1k
- Oncology 309
- Cell Biology 296
- Cancer Research 282
- Genetics 119
Countries citing papers authored by Julio C. Tapia
This map shows the geographic impact of Julio C. Tapia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julio C. Tapia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julio C. Tapia more than expected).
Fields of papers citing papers by Julio C. Tapia
This network shows the impact of papers produced by Julio C. Tapia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julio C. Tapia. The network helps show where Julio C. Tapia may publish in the future.
Co-authorship network of co-authors of Julio C. Tapia
This figure shows the co-authorship network connecting the top 25 collaborators of Julio C. Tapia. A scholar is included among the top collaborators of Julio C. Tapia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julio C. Tapia. Julio C. Tapia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 2 | |
| 4 | 1 | |
| 5 | 1 | |
| 6 | 6 | |
| 7 | 8 | |
| 8 | 3 | |
| 9 | 7 | |
| 10 | 1 | |
| 11 | 12 | |
| 12 | 40 | |
| 13 | 27 | |
| 14 | 95 | |
| 15 | 8 | |
| 16 | 60 | |
| 17 | 61 | |
| 18 | 9 | |
| 19 | 38 | |
| 20 | 12 |
About Julio C. Tapia
Julio C. Tapia is a scholar working on Cancer Research, Molecular Biology and Cell Biology, having authored 60 papers that have together received 1.6k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (8 papers), Wnt/β-catenin signaling in development and cancer (7 papers) and MicroRNA in disease regulation (5 papers). The work is most often cited by research in Cancer Research (282 citations), Sensory Systems (88 citations) and Cell Biology (296 citations). Julio C. Tapia has collaborated with scholars based in Chile, United States and Argentina. Frequent co-authors include Andrew F. G. Quest, Lisette Leyton, Diego A. Rodríguez, Vicente A. Torres, Eduardo Silva-Pavez, Ignacio Niechi, Ricardo Armisén, Marcelo Antonelli, Mario Galindo and Katherine Marcelain. Their work appears in journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and Molecular and Cellular Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.