John P. Wittschieben

1.2k total citations
14 papers, 1.0k citations indexed

About

John P. Wittschieben is a scholar working on Molecular Biology, Cancer Research and Plant Science. According to data from OpenAlex, John P. Wittschieben has authored 14 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Cancer Research and 5 papers in Plant Science. Recurrent topics in John P. Wittschieben's work include DNA Repair Mechanisms (9 papers), Carcinogens and Genotoxicity Assessment (7 papers) and Cancer therapeutics and mechanisms (5 papers). John P. Wittschieben is often cited by papers focused on DNA Repair Mechanisms (9 papers), Carcinogens and Genotoxicity Assessment (7 papers) and Cancer therapeutics and mechanisms (5 papers). John P. Wittschieben collaborates with scholars based in United States, Denmark and Israel. John P. Wittschieben's co-authors include Richard D. Wood, Birgitte Ø. Wittschieben, Gregory N. Gan, Stewart Shuman, Susanne M. Gollin, Shalini C. Reshmi, Sabine S. Lange, Errol C. Friedberg, Sigal Shachar and Sheera Adar and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

John P. Wittschieben

14 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
John P. Wittschieben United States	12	948	361	194	129	83	14	1.0k
Kenjiro Asagoshi Japan	17	952 1.0×	233 0.6×	182 0.9×	70 0.5×	87 1.0×	24	1.1k
Dmitry V. Bugreev Russia	17	1.4k 1.5×	305 0.8×	309 1.6×	194 1.5×	157 1.9×	30	1.4k
Jiang‐Cheng Shen United States	12	1.1k 1.2×	248 0.7×	129 0.7×	143 1.1×	184 2.2×	13	1.3k
Kelly M. Trujillo United States	15	1.9k 2.0×	381 1.1×	446 2.3×	200 1.6×	214 2.6×	19	2.0k
Mitsuo Wakasugi Japan	17	1.5k 1.6×	297 0.8×	276 1.4×	94 0.7×	158 1.9×	31	1.6k
Julia Karow United Kingdom	9	2.0k 2.1×	579 1.6×	257 1.3×	418 3.2×	200 2.4×	12	2.1k
Wendy Bussen United States	11	1.5k 1.6×	382 1.1×	376 1.9×	257 2.0×	141 1.7×	12	1.5k
Shoshana Squires United Kingdom	15	976 1.0×	280 0.8×	293 1.5×	80 0.6×	92 1.1×	18	1.1k
Brian S. Plosky United States	9	949 1.0×	287 0.8×	113 0.6×	63 0.5×	160 1.9×	10	992
Claire Lesca France	10	588 0.6×	121 0.3×	198 1.0×	36 0.3×	73 0.9×	10	654

Countries citing papers authored by John P. Wittschieben

Since Specialization

Citations

This map shows the geographic impact of John P. Wittschieben's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John P. Wittschieben with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John P. Wittschieben more than expected).

Fields of papers citing papers by John P. Wittschieben

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John P. Wittschieben. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John P. Wittschieben. The network helps show where John P. Wittschieben may publish in the future.

Co-authorship network of co-authors of John P. Wittschieben

This figure shows the co-authorship network connecting the top 25 collaborators of John P. Wittschieben. A scholar is included among the top collaborators of John P. Wittschieben based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John P. Wittschieben. John P. Wittschieben is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Lange, Sabine S., Ella Bedford, John P. Wittschieben, et al.. (2013). Dual role for mammalian DNA polymerase ζ in maintaining genome stability and proliferative responses. Proceedings of the National Academy of Sciences. 110(8). E687–96. 35 indexed citations

Lange, Sabine S., John P. Wittschieben, & Richard D. Wood. (2012). DNA polymerase zeta is required for proliferation of normal mammalian cells. Nucleic Acids Research. 40(10). 4473–4482. 49 indexed citations

Wittschieben, John P., et al.. (2010). Loss of DNA Polymerase ζ Enhances Spontaneous Tumorigenesis. Cancer Research. 70(7). 2770–2778. 58 indexed citations

Shachar, Sigal, Omer Ziv, Sharon Avkin, et al.. (2009). Two‐polymerase mechanisms dictate error‐free and error‐prone translesion DNA synthesis in mammals. The EMBO Journal. 28(4). 383–393. 228 indexed citations

Goff, Julie P., Donna Shields, Mineaki Seki, et al.. (2009). Lack of DNA Polymerase θ (POLQ) Radiosensitizes Bone Marrow Stromal CellsIn Vitroand Increases Reticulocyte Micronuclei after Total-Body Irradiation. Radiation Research. 172(2). 165–174. 60 indexed citations

Shachar, Sigal, Omer Ziv, Sharon Avkin, et al.. (2009). Two-polymerase mechanisms dictate error-free and error-prone translesion DNA synthesis in mammals. The EMBO Journal. 28(7). 992–992. 9 indexed citations

Gan, Gregory N., John P. Wittschieben, Birgitte Ø. Wittschieben, & Richard D. Wood. (2007). DNA polymerase zeta (pol ζ) in higher eukaryotes. Cell Research. 18(1). 174–183. 170 indexed citations

Wittschieben, John P., Shalini C. Reshmi, Susanne M. Gollin, & Richard D. Wood. (2006). Loss of DNA Polymerase ζ Causes Chromosomal Instability in Mammalian Cells. Cancer Research. 66(1). 134–142. 111 indexed citations

Wittschieben, John P., Mahmud K. K. Shivji, El–Nasir Lalani, et al.. (2000). Disruption of the developmentally regulated Rev3l gene causes embryonic lethality. Current Biology. 10(19). 1217–1220. 146 indexed citations

10.

Wittschieben, John P., Bent O. Petersen, & S. Shuman. (1998). Replacement of the active site tyrosine of vaccinia DNA topoisomerase by glutamate, cysteine or histidine converts the enzyme into a site-specific endonuclease. Nucleic Acids Research. 26(2). 490–496. 18 indexed citations

11.

Wittschieben, John P.. (1997). Mechanism of DNA transesterification by vaccinia topoisomerase: catalytic contributions of essential residues Arg-130, Gly-132, Tyr-136 and Lys-167. Nucleic Acids Research. 25(15). 3001–3008. 59 indexed citations

12.

Wang, Li‐Kai, John P. Wittschieben, & Stewart Shuman. (1997). Mutational Analysis of 26 Residues of Vaccinia DNA Topoisomerase Identifies Ser-204 as Important for DNA Binding and Cleavage. Biochemistry. 36(26). 7944–7950. 10 indexed citations

13.

Wittschieben, John P., et al.. (1996). Mutations Within a Conserved Region of Vaccinia Topoisomerase Affect the DNA Cleavage-Religation Equilibrium. Journal of Molecular Biology. 263(2). 181–195. 27 indexed citations

14.

Wittschieben, John P. & Stewart Shuman. (1994). Mutational analysis of vaccinia DNA topoisomerase defines amino acid residues essential for covalent catalysis.. Journal of Biological Chemistry. 269(47). 29978–29983. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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