Jonas Donner

1.3k total citations
36 papers, 761 citations indexed

About

Jonas Donner is a scholar working on Genetics, Molecular Biology and Neurology. According to data from OpenAlex, Jonas Donner has authored 36 papers receiving a total of 761 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Genetics, 14 papers in Molecular Biology and 5 papers in Neurology. Recurrent topics in Jonas Donner's work include Genetic and phenotypic traits in livestock (6 papers), Human-Animal Interaction Studies (6 papers) and Neurological diseases and metabolism (3 papers). Jonas Donner is often cited by papers focused on Genetic and phenotypic traits in livestock (6 papers), Human-Animal Interaction Studies (6 papers) and Neurological diseases and metabolism (3 papers). Jonas Donner collaborates with scholars based in Finland, United States and Germany. Jonas Donner's co-authors include Iiris Hovatta, Juuso Juhila, Heidi Anderson, Hannes Lohi, Jouko Lönnqvist, Laura Kananen, Sami Pirkola, Kaisa Silander, Joseph D. Terwilliger and Leena Peltonen and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Biological Psychiatry.

In The Last Decade

Jonas Donner

32 papers receiving 732 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jonas Donner Finland 12 242 199 131 117 82 36 761
Ivana Jarić Serbia 14 107 0.4× 207 1.0× 154 1.2× 62 0.5× 85 1.0× 33 834
Ana Magalhães Portugal 21 158 0.7× 258 1.3× 144 1.1× 113 1.0× 117 1.4× 45 1.1k
Т. И. Меркулова Russia 13 96 0.4× 287 1.4× 147 1.1× 80 0.7× 54 0.7× 69 627
Igor Vukobradovic Canada 5 110 0.5× 159 0.8× 121 0.9× 83 0.7× 66 0.8× 7 587
Hiroshi Ueno Japan 18 86 0.4× 290 1.5× 119 0.9× 56 0.5× 106 1.3× 62 1.3k
Juli Choi South Korea 16 71 0.3× 374 1.9× 120 0.9× 163 1.4× 139 1.7× 21 776
Michelle Cora United States 8 91 0.4× 154 0.8× 129 1.0× 27 0.2× 84 1.0× 26 806
Thiago Berti Kirsten Brazil 18 119 0.5× 101 0.5× 271 2.1× 235 2.0× 81 1.0× 63 1.1k
Leah C. Solberg Woods United States 19 224 0.9× 413 2.1× 155 1.2× 57 0.5× 225 2.7× 50 1.2k
Linda Kooistra United States 8 107 0.4× 151 0.8× 128 1.0× 30 0.3× 89 1.1× 11 885

Countries citing papers authored by Jonas Donner

Since Specialization
Citations

This map shows the geographic impact of Jonas Donner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonas Donner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonas Donner more than expected).

Fields of papers citing papers by Jonas Donner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonas Donner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonas Donner. The network helps show where Jonas Donner may publish in the future.

