José A. Martina

9.7k total citations · 3 hit papers
53 papers, 5.5k citations indexed

About

José A. Martina is a scholar working on Molecular Biology, Cell Biology and Immunology. According to data from OpenAlex, José A. Martina has authored 53 papers receiving a total of 5.5k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 26 papers in Cell Biology and 12 papers in Immunology. Recurrent topics in José A. Martina's work include Cellular transport and secretion (16 papers), Glycosylation and Glycoproteins Research (10 papers) and Autophagy in Disease and Therapy (10 papers). José A. Martina is often cited by papers focused on Cellular transport and secretion (16 papers), Glycosylation and Glycoproteins Research (10 papers) and Autophagy in Disease and Therapy (10 papers). José A. Martina collaborates with scholars based in United States, Argentina and Netherlands. José A. Martina's co-authors include Rosa Puertollano, Yong Chen, Marjan Guček, Heba I. Diab, Juan S. Bonifacino, Owen A. Brady, Hugo J. F. Maccioni, Jeong‐A Lim, José L. Daniotti and Nina Raben and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

José A. Martina

52 papers receiving 5.4k citations

Hit Papers

3 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

MTORC1 functions as a transcriptional regulator of autophagy by preventing nuclear transport of TFEB

2012 1.0k citations José A. Martina, Yong Chen et al. Autophagy profile →
Transcriptional Activation of Lysosomal Exocytosis Promotes Cellular Clearance

2011 570 citations Diego L. Medina, Alessandro Fraldi et al. Developmental Cell profile →
The Nutrient-Responsive Transcription Factor TFE3 Promotes Autophagy, Lysosomal Biogenesis, and Clearance of Cellular Debris

2014 503 citations José A. Martina, Heba I. Diab et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
José A. Martina United States	33	2.8k	2.2k	1.9k	1.1k	1.0k	53	5.5k
Chiara Di Malta Italy	15	2.5k 0.9×	3.3k 1.5×	1.3k 0.7×	1.1k 1.0×	1.3k 1.3×	19	5.6k
Francesco Vetrini United States	13	2.5k 0.9×	3.1k 1.4×	1.1k 0.6×	885 0.8×	977 1.0×	31	5.3k
Ian G. Ganley United Kingdom	40	4.1k 1.5×	4.4k 2.0×	1.6k 0.8×	950 0.9×	1.2k 1.1×	80	7.6k
Junji Ezaki Japan	24	2.2k 0.8×	3.0k 1.4×	1.3k 0.7×	522 0.5×	1.1k 1.0×	41	4.8k
Tuong Huynh United States	19	3.7k 1.3×	3.8k 1.7×	1.2k 0.6×	937 0.9×	1.2k 1.1×	19	6.7k
Ryan C. Russell United States	25	3.4k 1.2×	2.8k 1.3×	1.3k 0.7×	531 0.5×	602 0.6×	36	5.8k
Elena Shvets Israel	18	2.4k 0.9×	3.5k 1.6×	1.5k 0.7×	622 0.6×	583 0.6×	20	5.0k
Chantal Bauvy France	31	3.2k 1.1×	3.4k 1.6×	1.1k 0.6×	608 0.6×	545 0.5×	54	5.7k
Farah H. Siddiqi United Kingdom	16	1.9k 0.7×	2.5k 1.1×	1.1k 0.6×	542 0.5×	732 0.7×	23	4.3k
Maria Høyer-Hansen Denmark	22	2.5k 0.9×	2.4k 1.1×	1.1k 0.6×	663 0.6×	369 0.4×	27	4.5k

Countries citing papers authored by José A. Martina

Since Specialization

Citations

This map shows the geographic impact of José A. Martina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José A. Martina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José A. Martina more than expected).

Fields of papers citing papers by José A. Martina

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José A. Martina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José A. Martina. The network helps show where José A. Martina may publish in the future.

Co-authorship network of co-authors of José A. Martina

This figure shows the co-authorship network connecting the top 25 collaborators of José A. Martina. A scholar is included among the top collaborators of José A. Martina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José A. Martina. José A. Martina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

López‐Haber, Cynthia, et al.. (2025). The lysosomal carrier SLC29A3 supports antibacterial signaling, and promotes autophagy by activating TRPML1 in murine dendritic cells. Proceedings of the National Academy of Sciences. 122(48). e2511539122–e2511539122.

