John E. Harris

2.5k total citations
98 papers, 2.0k citations indexed

About

John E. Harris is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, John E. Harris has authored 98 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Physiology, 32 papers in Molecular Biology and 26 papers in Cellular and Molecular Neuroscience. Recurrent topics in John E. Harris's work include Pain Mechanisms and Treatments (30 papers), Neuroscience and Neuropharmacology Research (22 papers) and Ion channel regulation and function (8 papers). John E. Harris is often cited by papers focused on Pain Mechanisms and Treatments (30 papers), Neuroscience and Neuropharmacology Research (22 papers) and Ion channel regulation and function (8 papers). John E. Harris collaborates with scholars based in United Kingdom, United States and Ireland. John E. Harris's co-authors include R.W. Clarke, Paul W. Brand, Barry P. Clark, Ann E. Kingston, Victoria Chapman, Liam Drew, Stephen Baker, John Goldsworthy, C. E. Ford and T. W. Ford and has published in prestigious journals such as Nature, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

John E. Harris

97 papers receiving 1.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John E. Harris United Kingdom 26 636 612 453 237 156 98 2.0k
Paul Goldsmith United Kingdom 29 643 1.0× 1.4k 2.3× 318 0.7× 193 0.8× 45 0.3× 86 3.8k
Jennifer R. Deuis Australia 26 523 0.8× 1.2k 1.9× 888 2.0× 252 1.1× 98 0.6× 68 2.5k
Yûji Takahashi Japan 37 1.2k 1.9× 1.8k 3.0× 238 0.5× 104 0.4× 57 0.4× 191 5.5k
Olavi Eränkö Finland 31 1.2k 2.0× 1.1k 1.7× 634 1.4× 265 1.1× 50 0.3× 126 3.1k
David Erlij United States 31 1.2k 1.8× 1.6k 2.5× 385 0.8× 184 0.8× 44 0.3× 104 3.1k
Jerzy Z. Nowak Poland 24 1.1k 1.8× 1.5k 2.5× 273 0.6× 162 0.7× 24 0.2× 159 3.4k
Thomas V. Getchell United States 42 1.7k 2.6× 650 1.1× 403 0.9× 424 1.8× 57 0.4× 107 4.3k
Ewan St. John Smith United Kingdom 29 659 1.0× 1.1k 1.8× 975 2.2× 138 0.6× 48 0.3× 87 2.8k
C.E. Devine New Zealand 33 443 0.7× 1.3k 2.1× 427 0.9× 43 0.2× 266 1.7× 76 3.6k
Ken‐ichiro Kubo Japan 34 1.2k 1.9× 1.8k 2.9× 313 0.7× 107 0.5× 42 0.3× 93 4.1k

Countries citing papers authored by John E. Harris

Since Specialization
Citations

This map shows the geographic impact of John E. Harris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John E. Harris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John E. Harris more than expected).

Fields of papers citing papers by John E. Harris

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John E. Harris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John E. Harris. The network helps show where John E. Harris may publish in the future.

