John F. Cooper

1.2k total citations
20 papers, 832 citations indexed

About

John F. Cooper is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, John F. Cooper has authored 20 papers receiving a total of 832 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Oncology and 3 papers in Surgery. Recurrent topics in John F. Cooper's work include Receptor Mechanisms and Signaling (6 papers), Nicotinic Acetylcholine Receptors Study (6 papers) and Ion channel regulation and function (5 papers). John F. Cooper is often cited by papers focused on Receptor Mechanisms and Signaling (6 papers), Nicotinic Acetylcholine Receptors Study (6 papers) and Ion channel regulation and function (5 papers). John F. Cooper collaborates with scholars based in United States. John F. Cooper's co-authors include Jon Lindstrom, Mark E. Nelson, Alexander Kuryatov, Yan Zhou, Catherine H. Choi, Fan Wang, Kent T. Keyser, Felix Olale, Paul Whiteaker and Socrates J. Tzartos and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and Biochemistry.

In The Last Decade

John F. Cooper

20 papers receiving 788 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John F. Cooper United States 14 561 174 79 76 64 20 832
Changcheng Song United States 13 590 1.1× 103 0.6× 45 0.6× 161 2.1× 21 0.3× 16 870
Brian D. Bennett United States 19 770 1.4× 74 0.4× 83 1.1× 29 0.4× 19 0.3× 46 1.0k
Marius P. Sumandea United States 20 921 1.6× 51 0.3× 38 0.5× 48 0.6× 25 0.4× 27 1.6k
Anders Björk Sweden 10 439 0.8× 80 0.5× 212 2.7× 43 0.6× 33 0.5× 11 860
Darshana Patel United States 16 399 0.7× 235 1.4× 40 0.5× 109 1.4× 32 0.5× 20 856
Masahiro Mukaida Japan 15 247 0.4× 35 0.2× 20 0.3× 72 0.9× 35 0.5× 53 635
R C Venema United States 12 461 0.8× 93 0.5× 32 0.4× 29 0.4× 10 0.2× 16 824
Julie Acunzo France 8 441 0.8× 93 0.5× 53 0.7× 55 0.7× 16 0.3× 10 615
Wance Firdaus France 8 302 0.5× 65 0.4× 62 0.8× 69 0.9× 14 0.2× 8 506
N. A. Rao United States 16 268 0.5× 42 0.2× 33 0.4× 100 1.3× 12 0.2× 29 811

Countries citing papers authored by John F. Cooper

Since Specialization
Citations

This map shows the geographic impact of John F. Cooper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John F. Cooper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John F. Cooper more than expected).

Fields of papers citing papers by John F. Cooper

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John F. Cooper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John F. Cooper. The network helps show where John F. Cooper may publish in the future.

