José M. González‐Navajas

4.9k total citations · 1 hit paper
56 papers, 2.8k citations indexed

About

José M. González‐Navajas is a scholar working on Epidemiology, Hepatology and Immunology. According to data from OpenAlex, José M. González‐Navajas has authored 56 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Epidemiology, 26 papers in Hepatology and 21 papers in Immunology. Recurrent topics in José M. González‐Navajas's work include Liver Disease and Transplantation (25 papers), Liver Disease Diagnosis and Treatment (21 papers) and Immune Response and Inflammation (10 papers). José M. González‐Navajas is often cited by papers focused on Liver Disease and Transplantation (25 papers), Liver Disease Diagnosis and Treatment (21 papers) and Immune Response and Inflammation (10 papers). José M. González‐Navajas collaborates with scholars based in Spain, United States and Switzerland. José M. González‐Navajas's co-authors include Jong‐Dae Lee, Eyal Raz, Michael David, Rubén Francés, Pedro Zapater, José M. Such, Beatriz Lozano‐Ruiz, Pablo Bellot, Miguel Pérez‐Mateo and Carlos Muñoz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

José M. González‐Navajas

56 papers receiving 2.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Immunomodulatory functions of type I interferons

2012 786 citations José M. González‐Navajas, Jong‐Dae Lee et al. Nature reviews. Immunology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
José M. González‐Navajas Spain	28	1.2k	934	858	529	402	56	2.8k
Jiaren Sun United States	32	1.4k 1.2×	1.1k 1.2×	918 1.1×	308 0.6×	233 0.6×	76	3.5k
Stefania Crotta United Kingdom	24	1.6k 1.4×	843 0.9×	1.0k 1.2×	517 1.0×	267 0.7×	41	3.2k
Hisami Watanabe Japan	32	2.4k 2.1×	782 0.8×	654 0.8×	184 0.3×	470 1.2×	129	4.0k
Gabriele Sass United States	32	505 0.4×	1.5k 1.6×	611 0.7×	377 0.7×	267 0.7×	88	3.0k
Stephen H. Gregory United States	29	1.3k 1.1×	732 0.8×	587 0.7×	310 0.6×	192 0.5×	75	2.6k
Ping An United States	31	636 0.5×	701 0.8×	755 0.9×	386 0.7×	224 0.6×	76	2.5k
Andreas Lügering Germany	28	960 0.8×	596 0.6×	697 0.8×	232 0.4×	250 0.6×	58	2.6k
G. J. Bagby United States	20	1.6k 1.3×	572 0.6×	833 1.0×	359 0.7×	261 0.6×	36	3.3k
Kathryn E. Tanaka United States	24	1.0k 0.9×	894 1.0×	1.8k 2.1×	332 0.6×	440 1.1×	41	3.7k
Kazushi Sugimoto Japan	34	739 0.6×	1.9k 2.0×	1.1k 1.3×	1.2k 2.3×	647 1.6×	134	4.0k

Countries citing papers authored by José M. González‐Navajas

Since Specialization

Citations

This map shows the geographic impact of José M. González‐Navajas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José M. González‐Navajas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José M. González‐Navajas more than expected).

Fields of papers citing papers by José M. González‐Navajas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José M. González‐Navajas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José M. González‐Navajas. The network helps show where José M. González‐Navajas may publish in the future.

Co-authorship network of co-authors of José M. González‐Navajas

This figure shows the co-authorship network connecting the top 25 collaborators of José M. González‐Navajas. A scholar is included among the top collaborators of José M. González‐Navajas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José M. González‐Navajas. José M. González‐Navajas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Barragán, Eva, Esperanza Such, Cristina Gil, et al.. (2022). Identification of Leukemia-Associated Immunophenotypes by Databaseguided Flow Cytometry Provides a Highly Sensitive and Reproducible Strategy for the Study of Measurable Residual Disease in Acute Myeloblastic Leukemia. Cancers. 14(16). 4010–4010. 9 indexed citations

Lozano‐Ruiz, Beatriz, Ana M. Aransay, Ana R. Cortázar, et al.. (2022). Absent in Melanoma 2 (AIM2) Regulates the Stability of Regulatory T Cells. International Journal of Molecular Sciences. 23(4). 2230–2230. 12 indexed citations

Lozano, Beatriz Rodríguez, Pablo Bellot, Maria M. Rodríguez, et al.. (2020). Functionality of beta-adrenergic receptors in patients with cirrhosis treated chronically with non-selective beta-blockers. Hepatology International. 14(5). 858–868. 3 indexed citations

Lee, Jihyung, Junyan Zhang, Young‐Jun Chung, et al.. (2020). Inhibition of IRF4 in dendritic cells by PRR-independent and -dependent signals inhibit Th2 and promote Th17 responses. eLife. 9. 27 indexed citations

