Joshua A. Chu‐Tan

666 total citations
17 papers, 476 citations indexed

About

Joshua A. Chu‐Tan is a scholar working on Molecular Biology, Ophthalmology and Immunology. According to data from OpenAlex, Joshua A. Chu‐Tan has authored 17 papers receiving a total of 476 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Ophthalmology and 6 papers in Immunology. Recurrent topics in Joshua A. Chu‐Tan's work include Retinal Diseases and Treatments (6 papers), Retinal Development and Disorders (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (5 papers). Joshua A. Chu‐Tan is often cited by papers focused on Retinal Diseases and Treatments (6 papers), Retinal Development and Disorders (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (5 papers). Joshua A. Chu‐Tan collaborates with scholars based in Australia, United States and Ireland. Joshua A. Chu‐Tan's co-authors include Riccardo Natoli, Nilisha Fernando, Jan Provis, Matt Rutar, Krisztina Valter, Michele C. Madigan, Riemke Aggio‐Bruce, Yvette Wooff, Haihan Jiao and Ulrike Schümann and has published in prestigious journals such as Scientific Reports, Frontiers in Immunology and Investigative Ophthalmology & Visual Science.

In The Last Decade

Joshua A. Chu‐Tan

17 papers receiving 472 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Joshua A. Chu‐Tan Australia 12 278 211 104 102 79 17 476
Riemke Aggio‐Bruce Australia 12 296 1.1× 223 1.1× 96 0.9× 91 0.9× 83 1.1× 20 518
Yvette Wooff Australia 10 253 0.9× 168 0.8× 96 0.9× 81 0.8× 77 1.0× 17 427
Changmei Guo China 11 170 0.6× 145 0.7× 42 0.4× 46 0.5× 39 0.5× 24 341
Lvzhen Huang China 13 258 0.9× 234 1.1× 60 0.6× 34 0.3× 28 0.4× 43 460
Tetsuhiro Yasuma Japan 13 287 1.0× 419 2.0× 89 0.9× 57 0.6× 31 0.4× 20 694
Gesa Stute Germany 14 323 1.2× 441 2.1× 66 0.6× 140 1.4× 13 0.2× 24 612
Peng Shang United States 10 245 0.9× 172 0.8× 41 0.4× 67 0.7× 25 0.3× 23 444
Cristina Llombart Spain 8 169 0.6× 192 0.9× 41 0.4× 78 0.8× 21 0.3× 10 390
Ankush Madaan Canada 12 201 0.7× 114 0.5× 102 1.0× 53 0.5× 39 0.5× 14 490
Samih Alqawlaq Canada 7 169 0.6× 125 0.6× 31 0.3× 62 0.6× 18 0.2× 9 320

Countries citing papers authored by Joshua A. Chu‐Tan

Since Specialization
Citations

This map shows the geographic impact of Joshua A. Chu‐Tan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joshua A. Chu‐Tan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joshua A. Chu‐Tan more than expected).

Fields of papers citing papers by Joshua A. Chu‐Tan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joshua A. Chu‐Tan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joshua A. Chu‐Tan. The network helps show where Joshua A. Chu‐Tan may publish in the future.

