John C. Reed

6.3k total citations · 5 hit papers
13 papers, 5.0k citations indexed

About

John C. Reed is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, John C. Reed has authored 13 papers receiving a total of 5.0k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Immunology. Recurrent topics in John C. Reed's work include Cell death mechanisms and regulation (8 papers), Mitochondrial Function and Pathology (4 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). John C. Reed is often cited by papers focused on Cell death mechanisms and regulation (8 papers), Mitochondrial Function and Pathology (4 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). John C. Reed collaborates with scholars based in United States, France and Australia. John C. Reed's co-authors include Shu-ichi Matsuzawa, Hong‐Gang Wang, Ulf R. Rapp, Shigemi Matsuyama, Qunli Xu, Jean Velours, Leszek Rychlewski, Adam Godzik, Lukasz Jaroszewski and Pierre H. Vachon and has published in prestigious journals such as Nature, Cell and Journal of Clinical Investigation.

In The Last Decade

John C. Reed

13 papers receiving 4.9k citations

Hit Papers

Double identity for proteins of the Bcl-2 family 1996 2026 2006 2016 1997 2000 1998 1996 2001 400 800 1.2k

Peers

John C. Reed
Comparison fields: 5 of 119
  • Molecular Biology 3.8k
  • Oncology 1.0k
  • Immunology 789
  • Cancer Research 637
  • Epidemiology 386
Replace Natalie Roy with:
Natalie Roy Canada
Ambereen Ali United States
Elizabeth A. Slee United Kingdom
Joan Gil Spain
Ana Maria Ibrado United States
Megan J. Robinson United States
John C. Reed United States
Abelardo López‐Rivas Spain
Jennifer Jockel United States
Megan M. McLaughlin United States
Natalie Roy Canada View profile →
Citations per field, relative to John C. Reed
John C. Reed · 1×
Citations per year, relative to John C. Reed
John C. Reed · 1×

Countries citing papers authored by John C. Reed

Since Specialization
Citations

This map shows the geographic impact of John C. Reed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John C. Reed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John C. Reed more than expected).

Fields of papers citing papers by John C. Reed

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John C. Reed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John C. Reed. The network helps show where John C. Reed may publish in the future.

Co-authorship network of co-authors of John C. Reed

This figure shows the co-authorship network connecting the top 25 collaborators of John C. Reed. A scholar is included among the top collaborators of John C. Reed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John C. Reed. John C. Reed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
# Work Indexed citations
1 52
2
Siah-1, SIP, and Ebi Collaborate in a Novel Pathway for β-Catenin Degradation Linked to p53 Responses breakdown →
521
3 34
4 182
5 30
6 11
7 3
8
Mechanisms of Apoptosis breakdown →
1034
9 20
10 244
11
Bcl-2 family proteins breakdown →
925
12
Double identity for proteins of the Bcl-2 family breakdown →
1289
13
Bcl-2 Targets the Protein Kinase Raf-1 to Mitochondria breakdown →
688

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026