Jon T. Skare

2.8k total citations
45 papers, 2.2k citations indexed

About

Jon T. Skare is a scholar working on Parasitology, Infectious Diseases and Insect Science. According to data from OpenAlex, Jon T. Skare has authored 45 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Parasitology, 22 papers in Infectious Diseases and 16 papers in Insect Science. Recurrent topics in Jon T. Skare's work include Vector-borne infectious diseases (34 papers), Viral Infections and Vectors (19 papers) and Insect symbiosis and bacterial influences (9 papers). Jon T. Skare is often cited by papers focused on Vector-borne infectious diseases (34 papers), Viral Infections and Vectors (19 papers) and Insect symbiosis and bacterial influences (9 papers). Jon T. Skare collaborates with scholars based in United States, Canada and Germany. Jon T. Skare's co-authors include Kathleen Postle, Jenny A. Hyde, Brandon L. Garcia, D R Blanco, James N. Miller, Michael Lovett, Richard P. Darveau, Carrie L. Seachord, Magnus Höök and Jerome P. Trzeciakowski and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The Journal of Immunology.

In The Last Decade

Jon T. Skare

43 papers receiving 2.2k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Jon T. Skare 1.4k 946 502 495 490 45 2.2k
Taissia G. Popova 1.1k 0.8× 814 0.9× 509 1.0× 498 1.0× 199 0.4× 40 1.8k
Jason A. Carlyon 1.5k 1.0× 811 0.9× 341 0.7× 407 0.8× 232 0.5× 81 2.2k
Tao Lin 1.0k 0.7× 786 0.8× 357 0.7× 336 0.7× 230 0.5× 61 1.7k
Albert Mulenga 2.0k 1.4× 699 0.7× 1.2k 2.3× 385 0.8× 204 0.4× 82 2.5k
Carlos Termignoni 1.1k 0.8× 402 0.4× 621 1.2× 541 1.1× 198 0.4× 73 2.0k
Jenifer Coburn 2.7k 1.9× 1.9k 2.0× 439 0.9× 806 1.6× 445 0.9× 75 4.0k
Jinlin Zhou 1.5k 1.0× 675 0.7× 535 1.1× 489 1.0× 113 0.2× 124 2.2k
James L. Coleman 2.0k 1.4× 1.6k 1.7× 352 0.7× 294 0.6× 146 0.3× 51 2.6k
Mingqun Lin 897 0.6× 462 0.5× 296 0.6× 446 0.9× 165 0.3× 50 1.7k
Christian H. Eggers 2.1k 1.4× 1.3k 1.4× 1.0k 2.0× 193 0.4× 174 0.4× 32 2.5k

Countries citing papers authored by Jon T. Skare

Since Specialization
Citations

This map shows the geographic impact of Jon T. Skare's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon T. Skare with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon T. Skare more than expected).

Fields of papers citing papers by Jon T. Skare

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon T. Skare. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon T. Skare. The network helps show where Jon T. Skare may publish in the future.

