John D. Lee

3.8k total citations
66 papers, 2.0k citations indexed

About

John D. Lee is a scholar working on Neurology, Immunology and Molecular Biology. According to data from OpenAlex, John D. Lee has authored 66 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Neurology, 26 papers in Immunology and 22 papers in Molecular Biology. Recurrent topics in John D. Lee's work include Amyotrophic Lateral Sclerosis Research (25 papers), Complement system in diseases (25 papers) and Neuroinflammation and Neurodegeneration Mechanisms (19 papers). John D. Lee is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (25 papers), Complement system in diseases (25 papers) and Neuroinflammation and Neurodegeneration Mechanisms (19 papers). John D. Lee collaborates with scholars based in Australia, United States and United Kingdom. John D. Lee's co-authors include Trent M. Woodruff, Junyan Han, Richard J. Ulevitch, Peter G. Noakes, Xaria X. Li, Vinod Kumar, Liam G. Coulthard, Richard J. Clark, Jenny N. Fung and Marc J. Ruitenberg and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and SHILAP Revista de lepidopterología.

In The Last Decade

John D. Lee

58 papers receiving 2.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John D. Lee Australia 25 800 618 583 574 329 66 2.0k
Geneviève Soucy Canada 21 674 0.8× 479 0.8× 442 0.8× 592 1.0× 324 1.0× 34 2.0k
Refik Pul Germany 26 509 0.6× 493 0.8× 764 1.3× 359 0.6× 244 0.7× 94 2.2k
Isabella Wimmer Austria 18 653 0.8× 541 0.9× 990 1.7× 459 0.8× 182 0.6× 25 2.3k
Stefano Fumagalli Italy 25 580 0.7× 814 1.3× 1.1k 1.9× 405 0.7× 152 0.5× 46 2.2k
Shalina S. Ousman Canada 21 1.0k 1.3× 748 1.2× 728 1.2× 248 0.4× 358 1.1× 40 2.8k
Fiona M. Menzies United Kingdom 16 764 1.0× 273 0.4× 177 0.3× 549 1.0× 317 1.0× 25 1.8k
Azeb Tadesse Argaw United States 14 724 0.9× 341 0.6× 1.1k 1.9× 245 0.4× 203 0.6× 16 2.2k
Tamara Berkutzki Israel 11 481 0.6× 640 1.0× 677 1.2× 159 0.3× 300 0.9× 12 1.8k
Djordje Gverić United Kingdom 19 598 0.7× 783 1.3× 816 1.4× 390 0.7× 222 0.7× 27 2.5k
Yuji Nakatsuji Japan 28 904 1.1× 470 0.8× 417 0.7× 578 1.0× 222 0.7× 94 2.3k

Countries citing papers authored by John D. Lee

Since Specialization
Citations

This map shows the geographic impact of John D. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John D. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John D. Lee more than expected).

Fields of papers citing papers by John D. Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John D. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John D. Lee. The network helps show where John D. Lee may publish in the future.

