Johannes Holtschmidt
- Cancer Research top 10%
- Genetics top 5%
- Oncology
- Pulmonary and Respiratory Medicine
- Pathology and Forensic Medicine
- Co-authors
- Sherko KümmelEllen HeitzerSibylle LoiblSabine Langer-FreitagJochen B. GeiglSabine RiethdorfMalgorzata StoupiecManfred Westphal
- Topics
- Breast Cancer Treatment Studies (16 papers)HER2/EGFR in Cancer Research (9 papers)Advanced Breast Cancer Therapies (7 papers)
- Cited by
- Cancer ResearchGeneticsOncology
- Partner nations
- GermanySwitzerlandItaly
In The Last Decade
Johannes Holtschmidt
23 papers receiving 448 citations
Peers
Comparison fields: 5 of 55
- Cancer Research 275
- Genetics 187
- Oncology 185
- Pulmonary and Respiratory Medicine 101
- Pathology and Forensic Medicine 91
Countries citing papers authored by Johannes Holtschmidt
This map shows the geographic impact of Johannes Holtschmidt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Johannes Holtschmidt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Johannes Holtschmidt more than expected).
Fields of papers citing papers by Johannes Holtschmidt
This network shows the impact of papers produced by Johannes Holtschmidt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Johannes Holtschmidt. The network helps show where Johannes Holtschmidt may publish in the future.
Co-authorship network of co-authors of Johannes Holtschmidt
This figure shows the co-authorship network connecting the top 25 collaborators of Johannes Holtschmidt. A scholar is included among the top collaborators of Johannes Holtschmidt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Johannes Holtschmidt. Johannes Holtschmidt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 1 | |
| 4 | 3 | |
| 5 | 1 | |
| 6 | 2 | |
| 7 | 2 | |
| 8 | 16 | |
| 9 | 21 | |
| 10 | 10 | |
| 11 | 26 | |
| 12 | 3 | |
| 13 | 14 | |
| 14 | 1 | |
| 15 | 8 | |
| 16 | 4 | |
| 17 | 13 | |
| 18 | 9 | |
| 19 | 257 | |
| 20 | [Etiology and therapy of strophulus]. | 3 |
About Johannes Holtschmidt
Johannes Holtschmidt is a scholar working on Cancer Research, Oncology and Pathology and Forensic Medicine, having authored 26 papers that have together received 456 indexed citations. Recurring topics across this work include Breast Cancer Treatment Studies (16 papers), HER2/EGFR in Cancer Research (9 papers) and Advanced Breast Cancer Therapies (7 papers). The work is most often cited by research in Cancer Research (275 citations), Genetics (187 citations) and Oncology (185 citations). Johannes Holtschmidt has collaborated with scholars based in Germany, Switzerland and Italy. Frequent co-authors include Sherko Kümmel, Ellen Heitzer, Sibylle Loibl, Sabine Langer-Freitag, Jochen B. Geigl, Sabine Riethdorf, Malgorzata Stoupiec, Manfred Westphal, Markus Glatzel and Alexander Schulte. Their work appears in journals such as The Lancet, Journal of Clinical Oncology and Clinical Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.