Jon M. Nakamoto

1.5k total citations
26 papers, 977 citations indexed

About

Jon M. Nakamoto is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Genetics. According to data from OpenAlex, Jon M. Nakamoto has authored 26 papers receiving a total of 977 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 12 papers in Endocrinology, Diabetes and Metabolism and 12 papers in Genetics. Recurrent topics in Jon M. Nakamoto's work include Genetic Syndromes and Imprinting (7 papers), Sexual Differentiation and Disorders (5 papers) and Congenital heart defects research (4 papers). Jon M. Nakamoto is often cited by papers focused on Genetic Syndromes and Imprinting (7 papers), Sexual Differentiation and Disorders (5 papers) and Congenital heart defects research (4 papers). Jon M. Nakamoto collaborates with scholars based in United States, Greece and Finland. Jon M. Nakamoto's co-authors include Harvey W. Kaufman, Amy J. Blatt, Paul Herzmark, Taroh Iiri, Henry R. Bourne, Cornelis Van Dop, Geoffrey N. Hendy, Hiroyuki Koshiyama, Arlan L. Rosenbloom and Ólafur S. Indridason and has published in prestigious journals such as Nature, Journal of Clinical Investigation and The Journal of Clinical Endocrinology & Metabolism.

In The Last Decade

Jon M. Nakamoto

26 papers receiving 944 citations

Peers

Jon M. Nakamoto
Itsuro Hibi Japan
Ayako Tanae Japan
R. Rebourcet France
Ingrid Nermoen Norway
Felix Schreiner Germany
Armando Reyes‐Engel Spain
Karen D. Bradshaw United States
Kerstin Kirchhoff Germany
Nigel W. Rayner United Kingdom
John E. Nichols United States
Itsuro Hibi Japan
Jon M. Nakamoto
Citations per year, relative to Jon M. Nakamoto Jon M. Nakamoto (= 1×) peers Itsuro Hibi

Countries citing papers authored by Jon M. Nakamoto

Since Specialization
Citations

This map shows the geographic impact of Jon M. Nakamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon M. Nakamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon M. Nakamoto more than expected).

Fields of papers citing papers by Jon M. Nakamoto

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon M. Nakamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon M. Nakamoto. The network helps show where Jon M. Nakamoto may publish in the future.

Co-authorship network of co-authors of Jon M. Nakamoto

This figure shows the co-authorship network connecting the top 25 collaborators of Jon M. Nakamoto. A scholar is included among the top collaborators of Jon M. Nakamoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon M. Nakamoto. Jon M. Nakamoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Singh, Ila, et al.. (2021). Changes in Test Volumes During Coronavirus Disease 2019 (COVID-19): A Laboratory Stewardship Opportunity. Archives of Pathology & Laboratory Medicine. 145(7). 821–824. 7 indexed citations
2.
Regelmann, Molly O., Manmohan K. Kamboj, Bradley S. Miller, et al.. (2018). Adrenoleukodystrophy: Guidance for Adrenal Surveillance in Males Identified by Newborn Screen. The Journal of Clinical Endocrinology & Metabolism. 103(11). 4324–4331. 35 indexed citations
3.
4.
Yun, Yeo‐Min, Julianne Cook Botelho, Donald Walt Chandler, et al.. (2012). Performance Criteria for Testosterone Measurements Based on Biological Variation in Adult Males: Recommendations from the Partnership for the Accurate Testing of Hormones. Clinical Chemistry. 58(12). 1703–1710. 27 indexed citations
5.
Lekarev, Oksana, Andrew Lane, Guangyu Zhu, et al.. (2012). Erroneous prenatal diagnosis of congenital adrenal hyperplasia owing to a duplication of the CYP21A2 gene. Journal of Perinatology. 33(1). 76–78. 16 indexed citations
6.
Blatt, Amy J., Jon M. Nakamoto, & Harvey W. Kaufman. (2010). Gaps in Diabetes Screening During Pregnancy and Postpartum. Obstetrics and Gynecology. 117(1). 61–68. 78 indexed citations
7.
Nakamoto, Jon M.. (2009). Laboratory diagnosis of multiple pituitary hormone deficiencies: issues with testing of the growth and thyroid axes.. PubMed. 6 Suppl 2. 291–7. 1 indexed citations
8.
Mansukhani, Mahesh, Sharon E. Oberfield, Ilene Fennoy, et al.. (2007). Prenatal diagnosis of congenital lipoid adrenal hyperplasia (CLAH) by estriol amniotic fluid analysis and molecular genetic testing. Prenatal Diagnosis. 28(1). 11–14. 4 indexed citations
9.
10.
Pandian, Raj & Jon M. Nakamoto. (2004). Rational use of the laboratory for childhood and adult growth hormone deficiency. Clinics in Laboratory Medicine. 24(1). 141–174. 7 indexed citations
11.
Bastepe, Murat, Leopold F. Fröhlich, Geoffrey N. Hendy, et al.. (2003). Autosomal dominant pseudohypoparathyroidism type Ib is associated with a heterozygous microdeletion that likely disrupts a putative imprinting control element of GNAS. Journal of Clinical Investigation. 112(8). 1255–1263. 186 indexed citations
12.
Nakamoto, Jon M.. (2000). Myths and variations in normal pubertal development. Western Journal of Medicine. 172(3). 182–185. 11 indexed citations
13.
Kaiserman, Kevin B., Jon M. Nakamoto, Mitchell E. Geffner, & Edward R.B. McCabe. (1998). Minipuberty of infancy and adolescent pubertal function in adrenal hypoplasia congenita. The Journal of Pediatrics. 133(2). 300–302. 31 indexed citations
14.
Nakamoto, Jon M., et al.. (1998). Pseudohypoparathyroidism type Ia from maternal but not paternal transmission of a Gsα gene mutation. American Journal of Medical Genetics. 77(4). 261–267. 2 indexed citations
15.
Nakamoto, Jon M., et al.. (1996). Concurrent Hormone Resistance (Pseudohypoparathyroidism Type Ia) and Hormone Independence (Testotoxicosis) Caused by a Unique Mutation in the Gαs Gene. Biochemical and Molecular Medicine. 58(1). 18–24. 17 indexed citations
16.
Iiri, Taroh, et al.. (1994). Rapid GDP release from Gsα in patients with gain and loss of endocrine function. Nature. 371(6493). 164–168. 206 indexed citations
17.
Pelayo, J. C., et al.. (1994). A method for isolation of rat renal microvessels and mRNA localization. American Journal of Physiology-Renal Physiology. 267(3). F497–F503. 8 indexed citations
18.
Lippe, Barbara M. & Jon M. Nakamoto. (1993). Conventional and Nonconventional Uses of Growth Hormone. 179–235. 17 indexed citations
19.
Nakamoto, Jon M., et al.. (1992). Prevalence of three mutations in the Gsα gene among 24 families with pseudohypoparathyroidism type Ia. Biochemical and Biophysical Research Communications. 189(1). 343–349. 5 indexed citations
20.
Geffner, Mitchell E., et al.. (1991). Use of In Vitro Clonogenic Assays to Differentiate Acquired From Genetic Causes of Insulin Resistance. Diabetes. 40(1). 28–36. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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