Co-authorship network of co-authors of John D. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of John D. Lee. A scholar is included among the top collaborators of John D. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John D. Lee. John D. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fung, Jenny N., et al.. (2025). A detrimental role for complement C5aR1 activation in Huntington’s disease. Immunobiology. 230(4). 153080–153080.
2.
Woodruff, Trent M., et al.. (2025). Investigating the role of neuronally-expressed C5aR1 in motor neuron disease. Immunobiology. 230(4). 153079–153079.
3.
Fung, Jenny N., et al.. (2025). Complement C3a receptor signalling mitigates motor neuron degeneration by modulating neurotoxic astrocytes. Immunobiology. 230(4). 153081–153081.
4.
Fung, Jenny N., John D. Lee, & Trent M. Woodruff. (2025). Targeting C5a inflammation in ALS: Ex vivo findings from PMX205 phase Ib trial. Immunobiology. 230(4). 153003–153003.
5.
Fung, Jenny N., John D. Lee, Robert Adam, John D. O’Sullivan, & Trent M. Woodruff. (2025). Peripheral and central elevation of IL-8 in patients with Huntington’s disease. Molecular Immunology. 179. 84–93.
6.
Li, Xaria X., Rabina Giri, Vinod Kumar, et al.. (2025). Colon‐targeted complement C5a1 receptor inhibition using pH‐sensitive nanoparticles ameliorates experimental colitis. British Journal of Pharmacology. 182(16). 3852–3869. 2 indexed citations
7.
Cianciulli, Antonia, Liam G. Coulthard, Owen A. Hawksworth, et al.. (2023). Complement peptide receptors in GtoPdb v.2023.1. IUPHAR/BPS Guide to Pharmacology CITE. 2023(1). 1 indexed citations
8.
McDonald, Tanya S., et al.. (2022). Potential mechanisms to modify impaired glucose metabolism in neurodegenerative disorders. Journal of Cerebral Blood Flow & Metabolism. 43(1). 26–43. 19 indexed citations
9.
Amarilla, Alberto A., John D. Lee, Eduardo A. Albornoz, et al.. (2022). SARS‐CoV ‐2 triggers complement activation through interactions with heparan sulfate. Clinical & Translational Immunology. 11(8). e1413–e1413. 13 indexed citations
10.
Kumar, Vinod, et al.. (2021). In Vivo Pharmacodynamic Method to Assess Complement C5a Receptor Antagonist Efficacy. ACS Pharmacology & Translational Science. 5(1). 41–51. 5 indexed citations
11.
Li, Xaria X., John D. Lee, Jenny N. Fung, et al.. (2021). Development of Potent and Selective Agonists for Complement C5a Receptor 1 with In Vivo Activity. Journal of Medicinal Chemistry. 64(22). 16598–16608. 11 indexed citations
12.
Lee, John D., et al.. (2021). Chemical synthesis and characterisation of the complement C5 inhibitory peptide zilucoplan. Amino Acids. 53(1). 143–147. 14 indexed citations
13.
Wu, Mike C. L., John D. Lee, Marc J. Ruitenberg, & Trent M. Woodruff. (2020). Absence of the C5a Receptor C5aR2 Worsens Ischemic Tissue Injury by Increasing C5aR1-Mediated Neutrophil Infiltration. The Journal of Immunology. 205(10). 2834–2839. 22 indexed citations
14.
Lee, John D., Tanya S. McDonald, Jenny N. Fung, & Trent M. Woodruff. (2020). Absence of Receptor for Advanced Glycation End Product (RAGE) Reduces Inflammation and Extends Survival in the hSOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis. Molecular Neurobiology. 57(10). 4143–4155. 25 indexed citations
15.
McDonald, Tanya S., Pamela McCombe, Trent M. Woodruff, & John D. Lee. (2020). The potential interplay between energy metabolism and innate complement activation in amyotrophic lateral sclerosis. The FASEB Journal. 34(6). 7225–7233. 8 indexed citations
16.
Lee, John D., et al.. (2019). Therapeutic blockade of HMGB1 reduces early motor deficits, but not survival in the SOD1G93A mouse model of amyotrophic lateral sclerosis. Journal of Neuroinflammation. 16(1). 45–45. 20 indexed citations
17.
Li, Xaria X., John D. Lee, Claudia Kemper, & Trent M. Woodruff. (2019). The Complement Receptor C5aR2: A Powerful Modulator of Innate and Adaptive Immunity. The Journal of Immunology. 202(12). 3339–3348. 116 indexed citations
18.
Lee, John D., Eduardo A. Albornoz, Luke McAlary, et al.. (2019). The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins. Glia. 68(2). 407–421. 154 indexed citations
19.
Kumar, Vinod, John D. Lee, Richard J. Clark, & Trent M. Woodruff. (2018). Development and validation of a LC-MS/MS assay for pharmacokinetic studies of complement C5a receptor antagonists PMX53 and PMX205 in mice. Scientific Reports. 8(1). 8101–8101. 25 indexed citations
20.
Coulthard, Liam G., et al.. (2018). Complement C3a receptor modulates embryonic neural progenitor cell proliferation and cognitive performance. Molecular Immunology. 101. 176–181. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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