Co-authorship network of co-authors of Jonas Donner

This figure shows the co-authorship network connecting the top 25 collaborators of Jonas Donner. A scholar is included among the top collaborators of Jonas Donner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonas Donner. Jonas Donner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Letko, Anna, Pascale Quignon, Jonas Donner, et al.. (2024). A RETREG1 variant is associated with hereditary sensory and autonomic neuropathy with acral self‐mutilation in purebred German Spitz. Animal Genetics. 55(6). 810–819.
2.
Labadie, Julia, et al.. (2024). Association of FGF4L1 Retrogene Insertion with Prolapsed Gland of the Nictitans (Cherry Eye) in Dogs. Genes. 15(2). 198–198. 2 indexed citations
3.
Bannasch, Danika L., C. Elizabeth Boudreau, Molly E. Church, et al.. (2023). Association of the FGF4L2 retrogene with fibrocartilaginous embolic myelopathy in dogs. Journal of Veterinary Internal Medicine. 38(1). 258–267. 2 indexed citations
4.
Hytönen, Marjo K., et al.. (2023). A frameshift deletion inF8associated with hemophilia A in Labrador Retriever dogs. Animal Genetics. 54(5). 606–612.
6.
Flegel, Thomas, Hanna Müller, Alexandra Kehl, et al.. (2023). A loss-of-function variant in canine GLRA1 associates with a neurological disorder resembling human hyperekplexia. Human Genetics. 142(8). 1221–1230. 4 indexed citations
7.
Anderson, Heidi, Stephen Davison, Kaisa Kyöstilä, et al.. (2022). Genetic epidemiology of blood type, disease and trait variants, and genome-wide genetic diversity in over 11,000 domestic cats. PLoS Genetics. 18(6). e1009804–e1009804. 11 indexed citations
8.
Hytönen, Marjo K., Christopher B. Jackson, Pernilla Syrjä, et al.. (2021). In-frame deletion in canine PITRM1 is associated with a severe early-onset epilepsy, mitochondrial dysfunction and neurodegeneration. Human Genetics. 140(11). 1593–1609. 11 indexed citations
9.
Kyöstilä, Kaisa, Julia E. Niskanen, Meharji Arumilli, et al.. (2021). Intronic variant in POU1F1 associated with canine pituitary dwarfism. Human Genetics. 140(11). 1553–1562. 3 indexed citations
10.
Ochs, Matthias, Julia E. Niskanen, Meharji Arumilli, et al.. (2020). Recessive missense LAMP3 variant associated with defect in lamellar body biogenesis and fatal neonatal interstitial lung disease in dogs. PLoS Genetics. 16(3). e1008651–e1008651. 5 indexed citations
13.
Dreger, Dayna L., Angela M. Hughes, Balasubramanian Ganesan, et al.. (2019). True Colors: Commercially-acquired morphological genotypes reveal hidden allele variation among dog breeds, informing both trait ancestry and breed potential. PLoS ONE. 14(10). e0223995–e0223995. 21 indexed citations
14.
Donner, Jonas, Heidi Anderson, Stephen Davison, et al.. (2018). Frequency and distribution of 152 genetic disease variants in over 100,000 mixed breed and purebred dogs. PLoS Genetics. 14(4). e1007361–e1007361. 70 indexed citations
15.
Donner, Jonas, Maria Kaukonen, Heidi Anderson, et al.. (2016). Genetic Panel Screening of Nearly 100 Mutations Reveals New Insights into the Breed Distribution of Risk Variants for Canine Hereditary Disorders. PLoS ONE. 11(8). e0161005–e0161005. 41 indexed citations
16.
Donner, Jonas, Tessa Sipilä, Samuli Ripatti, et al.. (2012). Support for involvement of glutamate decarboxylase 1 and neuropeptide y in anxiety susceptibility. STM:n Hallinnonalan avoin julkaisuarkisto (Julkari). 1 indexed citations
17.
Hovatta, Iiris, Juuso Juhila, & Jonas Donner. (2010). Oxidative stress in anxiety and comorbid disorders. STM:n Hallinnonalan avoin julkaisuarkisto (Julkari). 7 indexed citations
18.
Hovatta, Iiris, Juuso Juhila, & Jonas Donner. (2010). Oxidative stress in anxiety and comorbid disorders. Neuroscience Research. 68(4). 261–275. 270 indexed citations
19.
Sipilä, Tessa, Laura Kananen, Dario Greco, et al.. (2010). An Association Analysis of Circadian Genes in Anxiety Disorders. Biological Psychiatry. 67(12). 1163–1170. 66 indexed citations
20.
Donner, Jonas, Sami Pirkola, Kaisa Silander, et al.. (2008). An Association Analysis of Murine Anxiety Genes in Humans Implicates Novel Candidate Genes for Anxiety Disorders. Biological Psychiatry. 64(8). 672–680. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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