John P. Pribis

1.7k total citations
19 papers, 914 citations indexed

About

John P. Pribis is a scholar working on Genetics, Molecular Biology and Immunology. According to data from OpenAlex, John P. Pribis has authored 19 papers receiving a total of 914 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Genetics, 6 papers in Molecular Biology and 6 papers in Immunology. Recurrent topics in John P. Pribis's work include Immune Response and Inflammation (5 papers), Bacterial Genetics and Biotechnology (4 papers) and Advanced Glycation End Products research (4 papers). John P. Pribis is often cited by papers focused on Immune Response and Inflammation (5 papers), Bacterial Genetics and Biotechnology (4 papers) and Advanced Glycation End Products research (4 papers). John P. Pribis collaborates with scholars based in United States, China and Israel. John P. Pribis's co-authors include Timothy R. Billiar, Juan B. Ochoa, Susan M. Rosenberg, Melanie J. Scott, P. J. Hastings, Yin Zhai, Xinmei Zhu, Yoram Vodovotz, Sodam Kim and Patricia Loughran and has published in prestigious journals such as Molecular Cell, PLoS ONE and Annals of Surgery.

In The Last Decade

John P. Pribis

19 papers receiving 895 citations

Peers

John P. Pribis
Comparison fields: 5 of 125
  • Molecular Biology 331
  • Immunology 227
  • Genetics 134
  • Clinical Biochemistry 125
  • Surgery 103
S Gollapudi United States
Manuel Conde Spain
Dustin Singer Switzerland
Jianling Li China
Janka Bábíčková Slovakia
John E. Burkhardt United States
Xiaojiao Zhang China
Xiaolei Hu China
S Gollapudi United States View profile →
Citations per field, relative to John P. Pribis
John P. Pribis · 1×
Citations per year, relative to John P. Pribis
John P. Pribis · 1×

Countries citing papers authored by John P. Pribis

Since Specialization
Citations

This map shows the geographic impact of John P. Pribis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John P. Pribis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John P. Pribis more than expected).

Fields of papers citing papers by John P. Pribis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John P. Pribis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John P. Pribis. The network helps show where John P. Pribis may publish in the future.

Co-authorship network of co-authors of John P. Pribis

This figure shows the co-authorship network connecting the top 25 collaborators of John P. Pribis. A scholar is included among the top collaborators of John P. Pribis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John P. Pribis. John P. Pribis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
# Title Journal Authors Indexed citations
1 ppGpp and RNA-polymerase backtracking guide antibiotic-induced mutable gambler cells Molecular Cell Yin Zhai, John P. Pribis et al. 14
2 Drugging evolution of antibiotic resistance at a regulatory network hub Science Advances Yin Zhai, John P. Pribis et al. 17
3 Stress-Induced Mutagenesis, Gambler Cells, and Stealth Targeting Antibiotic-Induced Evolution mBio John P. Pribis, Yin Zhai et al. 34
4 Two mechanisms of chromosome fragility at replication-termination sites in bacteria Science Advances Qian Mei, Devon M. Fitzgerald et al. 14
5 Gamblers: An Antibiotic-Induced Evolvable Cell Subpopulation Differentiated by Reactive-Oxygen-Induced General Stress Response Molecular Cell John P. Pribis, Libertad Garcı́a-Villada et al. 124
6 A checklist is associated with increased quality of reporting preclinical biomedical research: A systematic review PLoS ONE SeungHye Han, Tolani F. Olonisakin et al. 80
7 Holliday junction trap shows how cells use recombination and a junction-guardian role of RecQ helicase Science Advances Jun Xia, Qian Mei et al. 29
8 The HIV Protease Inhibitor Saquinavir Inhibits HMGBl-Driven Inflammation by Targeting the Interaction of Cathepsin V with TLR4/MyD88 Molecular Medicine John P. Pribis, Yousef Al‐Abed et al. 18
9 HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4 Arteriosclerosis Thrombosis and Vascular Biology Jingjing Cai, Hong Yuan et al. 62
10 Epsilon aminocaproic acid reduces blood transfusion and improves the coagulation test after pediatric open-heart surgery: a meta-analysis of 5 clinical trials. PubMed Jun Lü, Haoyu Meng et al. 17
11 The Central Role of Arginine Catabolism in T-Cell Dysfunction and Increased Susceptibility to Infection After Physical Injury Annals of Surgery Xinmei Zhu, John P. Pribis et al. 90
12 Identification of Pharmacological Modulators of HMGB1-Induced Inflammatory Response by Cell-Based Screening PLoS ONE Domokos Gerő, Petra Szoleczky et al. 31
13 Signaling of High Mobility Group Box 1 (HMGB1) through Toll-like Receptor 4 in Macrophages Requires CD14 Molecular Medicine Sodam Kim, Sun Young Kim et al. 138
14 Metabolomics in pneumonia and sepsis: an analysis of the GenIMS cohort study Intensive Care Medicine Christopher Seymour, Sachin Yende et al. 85
15 Oral intake of hydrogen-rich water inhibits intimal hyperplasia in arterialized vein grafts in rats Cardiovascular Research Qiang Sun, Tomohiro Kawamura et al. 43
16 New clinical score for disease activity at diagnosis in Langerhans cell histiocytosis The Korean Journal of Hematology Sun Young Kim, John P. Pribis et al. 2
17 Systemic Arginine Depletion After a Murine Model of Surgery or Trauma Journal of Parenteral and Enteral Nutrition John P. Pribis, Xinmei Zhu et al. 28
18 Nature of Myeloid Cells Expressing Arginase 1 in Peripheral Blood After Trauma The Journal of Trauma: Injury, Infection, and Critical Care P. Popovič, Mazen Zenati et al. 39
19 Stat 6-Dependent Induction of Myeloid Derived Suppressor Cells After Physical Injury Regulates Nitric Oxide Response to Endotoxin Annals of Surgery P. Popovič, John P. Pribis et al. 49

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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