Joshua Barry

1.3k total citations
24 papers, 550 citations indexed

About

Joshua Barry is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Joshua Barry has authored 24 papers receiving a total of 550 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Cellular and Molecular Neuroscience, 10 papers in Molecular Biology and 8 papers in Neurology. Recurrent topics in Joshua Barry's work include Neuroscience and Neuropharmacology Research (16 papers), Genetic Neurodegenerative Diseases (9 papers) and Neurological disorders and treatments (8 papers). Joshua Barry is often cited by papers focused on Neuroscience and Neuropharmacology Research (16 papers), Genetic Neurodegenerative Diseases (9 papers) and Neurological disorders and treatments (8 papers). Joshua Barry collaborates with scholars based in United States, France and Italy. Joshua Barry's co-authors include Chen Gu, Yuanzheng Gu, Carlos Cepeda, Michael S. Levine, Michelle Gray, Tara Wood, Peter Jukkola, Garnik Akopian, Thomas G. Wilson and Brian O’Neill and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and The Journal of Physiology.

In The Last Decade

Joshua Barry

23 papers receiving 547 citations

Peers

Joshua Barry

CMN

NEURO

GENET

Francesca Inverardi Italy

Jesús F. Torres-Peraza Spain

Agnieszka Münster‐Wandowski Germany

Alexander Jeans United Kingdom

María Hidalgo‐Figueroa Spain

Asheeta A. Prasad Australia

Chunjie Zhao China

Sónia Paixão Germany

Guanglin Xing United States

Chicheng Sun United States

Francesca Inverardi Italy

Joshua Barry

326 ×0.9

CMN

276 ×0.9

94 ×1.1

NEURO

201 ×2.4

95 ×1.6

GENET

Citations per year, relative to Joshua Barry Joshua Barry (= 1×) peers Francesca Inverardi

Countries citing papers authored by Joshua Barry

Since Specialization

Citations

This map shows the geographic impact of Joshua Barry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joshua Barry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joshua Barry more than expected).

Fields of papers citing papers by Joshua Barry

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joshua Barry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joshua Barry. The network helps show where Joshua Barry may publish in the future.

Co-authorship network of co-authors of Joshua Barry

This figure shows the co-authorship network connecting the top 25 collaborators of Joshua Barry. A scholar is included among the top collaborators of Joshua Barry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joshua Barry. Joshua Barry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cepeda, Carlos, Joshua Barry, & Sandra M. Holley. (2025). Abnormal neurodevelopment predisposes to cortical hyperexcitability in Huntington's disease. Journal of Huntington s Disease. 1161400299–1161400299.

Cepeda, Carlos, et al.. (2024). Corticostriatal maldevelopment in the R6/2 mouse model of juvenile Huntington's disease. Neurobiology of Disease. 204. 106752–106752. 4 indexed citations

Sun, Chao, Christophe Bosc, Joshua Barry, et al.. (2023). A cytoskeleton-membrane interaction conserved in fast-spiking neurons controls movement, emotion, and memory. Molecular Psychiatry. 28(9). 3994–4010. 5 indexed citations

Barry, Joshua, Chenyi Yang, Peyman Golshani, et al.. (2022). Dissociable effects of oxycodone on behavior, calcium transient activity, and excitability of dorsolateral striatal neurons. Frontiers in Neural Circuits. 16. 983323–983323. 2 indexed citations

Barry, Joshua, et al.. (2022). Calcium imaging: A versatile tool to examine Huntington’s disease mechanisms and progression. Frontiers in Neuroscience. 16. 1040113–1040113. 5 indexed citations

Barry, Joshua, et al.. (2021). Synaptic pathology in Huntington's disease: Beyond the corticostriatal pathway. Neurobiology of Disease. 162. 105574–105574. 22 indexed citations

Levinson, S. Rock, Joshua Barry, Michael S. Levine, et al.. (2020). Paroxysmal Discharges in Tissue Slices From Pediatric Epilepsy Surgery Patients: Critical Role of GABAB Receptors in the Generation of Ictal Activity. Frontiers in Cellular Neuroscience. 14. 54–54. 12 indexed citations

Barry, Joshua, Theodore A. Sarafian, Joseph B. Watson, Carlos Cepeda, & Michael S. Levine. (2020). Mechanisms underlying the enhancement of γ‐aminobutyric acid responses in the external globus pallidus of R6/2 Huntington’s disease model mice. Journal of Neuroscience Research. 98(11). 2349–2356. 8 indexed citations

Cepeda, Carlos, S. Rock Levinson, Hiroki Nariai, et al.. (2019). Pathological high frequency oscillations associate with increased GABA synaptic activity in pediatric epilepsy surgery patients. Neurobiology of Disease. 134. 104618–104618. 28 indexed citations

10.

Cepeda, Carlos, et al.. (2019). Developmental origins of cortical hyperexcitability in Huntington's disease: Review and new observations. Journal of Neuroscience Research. 97(12). 1624–1635. 18 indexed citations

11.

Barry, Joshua, Garnik Akopian, Carlos Cepeda, & Michael S. Levine. (2018). Striatal Direct and Indirect Pathway Output Structures Are Differentially Altered in Mouse Models of Huntington's Disease. Journal of Neuroscience. 38(20). 4678–4694. 27 indexed citations

12.

Cepeda, Carlos, S. Rock Levinson, Joshua Barry, et al.. (2018). Cellular antiseizure mechanisms of everolimus in pediatric tuberous sclerosis complex, cortical dysplasia, and non–mTOR‐mediated etiologies. Epilepsia Open. 3(S2). 180–190. 13 indexed citations

13.

Barry, Joshua, Yuanzheng Gu, Peter Jukkola, et al.. (2014). Ankyrin-G Directly Binds to Kinesin-1 to Transport Voltage-Gated Na+ Channels into Axons. Developmental Cell. 28(2). 117–131. 76 indexed citations

14.

Barry, Joshua. (2013). Function and Mechanism of Polarized Targeting of Neuronal Membrane Proteins. OhioLink ETD Center (Ohio Library and Information Network). 1 indexed citations

15.

Gu, Yuanzheng, Joshua Barry, & Chen Gu. (2013). Kv3 channel assembly, trafficking and activity are regulated by zinc through different binding sites. The Journal of Physiology. 591(10). 2491–2507. 14 indexed citations

16.

Barry, Joshua, Yuanzheng Gu, Andrew W. Dangel, et al.. (2013). Activation of conventional kinesin motors in clusters by shaw voltage-gated potassium channels. Journal of Cell Science. 126(Pt 9). 2027–41. 18 indexed citations

17.

Gu, Yuanzheng, et al.. (2011). Alternative Splicing Regulates Kv3.1 Polarized Targeting to Adjust Maximal Spiking Frequency. Journal of Biological Chemistry. 287(3). 1755–1769. 36 indexed citations

18.

Gu, Chen & Joshua Barry. (2011). Function and mechanism of axonal targeting of voltage-sensitive potassium channels. Progress in Neurobiology. 94(2). 115–132. 43 indexed citations

19.

Barry, Joshua, Yuanzheng Gu, & Chen Gu. (2010). Polarized targeting of L1‐CAM regulates axonal and dendritic bundling in vitro. European Journal of Neuroscience. 32(10). 1618–1631. 26 indexed citations

20.

Gu, Yuanzheng, et al.. (2010). Kinesin I Transports Tetramerized Kv3 Channels through the Axon Initial Segment via Direct Binding. Journal of Neuroscience. 30(47). 15987–16001. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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