John W. Crabb

14.7k total citations · 1 hit paper
213 papers, 11.2k citations indexed

About

John W. Crabb is a scholar working on Molecular Biology, Ophthalmology and Cell Biology. According to data from OpenAlex, John W. Crabb has authored 213 papers receiving a total of 11.2k indexed citations (citations by other indexed papers that have themselves been cited), including 144 papers in Molecular Biology, 67 papers in Ophthalmology and 35 papers in Cell Biology. Recurrent topics in John W. Crabb's work include Retinal Development and Disorders (60 papers), Retinal Diseases and Treatments (47 papers) and Retinoids in leukemia and cellular processes (25 papers). John W. Crabb is often cited by papers focused on Retinal Development and Disorders (60 papers), Retinal Diseases and Treatments (47 papers) and Retinoids in leukemia and cellular processes (25 papers). John W. Crabb collaborates with scholars based in United States, Germany and Japan. John W. Crabb's co-authors include Karen A. West, Masaru Miyagi, Joe G. Hollyfield, Xiaorong Gu, Robert G. Salomon, Arthur S. Polans, Mary E. Rayborn, Janina Buczyłko, Yan Lin and Kulwant S. Aulak and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

John W. Crabb

209 papers receiving 11.0k citations

Hit Papers

Drusen proteome analysis: An approach to the etiology of ... 2002 2026 2010 2018 2002 250 500 750

Peers

John W. Crabb
Comparison fields: 5 of 137
  • Molecular Biology 7.3k
  • Ophthalmology 3.2k
  • Cell Biology 1.6k
  • Cellular and Molecular Neuroscience 1.3k
  • Radiology, Nuclear Medicine and Imaging 1.3k
Replace Lincoln V. Johnson with:
Lincoln V. Johnson United States
Masaru Miyagi United States
Larry L. David United States
Leonard D. Kohn United States
Noa Noy United States
Mark R. Kelley United States
Didier Decaudin France
Nathanaël Larochette France
Christer Wernstedt Sweden
Lothar Schweigerer Germany
Lincoln V. Johnson United States View profile →
Citations per field, relative to John W. Crabb
John W. Crabb · 1×
Citations per year, relative to John W. Crabb
John W. Crabb · 1×

Countries citing papers authored by John W. Crabb

Since Specialization
Citations

This map shows the geographic impact of John W. Crabb's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John W. Crabb with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John W. Crabb more than expected).

Fields of papers citing papers by John W. Crabb

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John W. Crabb. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John W. Crabb. The network helps show where John W. Crabb may publish in the future.

Co-authorship network of co-authors of John W. Crabb

This figure shows the co-authorship network connecting the top 25 collaborators of John W. Crabb. A scholar is included among the top collaborators of John W. Crabb based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John W. Crabb. John W. Crabb is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 1
2 9
3
MYELIN-RELATED PROTEINS ARE DECREASED IN THE IMMEDIATE RETROLAMINAR OPTIC NERVE (ON) IN NON-HUMAN PRIMATES (NHP) IN EARLY EXPERIMENTAL GLAUCOMA (EEG)
1
4
Quantitative Proteomic Analysis of Non-Human Primate (NHP) Peripapillary (PP) Sclera in Early Experimental Glaucoma (EEG)
2
5
Significance of Hyaluronan in the Pathogenesis of Choroidal Neovascularization in Advanced Age-Related Macular Degeneration
1
6
Quantitative Proteomic Studies Implicate Mitochondrial Dysfunction in the Trabecular Meshwork in Glaucoma Pathology
1
7
Pentosidine and Carboxymethyllysine, Plasma Biomarkers for Age-Related Macular Degeneration
3
8
Comparison of Proteins in Dry and Wet AMD Bruch's Membrane
1
9
Quantitative Proteomic Analysis of Uveal Melanoma Tissues
1
10
Light Exposure Alters Carboxyethylpyrrole Modified Proteins in Rod Outer Segments
1
11
Does Lipofuscin Contain Protein? Amino Acid, Protein and Ultrastructural Analysis of Human Lipofuscin
2
12
Oxidative Modifications as Biomarkers for AMD
2
13
Modeling Early Stages of Age-Related Macular Degeneration (AMD) by Inducing Oxidative Stress in the RPE
1
14
Proteomics Suggests Glaucoma Pathogenesis May Involve Optic Nerve Peptidylarginine Deiminase
2
15
Fractionation of Retina for Proteomic Analysis.
0
16
Microsomal Glutathione S–Transferase 1 in the Retinal Pigment Epithelium: Protection Against Oxidative Stress and a Potential Role in Aging
2
17
Light Induced Protein Modifications and Lipid Oxidation Products in Rat Retina
3
18
Pigment epithelium-derived factor suppresses ischemia-induced retinal neovascularization and VEGF-induced migration and growth.
248
19
Molecular characterization of the mouse gene encoding cellular retinaldehyde-binding protein.
10
20 32

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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