Co-authorship network of co-authors of Joshua A. Chu‐Tan

This figure shows the co-authorship network connecting the top 25 collaborators of Joshua A. Chu‐Tan. A scholar is included among the top collaborators of Joshua A. Chu‐Tan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joshua A. Chu‐Tan. Joshua A. Chu‐Tan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Chu‐Tan, Joshua A., Yvette Wooff, Tim Karl, et al.. (2023). Voluntary exercise modulates pathways associated with amelioration of retinal degenerative diseases. Frontiers in Physiology. 14. 1116898–1116898. 3 indexed citations
2.
Wooff, Yvette, et al.. (2023). Short exposure to photo-oxidative damage triggers molecular signals indicative of early retinal degeneration. Frontiers in Immunology. 14. 1088654–1088654. 9 indexed citations
3.
Zeng, Shaoxue, Ting Zhang, Yingying Chen, et al.. (2022). Inhibiting the activation of MAPK (ERK1/2) in stressed Müller cells prevents photoreceptor degeneration. Theranostics. 12(15). 6705–6722. 15 indexed citations
4.
Chu‐Tan, Joshua A., et al.. (2021). Running to save sight: The effects of exercise on retinal health and function. Clinical and Experimental Ophthalmology. 50(1). 74–90. 15 indexed citations
5.
Chu‐Tan, Joshua A., Yvette Wooff, Ulrike Schümann, et al.. (2021). Functional microRNA targetome undergoes degeneration-induced shift in the retina. Molecular Neurodegeneration. 16(1). 60–60. 14 indexed citations
6.
7.
Chu‐Tan, Joshua A., Nilisha Fernando, Riemke Aggio‐Bruce, et al.. (2020). A method for gene knockdown in the retina using a lipid-based carrier. PubMed. 26. 48–62. 8 indexed citations
8.
Fernando, Nilisha, Riemke Aggio‐Bruce, Yvette Wooff, et al.. (2020). MicroRNA-223 Regulates Retinal Function and Inflammation in the Healthy and Degenerating Retina. Frontiers in Cell and Developmental Biology. 8. 516–516. 20 indexed citations
9.
Wooff, Yvette, Nilisha Fernando, Riemke Aggio‐Bruce, et al.. (2020). Caspase-1-dependent inflammasomes mediate photoreceptor cell death in photo-oxidative damage-induced retinal degeneration. Scientific Reports. 10(1). 2263–2263. 35 indexed citations
10.
Aggio‐Bruce, Riemke, et al.. (2020). Inhibition of microRNA-155 Protects Retinal Function Through Attenuation of Inflammation in Retinal Degeneration. Molecular Neurobiology. 58(2). 835–854. 18 indexed citations
11.
Natoli, Riccardo, Nilisha Fernando, Haihan Jiao, et al.. (2018). Obesity-induced metabolic disturbance drives oxidative stress and complement activation in the retinal environment.. PubMed. 24. 201–217. 19 indexed citations
12.
Fernando, Nilisha, Yvette Wooff, Riemke Aggio‐Bruce, et al.. (2018). Photoreceptor Survival Is Regulated by GSTO1-1 in the Degenerating Retina. Investigative Ophthalmology & Visual Science. 59(11). 4362–4362. 10 indexed citations
13.
Chu‐Tan, Joshua A., Matt Rutar, Riemke Aggio‐Bruce, et al.. (2018). MicroRNA-124 Dysregulation Is Associated With Retinal Inflammation and Photoreceptor Death in the Degenerating Retina. Investigative Ophthalmology & Visual Science. 59(10). 4094–4094. 52 indexed citations
14.
Natoli, Riccardo, Nilisha Fernando, Haihan Jiao, et al.. (2017). Retinal Macrophages Synthesize C3 and Activate Complement in AMD and in Models of Focal Retinal Degeneration. Investigative Ophthalmology & Visual Science. 58(7). 2977–2977. 75 indexed citations
15.
Natoli, Riccardo, Nilisha Fernando, Michele C. Madigan, et al.. (2017). Microglia-derived IL-1β promotes chemokine expression by Müller cells and RPE in focal retinal degeneration. Molecular Neurodegeneration. 12(1). 31–31. 109 indexed citations
16.
Chu‐Tan, Joshua A., Matt Rutar, Lauren Howitt, et al.. (2016). Efficacy of 670 nm Light Therapy to Protect against Photoreceptor Cell Death Is Dependent on the Severity of Damage. International Journal of Photoenergy. 2016. 1–12. 11 indexed citations
17.
Natoli, Riccardo, Matt Rutar, Yen‐Zhen Lu, et al.. (2016). The Role of Pyruvate in Protecting 661W Photoreceptor-Like Cells Against Light-Induced Cell Death. Current Eye Research. 41(11). 1473–1481. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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