John P. Mathis

800 total citations
17 papers, 710 citations indexed

About

John P. Mathis is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, John P. Mathis has authored 17 papers receiving a total of 710 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Cellular and Molecular Neuroscience, 16 papers in Molecular Biology and 4 papers in Physiology. Recurrent topics in John P. Mathis's work include Neuropeptides and Animal Physiology (16 papers), Receptor Mechanisms and Signaling (15 papers) and Hypothalamic control of reproductive hormones (3 papers). John P. Mathis is often cited by papers focused on Neuropeptides and Animal Physiology (16 papers), Receptor Mechanisms and Signaling (15 papers) and Hypothalamic control of reproductive hormones (3 papers). John P. Mathis collaborates with scholars based in United States. John P. Mathis's co-authors include Gavril W. Pasternak, Iris Lindberg, Grace C. Rossi, Jennifer P. Ryan‐Moro, Sharon R. Letchworth, Liza Leventhal, Nabil G. Seidah, Claude Lazure, Mary B. Breslin and Suzanne Benjannet and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Comparative Neurology and Brain Research.

In The Last Decade

John P. Mathis

17 papers receiving 691 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John P. Mathis United States 13 561 461 241 95 85 17 710
S. Zakarian United Kingdom 11 628 1.1× 465 1.0× 162 0.7× 32 0.3× 93 1.1× 15 852
H Bonin Canada 7 321 0.6× 555 1.2× 145 0.6× 60 0.6× 10 0.1× 8 680
Kazuhisa Kashimoto Japan 14 420 0.7× 338 0.7× 107 0.4× 13 0.1× 75 0.9× 32 608
Jean‐Paul Humbert France 12 178 0.3× 384 0.8× 46 0.2× 104 1.1× 88 1.0× 13 571
Ryutaro Moriyama Japan 12 81 0.1× 352 0.8× 87 0.4× 140 1.5× 110 1.3× 27 605
R H Goodman United States 7 361 0.6× 421 0.9× 49 0.2× 47 0.5× 42 0.5× 10 824
Joy S. C. Chan Hong Kong 10 322 0.6× 424 0.9× 91 0.4× 25 0.3× 11 0.1× 14 514
Jean F. Schaefer United States 7 197 0.4× 190 0.4× 103 0.4× 12 0.1× 36 0.4× 13 451
Iris Wulfsen Germany 15 274 0.5× 580 1.3× 61 0.3× 26 0.3× 22 0.3× 17 704
D. Gully France 12 727 1.3× 597 1.3× 83 0.3× 6 0.1× 171 2.0× 17 818

Countries citing papers authored by John P. Mathis

Since Specialization
Citations

This map shows the geographic impact of John P. Mathis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John P. Mathis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John P. Mathis more than expected).

Fields of papers citing papers by John P. Mathis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John P. Mathis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John P. Mathis. The network helps show where John P. Mathis may publish in the future.

Co-authorship network of co-authors of John P. Mathis

This figure shows the co-authorship network connecting the top 25 collaborators of John P. Mathis. A scholar is included among the top collaborators of John P. Mathis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John P. Mathis. John P. Mathis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
2.
Mathis, John P., et al.. (2001). Orphanin FQ/nociceptin and naloxone benzoylhydrazone activate distinct receptors in BE(2)-C human neuroblastoma cells. Neuroscience Letters. 299(3). 173–176. 10 indexed citations
3.
Silva, Robert M., Grace C. Rossi, John P. Mathis, et al.. (2000). Morphine and morphine-6β-glucuronide-induced feeding are differentially reduced by G-protein α-subunit antisense probes in rats. Brain Research. 876(1-2). 62–75. 20 indexed citations
4.
Letchworth, Sharon R., John P. Mathis, Grace C. Rossi, Richard J. Bodnar, & Gavril W. Pasternak. (2000). Autoradiographic localization of 125I[Tyr14]orphanin FQ/nociceptin and 125I[Tyr10]orphanin FQ/nociceptin(1–11) binding sites in rat brain. The Journal of Comparative Neurology. 423(2). 319–329. 3 indexed citations
5.
Letchworth, Sharon R., John P. Mathis, Grace C. Rossi, Richard J. Bodnar, & Gavril W. Pasternak. (2000). Autoradiographic localization of125I[Tyr14]orphanin FQ/nociceptin and125I[Tyr10]orphanin FQ/nociceptin(1-11) binding sites in rat brain. The Journal of Comparative Neurology. 423(2). 319–329. 43 indexed citations
6.
Mathis, John P., et al.. (1999). Identification of a high-affinity orphanin FQ/nociceptin(1-11) binding site in mouse brain. Synapse. 34(3). 181–186. 19 indexed citations
7.
Rossi, Grace C., Sharon R. Letchworth, John P. Mathis, et al.. (1998). Pharmacological characterization of Endomorphin-1 and Endomorphin-2 in Mouse Brain. Journal of Pharmacology and Experimental Therapeutics. 286(2). 1007–1013. 166 indexed citations
8.
Mathis, John P., et al.. (1998). Antinociceptive analogs of orphanin FQ/nociceptin(1–11). Life Sciences. 63(11). A161–A166. 18 indexed citations
9.
Leventhal, Liza, John P. Mathis, Grace C. Rossi, Gavril W. Pasternak, & Richard J. Bodnar. (1998). Orphan opioid receptor antisense probes block orphanin FQ-induced hyperphagia. European Journal of Pharmacology. 349(1). R1–R3. 47 indexed citations
10.
Mathis, John P., et al.. (1997). Biochemical Evidence for Orphanin FQ/Nociceptin Receptor Heterogeneity in Mouse Brain. Biochemical and Biophysical Research Communications. 230(2). 462–465. 107 indexed citations
11.
Johanning, Karla, John P. Mathis, & Iris Lindberg. (1996). Role of PC2 in Proenkephalin Processing: Antisense and Overexpression Studies. Journal of Neurochemistry. 66(3). 898–907. 37 indexed citations
12.
Brooks, Andrew I., Kelly M. Standifer, Grace C. Rossi, John P. Mathis, & Gavril W. Pasternak. (1996). Characterizing ?3 opioid receptors with a selective monoclonal antibody. Synapse. 22(3). 247–252. 12 indexed citations
13.
Johanning, Karla, John P. Mathis, & Iris Lindberg. (1996). Processing Site Blockade Results in More Efficient Conversion of Proenkephalin to Active Opioid Peptides. Journal of Biological Chemistry. 271(44). 27871–27878. 9 indexed citations
14.
Hornby, Pamela J., Spencer Rosenthal, John P. Mathis, Osvaldo Vindrola, & Iris Lindberg. (1993). Immunocytochemical Localization of the Neuropeptide-Synthesizing Enzyme PC1 in AtT-20 Cells. Neuroendocrinology. 58(5). 555–563. 25 indexed citations
15.
Breslin, Mary B., Iris Lindberg, Suzanne Benjannet, et al.. (1993). Differential processing of proenkephalin by prohormone convertases 1(3) and 2 and furin.. Journal of Biological Chemistry. 268(36). 27084–27093. 132 indexed citations
16.
Mathis, John P. & Iris Lindberg. (1992). Posttranslational processing of proenkephalin in AtT-20 cells: evidence for cleavage at a Lys-Lys site.. Endocrinology. 131(5). 2287–2296. 36 indexed citations
17.
Mathis, John P.. (1992). Posttranslational processing of proenkephalin in AtT-20 cells: evidence for cleavage at a Lys-Lys site. Endocrinology. 131(5). 2287–2296. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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