Jonathan M. Haldeman

920 total citations
12 papers, 419 citations indexed

About

Jonathan M. Haldeman is a scholar working on Surgery, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Jonathan M. Haldeman has authored 12 papers receiving a total of 419 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Surgery, 6 papers in Molecular Biology and 3 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Jonathan M. Haldeman's work include Pancreatic function and diabetes (6 papers), Cholesterol and Lipid Metabolism (3 papers) and Lipid metabolism and disorders (2 papers). Jonathan M. Haldeman is often cited by papers focused on Pancreatic function and diabetes (6 papers), Cholesterol and Lipid Metabolism (3 papers) and Lipid metabolism and disorders (2 papers). Jonathan M. Haldeman collaborates with scholars based in United States, Canada and Switzerland. Jonathan M. Haldeman's co-authors include Christopher B. Newgard, Hans E. Hohmeier, David A. D’Alessio, Megan E. Capozzi, Samuel B. Stephens, Sophie L. Lewandowski, Mackenzie D. Martin, Jonathan E. Campbell, Patrick E. MacDonald and Haopeng Lin and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Jonathan M. Haldeman

12 papers receiving 417 citations

Peers

Jonathan M. Haldeman
Jonathan M. Haldeman
Citations per year, relative to Jonathan M. Haldeman Jonathan M. Haldeman (= 1×) peers Austin Bautista

Countries citing papers authored by Jonathan M. Haldeman

Since Specialization
Citations

This map shows the geographic impact of Jonathan M. Haldeman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan M. Haldeman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan M. Haldeman more than expected).

Fields of papers citing papers by Jonathan M. Haldeman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan M. Haldeman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan M. Haldeman. The network helps show where Jonathan M. Haldeman may publish in the future.

Co-authorship network of co-authors of Jonathan M. Haldeman

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan M. Haldeman. A scholar is included among the top collaborators of Jonathan M. Haldeman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan M. Haldeman. Jonathan M. Haldeman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Katz, Liora S., Peng Wang, Luca Lambertini, et al.. (2023). Transcriptional activation of the Myc gene by glucose in β-cells requires a ChREBP-dependent 3-D chromatin interaction between the Myc and Pvt1 genes. Molecular Metabolism. 79. 101848–101848. 2 indexed citations
2.
Sargsyan, Ashot, Ludivine Doridot, Sarah Anissa Hannou, et al.. (2022). HGFAC is a ChREBP-regulated hepatokine that enhances glucose and lipid homeostasis. JCI Insight. 8(1). 16 indexed citations
3.
Sargsyan, Ashot, Ludivine Doridot, Sarah Anissa Hannou, et al.. (2021). 205-LB: HGFAC Is a ChREBP-Regulated Hepatokine That Enhances Glucose and Lipid Homeostasis. Diabetes. 70(Supplement_1). 1 indexed citations
4.
Haldeman, Jonathan M., et al.. (2019). Chromogranin B regulates early-stage insulin granule trafficking from the Golgi in pancreatic islet β-cells. Journal of Cell Science. 132(13). 38 indexed citations
5.
Capozzi, Megan E., Berit Svendsen, Sophie L. Lewandowski, et al.. (2019). β Cell tone is defined by proglucagon peptides through cAMP signaling. JCI Insight. 4(5). 178 indexed citations
6.
Haldeman, Jonathan M., Amanda E. Conway, Michelle Arlotto, et al.. (2018). Creation of versatile cloning platforms for transgene expression and dCas9-based epigenome editing. Nucleic Acids Research. 47(4). e23–e23. 23 indexed citations
7.
An, Jie, Liping Wang, Michael L. Patnode, et al.. (2018). Physiological mechanisms of sustained fumagillin-induced weight loss. JCI Insight. 3(5). 9 indexed citations
8.
Peterson, Brett S., et al.. (2017). Delayed apoptosis allows islet β-cells to implement an autophagic mechanism to promote cell survival. PLoS ONE. 12(2). e0172567–e0172567. 35 indexed citations
9.
Zhang, Lu, Thomas Becker, Jonathan M. Haldeman, et al.. (2016). A Pdx-1-Regulated Soluble Factor Activates Rat and Human Islet Cell Proliferation. Molecular and Cellular Biology. 36(23). 2918–2930. 18 indexed citations
10.
Conway, Amanda E., Jonathan M. Haldeman, Daniel S. Wechsler, & Catherine Lavau. (2014). A critical role for CRM1 in regulating HOXA gene transcription in CALM-AF10 leukemias. Leukemia. 29(2). 423–432. 32 indexed citations
11.
Jensen, Mette V., Jonathan M. Haldeman, Hengtao Zhang, et al.. (2013). Control of Voltage-gated Potassium Channel Kv2.2 Expression by Pyruvate-Isocitrate Cycling Regulates Glucose-stimulated Insulin Secretion. Journal of Biological Chemistry. 288(32). 23128–23140. 18 indexed citations
12.
Moss, Larry G., Jonathan C. Schisler, Jonathan M. Haldeman, et al.. (2013). Pdx-1 Activates Islet α- and β-Cell Proliferation via a Mechanism Regulated by Transient Receptor Potential Cation Channels 3 and 6 and Extracellular Signal-Regulated Kinases 1 and 2. Molecular and Cellular Biology. 33(20). 4017–4029. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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