Co-authorship network of co-authors of John E. Harris

This figure shows the co-authorship network connecting the top 25 collaborators of John E. Harris. A scholar is included among the top collaborators of John E. Harris based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John E. Harris. John E. Harris is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hunt, James, Helen Jenkins, John E. Harris, et al.. (2019). Clinical measurements performed during alfaxalone total intravenous anaesthesia for radiography and neurophysiological investigations in dogs. Veterinary Anaesthesia and Analgesia. 46(4). 483–487. 3 indexed citations
2.
White, Kate, Stuart W. Paine, & John E. Harris. (2017). A clinical evaluation of the pharmacokinetics and pharmacodynamics of intravenous alfaxalone in cyclodextrin in male and female rats following a loading dose and constant rate infusion. Veterinary Anaesthesia and Analgesia. 44(4). 865–875. 19 indexed citations
3.
Kelly, Sara, et al.. (2013). Spinal nociceptive reflexes are sensitized in the monosodium iodoacetate model of osteoarthritis pain in the rat. Osteoarthritis and Cartilage. 21(9). 1327–1335. 34 indexed citations
4.
Green, Martin, et al.. (2011). Risk factors associated with hair loss, ulceration, and swelling at the hock in freestall-housed UK dairy herds. Journal of Dairy Science. 94(6). 2952–2963. 60 indexed citations
5.
Harris, John E. & R.W. Clarke. (2006). Site-specific, inflammation-induced adaptations in withdrawal reflex pathways in the anesthetized rabbit. Brain Research. 1131(1). 106–111. 3 indexed citations
6.
Harris, John E., et al.. (2005). Valley "Ultra-Thin" - a novel alpha-amylase for true pH 4.5 liquefaction.. International sugar journal. 107(1283). 616–621. 1 indexed citations
7.
Harris, John E., et al.. (2004). Glutamate and tachykinin receptors in central sensitization of withdrawal reflexes in the decerebrated rabbit. Experimental Physiology. 89(2). 187–198. 11 indexed citations
8.
Jenkins, Sarah, Mark Worthington, John E. Harris, & R.W. Clarke. (2004). Differential modulation of withdrawal reflexes by a cannabinoid in the rabbit. Brain Research. 1012(1-2). 146–153. 12 indexed citations
9.
Becker, Christoph, et al.. (2003). Differences in the Activation Mechanism between the α and β Subunits of Human Meprin. Biological Chemistry. 384(5). 825–31. 77 indexed citations
10.
Harris, John E., et al.. (2002). Diffuse organization of central sensitization of withdrawal reflexes in the decerebrated, spinalized rabbit. Proceedings of The Physiological Society. 2 indexed citations
11.
12.
Clarke, R.W. & John E. Harris. (2001). The spatial organization of central sensitization of hind limb flexor reflexes in the decerebrated, spinalized rabbit. European Journal of Pain. 5(2). 175–185. 15 indexed citations
13.
Clarke, R.W., et al.. (2001). Adaptive changes in withdrawal reflexes after noxious stimulation at the heel and the toes in the decerebrated rabbit. Neuroscience Letters. 304(1-2). 120–122. 10 indexed citations
14.
Harris, John E., Liam Drew, & Victoria Chapman. (2000). Spinal anandamide inhibits nociceptive transmission via cannabinoid receptor activation in vivo. Neuroreport. 11(12). 2817–2819. 33 indexed citations
15.
Kingston, Anne, Michael J. O’Neill, Valeria Bruno, et al.. (1999). Neuroprotective Actions of Novel and Potent Ligands of Group I and Group II Metabotropic Glutamate Receptors. Annals of the New York Academy of Sciences. 890(1). 438–449. 47 indexed citations
16.
Harris, John E. & R.W. Clarke. (1993). Motor and cardiovascular effects of selective α2-adrenoceptor antagonists in the decerebrated rabbit. European Journal of Pharmacology. 237(2-3). 323–328. 14 indexed citations
17.
Clarke, R.W., John E. Harris, T. W. Ford, & Julian Taylor. (1992). Prolonged potentiation of transmission through a withdrawal reflex pathway after noxious stimulation of the heel in the rabbit. Pain. 49(1). 65–70. 18 indexed citations
18.
Harris, John E. & R.W. Clarke. (1992). An analysis of adrenergic influences on the sural-gastrocnemius reflex of the decerebrated rabbit. Experimental Brain Research. 92(2). 310–7. 12 indexed citations
19.
Harris, John E., et al.. (1991). Opioidergic inhibition of reflexes evoked by selective stimulation of sural nerve C fibres in the rabbit. Experimental Physiology. 76(6). 987–990. 3 indexed citations
20.
Harris, John E., et al.. (1973). The reversal of triparanol-induced cataracts in the rat. 3. Amino acid content and uptake of 14 C -AIB in cataractous and clearing lenses.. PubMed. 12(5). 385–8. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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