Gómez‐Orte, Eva, Xavier González, José A. Martina, et al.. (2024). Regulation of Caenorhabditis elegans HLH-30 subcellular localization dynamics: Evidence for a redox-dependent mechanism. Free Radical Biology and Medicine. 223. 369–383. 2 indexed citations

Martina, José A., et al.. (2022). The FACT complex facilitates expression of lysosomal and antioxidant genes through binding to TFEB and TFE3. Autophagy. 18(10). 2333–2349. 19 indexed citations

Chen, Hui, Mindy Smith, Jasmin Herz, et al.. (2021). The role of protease-activated receptor 1 signaling in CD8 T cell effector functions. iScience. 24(11). 103387–103387. 12 indexed citations

Martina, José A., Nina Raben, & Rosa Puertollano. (2020). SnapShot: Lysosomal Storage Diseases. Cell. 180(3). 602–602.e1. 28 indexed citations

Martina, José A., Eva Gómez‐Orte, José Antonio Bárcena, et al.. (2020). A conserved cysteine‐based redox mechanism sustains TFEB/HLH‐30 activity under persistent stress. The EMBO Journal. 40(3). e105793–e105793. 31 indexed citations

El–Houjeiri, Leeanna, Elite Possik, Tarika Vijayaraghavan, et al.. (2019). The Transcription Factors TFEB and TFE3 Link the FLCN-AMPK Signaling Axis to Innate Immune Response and Pathogen Resistance. Cell Reports. 26(13). 3613–3628.e6. 81 indexed citations

Martina, José A., Heba I. Diab, Owen A. Brady, & Rosa Puertollano. (2016). TFEB and TFE 3 are novel components of the integrated stress response. The EMBO Journal. 35(5). 479–495. 231 indexed citations

Wada, Shogo, Michael D. Neinast, Cholsoon Jang, et al.. (2016). The tumor suppressor FLCN mediates an alternate mTOR pathway to regulate browning of adipose tissue. Genes & Development. 30(22). 2551–2564. 91 indexed citations

10.

Martina, José A. & Rosa Puertollano. (2013). RRAG GTPases link nutrient availability to gene expression, autophagy and lysosomal biogenesis. Autophagy. 9(6). 928–930. 14 indexed citations

11.

Martina, José A., Yong Chen, Marjan Guček, & Rosa Puertollano. (2012). MTORC1 functions as a transcriptional regulator of autophagy by preventing nuclear transport of TFEB. Autophagy. 8(6). 903–914. 1024 indexed citations breakdown →

12.

Vergarajauregui, Silvia, José A. Martina, & Rosa Puertollano. (2011). LAPTMs regulate lysosomal function and interact with mucolipin 1: new clues for understanding mucolipidosis type IV. Journal of Cell Science. 124(3). 459–468. 44 indexed citations

13.

Martina, José A., Xufeng Wu, Marta Catálfamo, et al.. (2011). Imaging of lytic granule exocytosis in CD8+ cytotoxic T lymphocytes reveals a modified form of full fusion. Cellular Immunology. 271(2). 267–279. 15 indexed citations

14.

Medina, Diego L., Alessandro Fraldi, Valentina Bouchè, et al.. (2011). Transcriptional Activation of Lysosomal Exocytosis Promotes Cellular Clearance. Developmental Cell. 21(3). 421–430. 570 indexed citations breakdown →

15.

Crespo, Pilar M., et al.. (2006). Modulation of GalT1 and SialT1 Sub-Golgi Localization by SialT2 Expression Reveals an Organellar Level of Glycolipid Synthesis Control. Journal of Biological Chemistry. 281(43). 32852–32860. 30 indexed citations

16.

Gwynn, Babette, José A. Martina, Juan S. Bonifacino, et al.. (2004). Reduced pigmentation (rp), a mouse model of Hermansky-Pudlak syndrome, encodes a novel component of the BLOC-1 complex. Blood. 104(10). 3181–3189. 45 indexed citations

17.

Martina, José A., Kengo Moriyama, & Juan S. Bonifacino. (2003). BLOC-3, a Protein Complex Containing the Hermansky-Pudlak Syndrome Gene Products HPS1 and HPS4. Journal of Biological Chemistry. 278(31). 29376–29384. 102 indexed citations

18.

Daniotti, José L., et al.. (1999). Mouse beta 1,3-galactosyltransferase (GA1/GM1/GD1b synthase): protein characterization, tissue expression, and developmental regulation in neural retina.. PubMed. 58(2). 318–27. 14 indexed citations

19.

Martina, José A., José L. Daniotti, & Hugo J. F. Maccioni. (1998). Influence of N-Glycosylation and N-Glycan Trimming on the Activity and Intracellular Traffic of GD3 Synthase. Journal of Biological Chemistry. 273(6). 3725–3731. 73 indexed citations

20.

Martina, José A., José L. Daniotti, & Hugo J. F. Maccioni. (1995). A UDP‐Sugar Pyrophosphatase Is Developmentally Regulated in the Rat Retina. Journal of Neurochemistry. 64(3). 1274–1280. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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