John Mowbray
Impact in
- Clinical Biochemistry top 1%
- Metabolism and Genetic Disorders
- Biochemistry top 5%
Papers in
-
- Metabolism and Genetic Disorders 17
-
- Adenosine and Purinergic Signaling 7
- Adipose Tissue and Metabolism 4
- Co-authors
- J. H. OttawayVivian MosesDavid BatesDavid I. PerrettWendy E. ThomasW. L. HutchinsonM J HolnessPeter G. Morris
- Journals
- Biochemical Journal (11 papers)Biochemical Society Transactions (11 papers)European Journal of Biochemistry (9 papers)FEBS Letters (7 papers)Bioscience Reports (2 papers)
- Partner nations
- United KingdomVenezuelaUnited States
In The Last Decade
John Mowbray
57 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 106
- Clinical Biochemistry 262
- Biochemistry 136
- Physiology 85
- Molecular Biology 814
- Physiology 301
Countries citing papers authored by John Mowbray
This map shows the geographic impact of John Mowbray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Mowbray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Mowbray more than expected).
Fields of papers citing papers by John Mowbray
This network shows the impact of papers produced by John Mowbray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Mowbray. The network helps show where John Mowbray may publish in the future.
Co-authorship network
The 25 scholars most cited alongside John Mowbray, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2002 | 4 | |
| 2 | 1998 | 1 | |
| 3 | 1996 | 19 | |
| 4 | 1994 | 5 | |
| 5 | 1993 | 1 | |
| 6 | 1991 | 3 | |
| 7 | 1987 | 11 | |
| 8 | 1987 | 2 | |
| 9 | 1987 | 25 | |
| 10 | 1987 | 1 | |
| 11 | 1983 | 4 | |
| 12 | 1981 | 12 | |
| 13 | 1980 | 1 | |
| 14 | 1979 | 6 | |
| 15 | 1978 | 27 | |
| 16 | 1978 | 8 | |
| 17 | 1975 | 46 | |
| 18 | 1974 | 26 | |
| 19 | 1973 | 39 | |
| 20 | 1962 | 1 |
About John Mowbray
John Mowbray is a scholar working on Clinical Biochemistry, Physiology, Biochemistry, Molecular Biology and Cell Biology, having authored 57 papers that have together received 1.2k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (19 papers), Metabolism and Genetic Disorders (17 papers), Ion channel regulation and function (9 papers), Adenosine and Purinergic Signaling (7 papers), Muscle metabolism and nutrition (5 papers), RNA and protein synthesis mechanisms (5 papers), Amino Acid Enzymes and Metabolism (4 papers) and Adipose Tissue and Metabolism (4 papers). The work is most often cited by research in Clinical Biochemistry (262 citations), Biochemistry (136 citations), Physiology (85 citations), Molecular Biology (814 citations) and Physiology (301 citations). John Mowbray has collaborated with scholars based in United Kingdom, Venezuela and United States. Frequent co-authors include J. H. Ottaway, Vivian Moses, David Bates, David I. Perrett, Wendy E. Thomas, W. L. Hutchinson, M J Holness, Peter G. Morris, John M. Land and J. B. Clark. Their work appears in journals such as Biochemical Journal, Biochemical Society Transactions, European Journal of Biochemistry, FEBS Letters and Bioscience Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.