Jonathan Eggenschwiler

4.5k total citations · 1 hit paper
26 papers, 3.4k citations indexed

About

Jonathan Eggenschwiler is a scholar working on Molecular Biology, Genetics and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Jonathan Eggenschwiler has authored 26 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 17 papers in Genetics and 4 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Jonathan Eggenschwiler's work include Hedgehog Signaling Pathway Studies (17 papers), Genetic and Kidney Cyst Diseases (13 papers) and Epigenetics and DNA Methylation (13 papers). Jonathan Eggenschwiler is often cited by papers focused on Hedgehog Signaling Pathway Studies (17 papers), Genetic and Kidney Cyst Diseases (13 papers) and Epigenetics and DNA Methylation (13 papers). Jonathan Eggenschwiler collaborates with scholars based in United States, South Korea and Japan. Jonathan Eggenschwiler's co-authors include Kathryn V. Anderson, Argiris Efstratiadis, Philip A. Leighton, Shirley M. Tilghman, Robert S. Ingram, Thomas Ludwig, Peter Fisher, Hyuk Wan Ko, Marsha L. Davenport and A. Joseph D’Ercole and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Jonathan Eggenschwiler

26 papers receiving 3.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Disruption of imprinting caused by deletion of the H19 gene region in mice

1995 627 citations Philip A. Leighton, Jonathan Eggenschwiler et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jonathan Eggenschwiler United States	19	2.9k	2.2k	632	437	223	26	3.4k
Beyhan Tüysüz Türkiye	24	1.4k 0.5×	988 0.5×	171 0.3×	469 1.1×	70 0.3×	139	2.5k
Jay W. Ellison United States	23	1.2k 0.4×	1.2k 0.6×	438 0.7×	72 0.2×	109 0.5×	52	2.6k
Vincent El Ghouzzi France	27	1.1k 0.4×	1.0k 0.5×	185 0.3×	233 0.5×	46 0.2×	44	2.1k
Yasemin Alanay Türkiye	26	994 0.3×	1.0k 0.5×	179 0.3×	162 0.4×	66 0.3×	108	2.1k
Israela Lerer Israel	29	1.2k 0.4×	972 0.4×	301 0.5×	108 0.2×	45 0.2×	77	2.5k
Keiko Wakui Japan	28	1.3k 0.5×	1.4k 0.6×	283 0.4×	190 0.4×	27 0.1×	89	2.3k
Koji Muroya Japan	28	1.9k 0.7×	1.9k 0.9×	301 0.5×	47 0.1×	719 3.2×	119	3.1k
Holger Tönnies Germany	23	927 0.3×	833 0.4×	179 0.3×	90 0.2×	180 0.8×	55	1.6k
Corinne Stoetzel France	30	2.5k 0.9×	1.8k 0.8×	133 0.2×	273 0.6×	22 0.1×	67	3.3k
Nursel Elçioğlu Türkiye	19	1.2k 0.4×	1.3k 0.6×	143 0.2×	195 0.4×	33 0.1×	70	2.0k

Countries citing papers authored by Jonathan Eggenschwiler

Since Specialization

Citations

This map shows the geographic impact of Jonathan Eggenschwiler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan Eggenschwiler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan Eggenschwiler more than expected).

Fields of papers citing papers by Jonathan Eggenschwiler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan Eggenschwiler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan Eggenschwiler. The network helps show where Jonathan Eggenschwiler may publish in the future.

Co-authorship network of co-authors of Jonathan Eggenschwiler

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan Eggenschwiler. A scholar is included among the top collaborators of Jonathan Eggenschwiler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan Eggenschwiler. Jonathan Eggenschwiler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Eggenschwiler, Jonathan, et al.. (2024). A new cell culture resource for investigations of reptilian gene function. Development. 151(22). 2 indexed citations

Lupu, Floria, et al.. (2017). Cell cycle-related kinase regulates mammalian eye development through positive and negative regulation of the Hedgehog pathway. Developmental Biology. 434(1). 24–35. 10 indexed citations

Brown, Desmond, Hankyu Lee, Floria Lupu, et al.. (2017). Cell Cycle-Related Kinase (CCRK) regulates ciliogenesis and Hedgehog signaling in mice. PLoS Genetics. 13(8). e1006912–e1006912. 35 indexed citations