Co-authorship network of co-authors of Jon T. Skare

This figure shows the co-authorship network connecting the top 25 collaborators of Jon T. Skare. A scholar is included among the top collaborators of Jon T. Skare based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon T. Skare. Jon T. Skare is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Booth, Charles E., et al.. (2025). BBK32 attenuates antibody-dependent complement-mediated killing of infectious Borreliella burgdorferi isolates. PLoS Pathogens. 21(7). e1013361–e1013361.
3.
Booth, Charles E., et al.. (2023). Conformational dynamics of complement protease C1r inhibitor proteins from Lyme disease– and relapsing fever–causing spirochetes. Journal of Biological Chemistry. 299(8). 104972–104972. 4 indexed citations
4.
Khandelwal, Sanjay, Simone Sartoretto, Grace M. Lee, et al.. (2022). Minimal role for the alternative pathway in complement activation by HIT immune complexes. Journal of Thrombosis and Haemostasis. 20(11). 2656–2665. 4 indexed citations
5.
Booth, Charles E., et al.. (2022). Borrelia miyamotoi FbpA and FbpB Are Immunomodulatory Outer Surface Lipoproteins With Distinct Structures and Functions. Frontiers in Immunology. 13. 886733–886733. 8 indexed citations
6.
Hammel, Michal, et al.. (2021). A Structural Basis for Inhibition of the Complement Initiator Protease C1r by Lyme Disease Spirochetes. The Journal of Immunology. 207(11). 2856–2867. 15 indexed citations
7.
Coburn, Jenifer, Brandon L. Garcia, Linden T. Hu, et al.. (2020). Lyme Disease Pathogenesis. Current Issues in Molecular Biology. 42. 473–518. 59 indexed citations
8.
Skare, Jon T. & Brandon L. Garcia. (2020). Complement Evasion by Lyme Disease Spirochetes. Trends in Microbiology. 28(11). 889–899. 47 indexed citations
9.
Troy, Erin B., Steven J. Norris, Tao Lin, et al.. (2020). The intergenic small non-coding RNA ittA is required for optimal infectivity and tissue tropism in Borrelia burgdorferi. PLoS Pathogens. 16(5). e1008423–e1008423. 13 indexed citations
10.
Kern, Aurélie, Bijaya Sharma, Tao Lin, et al.. (2019). Genome-wide screen identifies novel genes required for Borrelia burgdorferi survival in its Ixodes tick vector. PLoS Pathogens. 15(5). e1007644–e1007644. 26 indexed citations
12.
Zhi, Hui, Jialei Xie, & Jon T. Skare. (2018). The Classical Complement Pathway Is Required to Control Borrelia burgdorferi Levels During Experimental Infection. Frontiers in Immunology. 9. 959–959. 23 indexed citations
13.
Hyde, Jenny A., Lihui Gao, Xin Li, et al.. (2017). A high-throughput genetic screen identifies previously uncharacterized Borrelia burgdorferi genes important for resistance against reactive oxygen and nitrogen species. PLoS Pathogens. 13(2). e1006225–e1006225. 32 indexed citations
14.
Hyde, Jenny A. & Jon T. Skare. (2017). Detection of Bioluminescent Borrelia burgdorferi from In Vitro Cultivation and During Murine Infection. Methods in molecular biology. 1690. 241–257. 6 indexed citations
15.
Gupta, Nupur, et al.. (2016). Biomechanics of Borrelia burgdorferi Vascular Interactions. Cell Reports. 16(10). 2593–2604. 46 indexed citations
16.
Garcia, Brandon L., et al.. (2016). Borrelia burgdorferi BBK32 Inhibits the Classical Pathway by Blocking Activation of the C1 Complement Complex. PLoS Pathogens. 12(1). e1005404–e1005404. 94 indexed citations
17.
Hyde, Jenny A., Eric H. Weening, Mi-Hee Chang, et al.. (2011). Bioluminescent imaging of Borrelia burgdorferi in vivo demonstrates that the fibronectin‐binding protein BBK32 is required for optimal infectivity. Molecular Microbiology. 82(1). 99–113. 90 indexed citations
18.
Hyde, Jenny A., Dana K. Shaw, Roger Smith, Jerome P. Trzeciakowski, & Jon T. Skare. (2009). The BosR regulatory protein of Borrelia burgdorferi interfaces with the RpoS regulatory pathway and modulates both the oxidative stress response and pathogenic properties of the Lyme disease spirochete. Molecular Microbiology. 74(6). 1344–1355. 103 indexed citations
19.
Foley, Denise, Jon T. Skare, Elizabeth A. Wagar, et al.. (1995). Rabbit model of Lyme borreliosis: erythema migrans, infection-derived immunity, and identification of Borrelia burgdorferi proteins associated with virulence and protective immunity.. Journal of Clinical Investigation. 96(2). 965–975. 39 indexed citations
20.
Skare, Jon T. & Kathleen Postle. (1991). Evidence for a TonB‐dependent energy transduction complex in Escherichia coli. Molecular Microbiology. 5(12). 2883–2890. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026