Co-authorship network of co-authors of John F. Cooper

This figure shows the co-authorship network connecting the top 25 collaborators of John F. Cooper. A scholar is included among the top collaborators of John F. Cooper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John F. Cooper. John F. Cooper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Monaco, Maria Chiara, Samantha S. Soldan, Chenhe Su, et al.. (2023). EBNA1 Inhibitors Block Proliferation of Spontaneous Lymphoblastoid Cell Lines From Patients With Multiple Sclerosis and Healthy Controls. Neurology Neuroimmunology & Neuroinflammation. 10(5). 12 indexed citations
2.
Lucero, Linda, Maegan M. Weltzin, J. Brek Eaton, et al.. (2015). Differential α4(+)/(−)β2 Agonist-binding Site Contributions to α4β2 Nicotinic Acetylcholine Receptor Function within and between Isoforms. Journal of Biological Chemistry. 291(5). 2444–2459. 24 indexed citations
3.
Eaton, J. Brek, Linda Lucero, Harrison J. Stratton, et al.. (2013). The Unique α4(+)/(−)α4 Agonist Binding Site in (α4)3(β2)2 Subtype Nicotinic Acetylcholine Receptors Permits Differential Agonist Desensitization Pharmacology versus the (α4)2(β2)3 Subtype. Journal of Pharmacology and Experimental Therapeutics. 348(1). 46–58. 49 indexed citations
4.
Marks, Michael J., Tristan McClure‐Begley, Paul Whiteaker, et al.. (2011). Increased Nicotinic Acetylcholine Receptor Protein Underlies Chronic Nicotine-Induced Up-Regulation of Nicotinic Agonist Binding Sites in Mouse Brain. Journal of Pharmacology and Experimental Therapeutics. 337(1). 187–200. 56 indexed citations
5.
Whiteaker, Paul, John F. Cooper, Outi Salminen, et al.. (2006). Immunolabeling demonstrates the interdependence of mouse brain α4 and β2 nicotinic acetylcholine receptor subunit expression. The Journal of Comparative Neurology. 499(6). 1016–1038. 42 indexed citations
6.
Zhou, Yan, Mark E. Nelson, Alexander Kuryatov, et al.. (2003). Human α4β2 Acetylcholine Receptors Formed from Linked Subunits. Journal of Neuroscience. 23(27). 9004–9015. 145 indexed citations
7.
Cooper, John F., Wyland F. Leadbetter, & Richard Chute. (2002). The thoracoabdominal approach for retroperitoneal gland dissection: Its application to testis tumors. The Journal of Urology. 167(2 Part 2). 920–926. 10 indexed citations
8.
Wang, Fan, Mark E. Nelson, Alexander Kuryatov, et al.. (1998). Chronic Nicotine Treatment Up-regulates Human α3β2 but Not α3β4 Acetylcholine Receptors Stably Transfected in Human Embryonic Kidney Cells. Journal of Biological Chemistry. 273(44). 28721–28732. 158 indexed citations
9.
Cooper, John F., Michael J. Lichtenstein, Barney S. Graham, & William Schaffner. (1989). Mycobacterium chelonae: A cause of nodular skin lesions with a proclivity for renal transplant recipients. The American Journal of Medicine. 86(2). 173–177. 64 indexed citations
10.
Whiting, Paul J., John F. Cooper, & Jon Lindstrom. (1987). Antibodies in sera from patients with myasthenia gravis do not bind to nicotinic acetylcholine receptors from human brain. Journal of Neuroimmunology. 16(2). 205–213. 36 indexed citations
11.
Lindstrom, Jon, John F. Cooper, & Socrates J. Tzartos. (1980). Acetylcholine receptors from Torpedo and Electrophorus have similar subunit structures. Biochemistry. 19(7). 1454–1458. 61 indexed citations
12.
Hurwitz, Richard S., et al.. (1980). A Neuro-Ectodermal Tumor in the Bladder. The Journal of Urology. 124(3). 417–421. 13 indexed citations
13.
Cooper, John F.. (1980). The Radioimmunochemical Measurement of Prostatic Acid Phosphatase: Current State of the Art. Urologic Clinics of North America. 7(3). 653–665. 24 indexed citations
14.
Cooper, John F. & William D. Finkle. (1980). Current experience with radioimmunoassay techniques for prostatic acid phosphatase. The Prostate. 1(4). 441–450. 4 indexed citations
15.
Cooper, John F., et al.. (1978). Radioimmunochemical Measurement of Bone Marrow Prostatic Acid Phosphatase. The Journal of Urology. 119(3). 392–395. 17 indexed citations
16.
Cooper, John F., et al.. (1967). The Technique of Tetracycline Fluorescence in Urinary Sediments. The Journal of Urology. 98(3). 397–401. 2 indexed citations
17.
Cooper, John F., et al.. (1967). The Productivity of Prostatic Needle Biopsy in the Recognition of Early Prostatic Cancer. The Journal of Urology. 97(5). 894–898. 3 indexed citations
18.
19.
Cooper, John F., et al.. (1959). The Role of Citric Acid in the Physiology of the Prostate: A Preliminary Report. The Journal of Urology. 81(1). 157–164. 19 indexed citations
20.
Cooper, John F., et al.. (1952). Uretero-Enterostomy: Experimental Studies. The Journal of Urology. 67(6). 873–879. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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