Lupu, Floria, et al.. (2017). Proper ciliary assembly is critical for restricting Hedgehog signaling during early eye development in mice. Developmental Biology. 430(1). 32–40. 15 indexed citations

Sornborger, Andrew, Jie Li, Floria Lupu, et al.. (2017). MiCASA is a new method for quantifying cellular organization. Nature Communications. 8(1). 15619–15619. 2 indexed citations

O’Connell, Joyce T., et al.. (2014). Rab23 regulates Nodal signaling in vertebrate left–right patterning independently of the Hedgehog pathway. Developmental Biology. 391(2). 182–195. 32 indexed citations

Liu, Aimin & Jonathan Eggenschwiler. (2013). Identifying Essential Genes in Mouse Development via an ENU-Based Forward Genetic Approach. Methods in molecular biology. 1092. 95–118. 4 indexed citations

Ocbina, Polloneal Jymmiel R., Jonathan Eggenschwiler, Ivan P. Moskowitz, & Kathryn V. Anderson. (2011). Complex interactions between genes controlling trafficking in primary cilia. Nature Genetics. 43(6). 547–553. 152 indexed citations

Ko, Hyuk Wan, et al.. (2010). Broad-Minded Links Cell Cycle-Related Kinase to Cilia Assembly and Hedgehog Signal Transduction. Developmental Cell. 18(2). 237–247. 80 indexed citations

10.

Walczak‐Sztulpa, Joanna, Jonathan Eggenschwiler, Daniel P. S. Osborn, et al.. (2010). Cranioectodermal Dysplasia, Sensenbrenner Syndrome, Is a Ciliopathy Caused by Mutations in the IFT122 Gene. The American Journal of Human Genetics. 86(6). 949–956. 149 indexed citations

11.

Ko, Hyuk Wan, Aimin Liu, & Jonathan Eggenschwiler. (2009). Analysis of Hedgehog Signaling in Mouse Intraflagellar Transport Mutants. Methods in cell biology. 93. 347–369. 6 indexed citations

12.

Ko, Hyuk Wan, et al.. (2009). Tubby-like protein 3 (TULP3) regulates patterning in the mouse embryo through inhibition of Hedgehog signaling. Human Molecular Genetics. 18(10). 1740–1754. 85 indexed citations

13.

Ko, Hyuk Wan, et al.. (2008). FKBP8 cell-autonomously controls neural tube patterning through a Gli2- and Kif3a-dependent mechanism. Developmental Biology. 321(1). 27–39. 56 indexed citations

14.

Eggenschwiler, Jonathan & Kathryn V. Anderson. (2007). Cilia and Developmental Signaling. Annual Review of Cell and Developmental Biology. 23(1). 345–373. 445 indexed citations

15.

Eggenschwiler, Jonathan, Oleg V. Bulgakov, Jian Qin, Tiansen Li, & Kathryn V. Anderson. (2005). Mouse Rab23 regulates Hedgehog signaling from Smoothened to Gli proteins. Developmental Biology. 290(1). 1–12. 120 indexed citations

16.

Caspary, Tamara, María J. García‐García, Danwei Huangfu, et al.. (2002). Mouse Dispatched homolog1 Is Required for Long-Range, but Not Juxtacrine, Hh Signaling. Current Biology. 12(18). 1628–1632. 137 indexed citations

17.

Eggenschwiler, Jonathan, et al.. (2001). Rab23 is an essential negative regulator of the mouse Sonic hedgehog signalling pathway. Nature. 412(6843). 194–198. 295 indexed citations

18.

Eggenschwiler, Jonathan & Kathryn V. Anderson. (2000). Dorsal and Lateral Fates in the Mouse Neural Tube Require the Cell-Autonomous Activity of the open brain Gene. Developmental Biology. 227(2). 648–660. 59 indexed citations

19.

Eggenschwiler, Jonathan, Thomas Ludwig, Peter Fisher, et al.. (1997). Mouse mutant embryos overexpressing IGF-II exhibit phenotypic features of the Beckwith–Wiedemann and Simpson–Golabi–Behmel syndromes. Genes & Development. 11(23). 3128–3142. 270 indexed citations

20.

Ludwig, Thomas, Jonathan Eggenschwiler, Peter Fisher, et al.. (1996). Mouse Mutants Lacking the Type 2 IGF Receptor (IGF2R) Are Rescued from Perinatal Lethality inIgf2andIgf1rNull Backgrounds. Developmental Biology. 177(2). 517–535. 382 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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