Caparrós, Esther, Oriol Juanola, Isabel Gómez‐Hurtado, et al.. (2020). Liver Sinusoidal Endothelial Cells Contribute to Hepatic Antigen-Presenting Cell Function and Th17 Expansion in Cirrhosis. Cells. 9(5). 1227–1227. 14 indexed citations

Lozano‐Ruiz, Beatriz, Gloria Peiró, Isabel Gómez‐Hurtado, et al.. (2018). AIM2 deficiency reduces the development of hepatocellular carcinoma in mice. International Journal of Cancer. 143(11). 2997–3007. 40 indexed citations

García‐Martinez, Irma, Rubén Francés, José M. González‐Navajas, et al.. (2018). Treatment with non‐selective beta‐blockers affects the systemic inflammatory response to bacterialDNAin patients with cirrhosis. Liver International. 38(12). 2219–2227. 16 indexed citations

Caparrós, Esther, et al.. (2018). LSECtin and CD206 participate in the regulation of the hepatic inflammatory response to E coli in a model of cirrhosis. Journal of Hepatology. 68. S472–S473. 1 indexed citations

de‐Madaria, Enrique, Beatriz Lozano‐Ruiz, José M. Such, et al.. (2017). The expression and activation of the AIM2 inflammasome correlates with inflammation and disease severity in patients with acute pancreatitis. Pancreatology. 17(3). 364–371. 25 indexed citations

10.

Gómez‐Hurtado, Isabel, Irma García‐Martinez, Pedro Zapater, et al.. (2017). Norfloxacin is more effective than Rifaximin in avoiding bacterial translocation in an animal model of cirrhosis. Liver International. 38(2). 295–302. 14 indexed citations

11.

Ampuero, Javier, Pablo Bellot, Rocío Gallego‐Durán, et al.. (2016). Lactulose reduces bacterial DNA translocation, which worsens neurocognitive shape in cirrhotic patients with minimal hepatic encephalopathy. Liver International. 37(2). 212–223. 30 indexed citations

12.

Bertin, Samuel, Beatriz Lozano‐Ruiz, Irma García‐Martinez, et al.. (2014). Dual-specificity phosphatase 6 regulates CD4+ T-cell functions and restrains spontaneous colitis in IL-10-deficient mice. Mucosal Immunology. 8(3). 505–515. 40 indexed citations

13.

Juanola, Oriol, Ana Gutiérrez, Laura Sempere, et al.. (2014). Anti-TNF-alpha loss of response is associated with a decreased percentage of FoxP3+ T cells and a variant NOD2 genotype in patients with Crohn’s disease. Journal of Gastroenterology. 50(7). 758–768. 15 indexed citations

14.

Lee, Shee Eun, Xiangli Li, Jong‐Dae Lee, et al.. (2012). Type I Interferons Maintain Foxp3 Expression and T-Regulatory Cell Functions Under Inflammatory Conditions in Mice. Gastroenterology. 143(1). 145–154. 64 indexed citations

15.

Zapater, Pedro, Isabel Gómez‐Hurtado, Gloria Peiró, et al.. (2012). Beta-Adrenergic Receptor 1 Selective Antagonism Inhibits Norepinephrine-Mediated TNF-Alpha Downregulation in Experimental Liver Cirrhosis. PLoS ONE. 7(8). e43371–e43371. 13 indexed citations

16.

González‐Navajas, José M., Jong‐Dae Lee, Michael David, & Eyal Raz. (2012). Immunomodulatory functions of type I interferons. Nature reviews. Immunology. 12(2). 125–135. 786 indexed citations breakdown →

17.

Lee, Sung Hee, Lili Hu, José M. González‐Navajas, et al.. (2010). ERK activation drives intestinal tumorigenesis in Apcmin/+ mice. Nature Medicine. 16(6). 665–670. 168 indexed citations

18.

González‐Navajas, José M., Sean Fine, Jason Law, et al.. (2010). TLR4 signaling in effector CD4+ T cells regulates TCR activation and experimental colitis in mice. Journal of Clinical Investigation. 120(2). 570–581. 114 indexed citations

19.

Lee, Jong‐Dae, Kazumichi Abe, Kyoko Katakura, José M. González‐Navajas, & Eyal Raz. (2008). THE PROTECTIVE EFFECTS OF TYPE-1 INTERFERON IN MODELS OF INTESTINAL INFLAMMATION. Advances in experimental medicine and biology. 633. 1–6. 2 indexed citations

20.

Francés, Rubén, Maite Chiva, Elisabet Sánchez, et al.. (2006). Bacterial translocation is downregulated by anti-TNF-α monoclonal antibody administration in rats with cirrhosis and ascites. Journal of Hepatology. 46(5). 797–803. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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