José Pavı́a

1.4k total citations
49 papers, 1.0k citations indexed

About

José Pavı́a is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Biochemistry. According to data from OpenAlex, José Pavı́a has authored 49 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 9 papers in Biochemistry. Recurrent topics in José Pavı́a's work include Receptor Mechanisms and Signaling (12 papers), Neuroscience and Neuropharmacology Research (10 papers) and Blood transfusion and management (9 papers). José Pavı́a is often cited by papers focused on Receptor Mechanisms and Signaling (12 papers), Neuroscience and Neuropharmacology Research (10 papers) and Blood transfusion and management (9 papers). José Pavı́a collaborates with scholars based in Spain, Italy and United States. José Pavı́a's co-authors include Manuel Múñoz, Elisa Martín‐Montañez, Susana Gómez‐Ramírez, Felipe Sánchez de la Cuesta, Michael Auerbach, Aryeh Shander, Edward C. Hulme, Federica Boraldi, María García‐Fernández and Francisco Sendra‐Portero and has published in prestigious journals such as Free Radical Biology and Medicine, European Journal of Biochemistry and Biochemical Pharmacology.

In The Last Decade

José Pavı́a

46 papers receiving 986 citations

Peers

José Pavı́a
José Pavı́a
Citations per year, relative to José Pavı́a José Pavı́a (= 1×) peers Maithili Sashindranath

Countries citing papers authored by José Pavı́a

Since Specialization
Citations

This map shows the geographic impact of José Pavı́a's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José Pavı́a with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José Pavı́a more than expected).

Fields of papers citing papers by José Pavı́a

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José Pavı́a. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José Pavı́a. The network helps show where José Pavı́a may publish in the future.

Co-authorship network of co-authors of José Pavı́a

This figure shows the co-authorship network connecting the top 25 collaborators of José Pavı́a. A scholar is included among the top collaborators of José Pavı́a based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José Pavı́a. José Pavı́a is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Baños, Josep-Eladı́, Encarnación Blanco‐Reina, Inmaculada Bellido-Estévez, et al.. (2024). Beyond lectures and practical courses: Teaching pharmacology using imaginative pedagogical tools. Pharmacological Research. 202. 107130–107130. 9 indexed citations
3.
Romero‐Zerbo, Silvana‐Yanina, Silvia Claros, Federica Boraldi, et al.. (2024). New molecular mechanisms to explain the neuroprotective effects of insulin-like growth factor II in a cellular model of Parkinson’s disease. Journal of Advanced Research. 67. 349–359. 8 indexed citations
4.
Domínguez-Pinos, Dolores, et al.. (2023). Improving Oral Presentation Skills for Radiology Residents through Clinical Session Meetings in the Virtual World Second Life. International Journal of Environmental Research and Public Health. 20(6). 4738–4738. 8 indexed citations
5.
Martín‐Montañez, Elisa, David Ladrón de Guevara‐Miranda, Carmelo Millón, et al.. (2021). Insulin-like growth factor II prevents oxidative and neuronal damage in cellular and mice models of Parkinson's disease. Redox Biology. 46. 102095–102095. 22 indexed citations
6.
Claros, Silvia, Mauro Martinelli, Federica Boraldi, et al.. (2021). Impact of Glucocorticoid on a Cellular Model of Parkinson’s Disease: Oxidative Stress and Mitochondrial Function. Brain Sciences. 11(8). 1106–1106. 9 indexed citations
7.
Martín‐Montañez, Elisa, Federica Boraldi, Silvia Claros, et al.. (2020). Neuronal Metabolism and Neuroprotection: Neuroprotective Effect of Fingolimod on Menadione-Induced Mitochondrial Damage. Cells. 10(1). 34–34. 9 indexed citations
8.
Pavı́a, José, et al.. (2018). Posibilidades del entorno virtual tridimensional Second Life® para la formación en radiología. Radiología. 60(4). 273–279. 15 indexed citations
9.
Martín‐Montañez, Elisa, Carmelo Millón, Federica Boraldi, et al.. (2017). IGF-II promotes neuroprotection and neuroplasticity recovery in a long-lasting model of oxidative damage induced by glucocorticoids. Redox Biology. 13. 69–81. 47 indexed citations
10.
Suardíaz, Margarita, Diego Clemente, José Pavı́a, et al.. (2016). Recombinant soluble IFN receptor (sIFNAR2) exhibits intrinsic therapeutic efficacy in a murine model of Multiple Sclerosis. Neuropharmacology. 110(Pt A). 480–492. 5 indexed citations
11.
Múñoz, Manuel, Susana Gómez‐Ramírez, Aryeh Shander, et al.. (2015). ‘Fit to fly’: overcoming barriers to preoperative haemoglobin optimization in surgical patients. British Journal of Anaesthesia. 115(1). 15–24. 158 indexed citations
12.
Gómez‐Ramírez, Susana, et al.. (2015). “Patient blood management” en cirugía ortopédica. Revista Española de Cirugía Ortopédica y Traumatología. 59(3). 137–149. 24 indexed citations
13.
Martín‐Montañez, Elisa, Maria Acevedo-Calado, Juan F. López‐Téllez, et al.. (2010). Regulator of G-protein signaling 14 protein modulates Ca2+ influx through Cav1 channels. Neuroreport. 21(16). 1034–1039. 3 indexed citations
14.
Montiel, Mercedes, José Pavı́a, Santo Marsigliante, & Eugenio Gutiérrez Jiménez. (2001). Activation of muscarinic acetylcholine receptors induces Ca2+ mobilization in FRT cells. Cellular Signalling. 13(3). 207–212. 11 indexed citations
15.
Cruz, J.P. De La, José Pavı́a, José Antonio González Correa, P. Ortiz, & Felipe Sánchez de la Cuesta. (2000). Effects of chronic administration of S-adenosyl-l-methionine on brain oxidative stress in rats. Naunyn-Schmiedeberg s Archives of Pharmacology. 361(1). 47–52. 22 indexed citations
16.
Pavı́a, José, et al.. (1998). Alzheimer's disease: relationship between muscarinic cholinergic receptors, β‐amyloid and tau proteins. Fundamental and Clinical Pharmacology. 12(5). 473–481. 41 indexed citations
17.
Lu, Zhi-Liang, C.A.M. Curtis, Philip Jones, José Pavı́a, & Edward C. Hulme. (1997). The Role of the Aspartate-Arginine-Tyrosine Triad in the m1 Muscarinic Receptor: Mutations of Aspartate 122 and Tyrosine 124 Decrease Receptor Expression but Do Not Abolish Signaling. Molecular Pharmacology. 51(2). 234–241. 77 indexed citations
18.
Muñoz, Manuel J., et al.. (1996). Angiotensin ii receptor in human placental syncytiotrophoblast plasma membranes. Life Sciences. 58(11). 877–882. 9 indexed citations
19.
Crespo, Francisco Martos, et al.. (1992). Muscarinic receptor subtypes in human and rat colon smooth muscle. Biochemical Pharmacology. 43(11). 2413–2419. 52 indexed citations
20.
Cruz, J.P. De La, José Pavı́a, Juan Antonio García-Arnés, & Felipe Sánchez de la Cuesta. (1988). Effects of triflusal and acetylsalicylic acid on platelet aggregation in whole blood of diabetic patients. European Journal Of Haematology. 40(3). 232–236. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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