Joseph P. Garay

1.9k total citations
16 papers, 978 citations indexed

About

Joseph P. Garay is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Joseph P. Garay has authored 16 papers receiving a total of 978 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 6 papers in Oncology and 4 papers in Genetics. Recurrent topics in Joseph P. Garay's work include PI3K/AKT/mTOR signaling in cancer (4 papers), CRISPR and Genetic Engineering (3 papers) and Estrogen and related hormone effects (3 papers). Joseph P. Garay is often cited by papers focused on PI3K/AKT/mTOR signaling in cancer (4 papers), CRISPR and Genetic Engineering (3 papers) and Estrogen and related hormone effects (3 papers). Joseph P. Garay collaborates with scholars based in United States, Netherlands and Spain. Joseph P. Garay's co-authors include Ben Ho Park, Josh Lauring, Abde M. Abukhdeir, Hiroyuki Konishi, John P. Gustin, Charles M. Perou, Aatish Thennavan, Siyao Liu, Bedri Karakas and Yuko Konishi and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Blood.

In The Last Decade

Joseph P. Garay

16 papers receiving 961 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Joseph P. Garay United States	12	567	467	235	223	173	16	978
Rajita Vatapalli United States	13	807 1.4×	370 0.8×	185 0.8×	236 1.1×	180 1.0×	21	1.1k
Davide Pellacani United Kingdom	19	513 0.9×	352 0.8×	261 1.1×	135 0.6×	216 1.2×	34	907
Ina Klebba United States	12	745 1.3×	814 1.7×	311 1.3×	256 1.1×	136 0.8×	19	1.4k
Sharon K. Huang United States	17	902 1.6×	433 0.9×	450 1.9×	298 1.3×	114 0.7×	23	1.3k
Véronique Scott France	18	492 0.9×	509 1.1×	338 1.4×	147 0.7×	366 2.1×	39	1.0k
Frank P. Vendetti United States	11	843 1.5×	411 0.9×	162 0.7×	195 0.9×	120 0.7×	18	1.1k
Catherine Andrieu France	10	427 0.8×	434 0.9×	382 1.6×	134 0.6×	138 0.8×	11	916
Shaveta Vinayak United States	15	429 0.8×	790 1.7×	351 1.5×	193 0.9×	239 1.4×	45	1.2k
Elizabeth Ruth Plummer United Kingdom	19	836 1.5×	961 2.1×	181 0.8×	137 0.6×	116 0.7×	69	1.3k
Samuel W. Brady United States	14	608 1.1×	588 1.3×	187 0.8×	93 0.4×	188 1.1×	25	1.1k

Countries citing papers authored by Joseph P. Garay

Since Specialization

Citations

This map shows the geographic impact of Joseph P. Garay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph P. Garay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph P. Garay more than expected).

Fields of papers citing papers by Joseph P. Garay

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph P. Garay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph P. Garay. The network helps show where Joseph P. Garay may publish in the future.

Co-authorship network of co-authors of Joseph P. Garay

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph P. Garay. A scholar is included among the top collaborators of Joseph P. Garay based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph P. Garay. Joseph P. Garay is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Liu, Moqing, Joseph P. Garay, Alexander T. Fields, et al.. (2023). Pilot study of frozen platelet extracellular vesicles as a therapeutic agent in hemorrhagic shock in rats. The Journal of Trauma: Injury, Infection, and Critical Care. 96(3). 364–370. 3 indexed citations

Garay, Joseph P., Rebecca Smith, Kaylyn L. Devlin, et al.. (2021). Sensitivity to targeted therapy differs between HER2-amplified breast cancer cells harboring kinase and helical domain mutations in PIK3CA. Breast Cancer Research. 23(1). 81–81. 8 indexed citations

Liu, Siyao, Aatish Thennavan, Joseph P. Garay, J. S. Marron, & Charles M. Perou. (2021). MultiK: an automated tool to determine optimal cluster numbers in single-cell RNA sequencing data. Genome biology. 22(1). 232–232. 20 indexed citations

Shen, Li, et al.. (2021). CRISPR‐based knock‐in mutagenesis of the pioneer transcription factor FOXA1: optimization of strategies for multi‐allelic proteins in cancer cells. FEBS Open Bio. 11(6). 1537–1551. 3 indexed citations

Hollern, Daniel P., Nuo Xu, Aatish Thennavan, et al.. (2019). B Cells and T Follicular Helper Cells Mediate Response to Checkpoint Inhibitors in High Mutation Burden Mouse Models of Breast Cancer. Cell. 179(5). 1191–1206.e21. 307 indexed citations

Wong, Hong Yuen, Grace M. Wang, Sarah Croessmann, et al.. (2015). TMSB4Yis a candidate tumor suppressor on the Y chromosome and is deleted in male breast cancer. Oncotarget. 6(42). 44927–44940. 29 indexed citations

Mohseni, Morassa, Justin Cidado, Sarah Croessmann, et al.. (2014). MACROD2 overexpression mediates estrogen independent growth and tamoxifen resistance in breast cancers. Proceedings of the National Academy of Sciences. 111(49). 17606–17611. 52 indexed citations

Garay, Joseph P. & Joe W. Gray. (2012). Omics and therapy – A basis for precision medicine. Molecular Oncology. 6(2). 128–139. 30 indexed citations

Garay, Joseph P. & Ben Ho Park. (2012). Androgen receptor as a targeted therapy for breast cancer.. PubMed. 2(4). 434–45. 77 indexed citations

10.

Higgins, Michaela J., Julia A. Beaver, Hong Yuen Wong, et al.. (2011). PIK3CAmutations and EGFR overexpression predict for lithium sensitivity in human breast epithelial cells. Cancer Biology & Therapy. 11(3). 358–367. 7 indexed citations

11.

Lauring, Josh, David Cosgrove, Stefani C. Fontana, et al.. (2010). Knock in of the AKT1 E17K mutation in human breast epithelial cells does not recapitulate oncogenic PIK3CA mutations. Oncogene. 29(16). 2337–2345. 44 indexed citations

12.

Gustin, John P., Bedri Karakas, Michele B. Weiss, et al.. (2009). Knockin of mutant PIK3CA activates multiple oncogenic pathways. Proceedings of the National Academy of Sciences. 106(8). 2835–2840. 120 indexed citations

13.

Konishi, Hiroyuki, Bedri Karakas, Abde M. Abukhdeir, et al.. (2007). Knock-in of Mutant K- ras in Nontumorigenic Human Epithelial Cells as a New Model for Studying K- ras –Mediated Transformation. Cancer Research. 67(18). 8460–8467. 74 indexed citations

14.

Lauring, Josh, Abde M. Abukhdeir, Hiroyuki Konishi, et al.. (2007). The multiple myeloma–associated MMSET gene contributes to cellular adhesion, clonogenic growth, and tumorigenicity. Blood. 111(2). 856–864. 117 indexed citations

15.

Konishi, Hiroyuki, Josh Lauring, Joseph P. Garay, et al.. (2007). A PCR-based high-throughput screen with multiround sample pooling: application to somatic cell gene targeting. Nature Protocols. 2(11). 2865–2874. 17 indexed citations

16.

Abukhdeir, Abde M., Michele Vítolo, Pedram Argani, et al.. (2007). Tamoxifen-stimulated growth of breast cancer due to p21 loss. Proceedings of the National Academy of Sciences. 105(1). 288–293. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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