Jonathan I. Millen
Impact in
- Aging top 10%
- Genetics, Aging, and Longevity in Model Organisms
- Geriatrics and Gerontology top 10%
- Sirtuins and Resveratrol in Medicine
Papers in
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- Cancer therapeutics and mechanisms 1
- Nuclear Structure and Function 1
- DNA Repair Mechanisms 1
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- Cellular transport and secretion 3
- Co-authors
- David S. Goldfarb (6 shared papers)Roswitha Krick (2 shared papers)Michael Thumm (2 shared papers)Eeva‐Liisa Eskelinen (1 shared paper)Petra Schlotterhose (1 shared paper)Sebastian Bremer (1 shared paper)Stefanie Jarolim (1 shared paper)Michael Breitenbach (1 shared paper)
- Journals
- FEMS Yeast Research (1 paper)Traffic (1 paper)Autophagy (1 paper)Molecular Biology of the Cell (1 paper)Applied Microbiology and Biotechnology (1 paper)
- Partner nations
- United StatesGermanyFinland
In The Last Decade
Jonathan I. Millen
7 papers receiving 351 citations
Peers
Comparison fields: 5 of 51
- Aging 35
- Geriatrics and Gerontology 46
- Cell Biology 113
- Physiology 19
- Epidemiology 142
Countries citing papers authored by Jonathan I. Millen
This map shows the geographic impact of Jonathan I. Millen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan I. Millen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan I. Millen more than expected).
Fields of papers citing papers by Jonathan I. Millen
This network shows the impact of papers produced by Jonathan I. Millen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan I. Millen. The network helps show where Jonathan I. Millen may publish in the future.
Co-authors
The 25 scholars most cited alongside Jonathan I. Millen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 147 | |
| 2 | 2004 | 80 | |
| 3 | 2015 | 40 | |
| 4 | 2009 | 37 | |
| 5 | 2008 | 26 | |
| 6 | 2011 | 17 | |
| 7 | 2017 | 9 |
About Jonathan I. Millen
Jonathan I. Millen is a scholar working on Molecular Biology, Cell Biology, Epidemiology, Infectious Diseases and Pharmacology, having authored 7 papers that have together received 356 indexed citations. Recurring topics across this work include Cellular transport and secretion (3 papers), Autophagy in Disease and Therapy (2 papers), Metabolism and Genetic Disorders (1 paper), Cancer therapeutics and mechanisms (1 paper), Bacterial Genetics and Biotechnology (1 paper), Nuclear Structure and Function (1 paper), Parkinson's Disease Mechanisms and Treatments (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Aging (35 citations), Geriatrics and Gerontology (46 citations), Cell Biology (113 citations), Physiology (19 citations) and Epidemiology (142 citations). Jonathan I. Millen has collaborated with scholars based in United States, Germany and Finland. Frequent co-authors include David S. Goldfarb, Roswitha Krick, Michael Thumm, Eeva‐Liisa Eskelinen, Petra Schlotterhose, Sebastian Bremer, Stefanie Jarolim, Michael Breitenbach, Peter Laun and Gino Heeren. Their work appears in journals such as FEMS Yeast Research, Traffic, Autophagy, Molecular Biology of the Cell and Applied Microbiology and Biotechnology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.