Jonathan A. Deane

2.6k total citations · 1 hit paper
16 papers, 2.0k citations indexed

About

Jonathan A. Deane is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Jonathan A. Deane has authored 16 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Immunology, 7 papers in Molecular Biology and 5 papers in Oncology. Recurrent topics in Jonathan A. Deane's work include Immune Response and Inflammation (6 papers), T-cell and B-cell Immunology (6 papers) and Immune Cell Function and Interaction (6 papers). Jonathan A. Deane is often cited by papers focused on Immune Response and Inflammation (6 papers), T-cell and B-cell Immunology (6 papers) and Immune Cell Function and Interaction (6 papers). Jonathan A. Deane collaborates with scholars based in United States, Switzerland and Italy. Jonathan A. Deane's co-authors include Silvia Bolland, Prapaporn Pisitkun, Т. Н. Тарасенко, Michael J. Difilippantonio, Anne B. Satterthwaite, Lionel Feigenbaum, Richard A. Flavell, Jerrold M. Ward, Terrence Town and Lewis C. Cantley and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Blood.

In The Last Decade

Jonathan A. Deane

16 papers receiving 2.0k citations

Hit Papers

Autoreactive B Cell Responses to RNA-Related Antigens Due... 2006 2026 2012 2019 2006 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jonathan A. Deane United States 13 1.5k 589 495 292 153 16 2.0k
Marie‐Laure Santiago‐Raber Switzerland 26 1.5k 1.0× 475 0.8× 591 1.2× 352 1.2× 102 0.7× 46 2.1k
Robert J. Rigby Australia 15 1.8k 1.2× 314 0.5× 318 0.6× 229 0.8× 161 1.1× 21 2.1k
Leonie M. van Duivenvoorde Netherlands 22 1.9k 1.3× 386 0.7× 535 1.1× 599 2.1× 121 0.8× 46 2.5k
Chen‐Feng Qi United States 22 1.6k 1.1× 625 1.1× 127 0.3× 544 1.9× 169 1.1× 57 2.4k
Joel Schiffenbauer United States 22 1.2k 0.8× 525 0.9× 515 1.0× 217 0.7× 240 1.6× 43 1.9k
Michael Hahne France 11 1.1k 0.7× 652 1.1× 162 0.3× 232 0.8× 92 0.6× 12 1.7k
Stephen B. Gauld United States 21 1.2k 0.8× 361 0.6× 120 0.2× 296 1.0× 174 1.1× 40 1.8k
Ramón Gimeno Spain 19 877 0.6× 347 0.6× 258 0.5× 548 1.9× 135 0.9× 42 1.5k
Nikita Kolhatkar United States 13 1.2k 0.8× 433 0.7× 174 0.4× 682 2.3× 124 0.8× 18 1.8k
Barbara J. Vilen United States 18 860 0.6× 271 0.5× 175 0.4× 115 0.4× 95 0.6× 31 1.2k

Countries citing papers authored by Jonathan A. Deane

Since Specialization
Citations

This map shows the geographic impact of Jonathan A. Deane's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan A. Deane with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan A. Deane more than expected).

Fields of papers citing papers by Jonathan A. Deane

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan A. Deane. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan A. Deane. The network helps show where Jonathan A. Deane may publish in the future.

Co-authorship network of co-authors of Jonathan A. Deane

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan A. Deane. A scholar is included among the top collaborators of Jonathan A. Deane based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan A. Deane. Jonathan A. Deane is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Deane, Jonathan A., Chun Li, Shailaja Kasibhatla, et al.. (2019). Abstract 4128: Identification and characterization of LHC165, a TLR7 agonist designed for localized intratumoral therapies. Cancer Research. 79(13_Supplement). 4128–4128. 9 indexed citations
2.
Deane, Jonathan A., et al.. (2019). Abstract 4128: Identification and characterization of LHC165, a TLR7 agonist designed for localized intratumoral therapies. Immunology. 4128–4128. 2 indexed citations
3.
Annable, Tami, et al.. (2014). Using Poly I:C as an adjuvant does not induce or exacerbate models of systemic lupus erythematosus. Autoimmunity. 48(1). 29–39. 8 indexed citations
4.
Walsh, Elizabeth R., Prapaporn Pisitkun, Elisaveta Voynova, et al.. (2012). Dual signaling by innate and adaptive immune receptors is required for TLR7-induced B-cell–mediated autoimmunity. Proceedings of the National Academy of Sciences. 109(40). 16276–16281. 82 indexed citations
5.
Crampton, Steve P., Jonathan A. Deane, Lionel Feigenbaum, & Silvia Bolland. (2011). Ifih1 Gene Dose Effect Reveals MDA5-Mediated Chronic Type I IFN Gene Signature, Viral Resistance, and Accelerated Autoimmunity. The Journal of Immunology. 188(3). 1451–1459. 66 indexed citations
6.
Oak, Jean, et al.. (2009). The p85β regulatory subunit of phosphoinositide 3-kinase has unique and redundant functions in B cells. Autoimmunity. 42(5). 447–458. 15 indexed citations
7.
Matheu, Melanie P., et al.. (2007). Class IA Phosphoinositide 3-Kinase Modulates Basal Lymphocyte Motility in the Lymph Node. The Journal of Immunology. 179(4). 2261–2269. 35 indexed citations
8.
Deane, Jonathan A., Prapaporn Pisitkun, Lionel Feigenbaum, et al.. (2007). Control of Toll-like Receptor 7 Expression Is Essential to Restrict Autoimmunity and Dendritic Cell Proliferation. Immunity. 27(5). 801–810. 457 indexed citations
9.
Pisitkun, Prapaporn, Jonathan A. Deane, Michael J. Difilippantonio, et al.. (2006). Autoreactive B Cell Responses to RNA-Related Antigens Due to TLR7 Gene Duplication. Science. 312(5780). 1669–1672. 694 indexed citations breakdown →
10.
Oak, Jean, Jonathan A. Deane, Michael G. Kharas, et al.. (2006). Sjögren's syndrome-like disease in mice with T cells lacking class 1A phosphoinositide-3-kinase. Proceedings of the National Academy of Sciences. 103(45). 16882–16887. 54 indexed citations
11.
Deane, Jonathan A. & Silvia Bolland. (2006). Nucleic Acid-Sensing TLRs as Modifiers of Autoimmunity. The Journal of Immunology. 177(10). 6573–6578. 59 indexed citations
12.
Deane, Jonathan A., Michael G. Kharas, Jean Oak, et al.. (2006). T-cell function is partially maintained in the absence of class IA phosphoinositide 3-kinase signaling. Blood. 109(7). 2894–2902. 50 indexed citations
13.
Brachmann, Saskia M., Claudine M. Yballe, Metello Innocenti, et al.. (2005). Role of Phosphoinositide 3-Kinase Regulatory Isoforms in Development and Actin Rearrangement. Molecular and Cellular Biology. 25(7). 2593–2606. 104 indexed citations
14.
Deane, Jonathan A., et al.. (2004). Enhanced T Cell Proliferation in Mice Lacking the p85β Subunit of Phosphoinositide 3-Kinase. The Journal of Immunology. 172(11). 6615–6625. 56 indexed citations
15.
Kharas, Michael G., et al.. (2004). Phosphoinositide 3-kinase signaling is essential for ABL oncogene–mediated transformation of B-lineage cells. Blood. 103(11). 4268–4275. 73 indexed citations
16.
Deane, Jonathan A., et al.. (2004). P hosphoinositide 3-K inase : Diverse Roles in Immune Cell Activation. Annual Review of Immunology. 22(1). 563–598. 279 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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