Joanne L. Lomas

578 total citations
11 papers, 486 citations indexed

About

Joanne L. Lomas is a scholar working on Molecular Biology, Epidemiology and Immunology. According to data from OpenAlex, Joanne L. Lomas has authored 11 papers receiving a total of 486 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Epidemiology and 5 papers in Immunology. Recurrent topics in Joanne L. Lomas's work include Cell death mechanisms and regulation (5 papers), Immune Response and Inflammation (4 papers) and Sepsis Diagnosis and Treatment (4 papers). Joanne L. Lomas is often cited by papers focused on Cell death mechanisms and regulation (5 papers), Immune Response and Inflammation (4 papers) and Sepsis Diagnosis and Treatment (4 papers). Joanne L. Lomas collaborates with scholars based in United States. Joanne L. Lomas's co-authors include Alfred Ayala, Chun‐Shiang Chung, Grace Y. Song, H. Hank Simms, Patricia S. Grutkoski, Irshad H. Chaudry, William G. Cioffi, Lesley Doughty, Brian W. LeBlanc and Jonathan S. Reichner and has published in prestigious journals such as American Journal Of Pathology, Journal of Leukocyte Biology and The International Journal of Biochemistry & Cell Biology.

In The Last Decade

Joanne L. Lomas

11 papers receiving 477 citations

Peers

Countries citing papers authored by Joanne L. Lomas

Since Specialization

Citations

This map shows the geographic impact of Joanne L. Lomas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joanne L. Lomas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joanne L. Lomas more than expected).

Fields of papers citing papers by Joanne L. Lomas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joanne L. Lomas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joanne L. Lomas. The network helps show where Joanne L. Lomas may publish in the future.

Co-authorship network of co-authors of Joanne L. Lomas

This figure shows the co-authorship network connecting the top 25 collaborators of Joanne L. Lomas. A scholar is included among the top collaborators of Joanne L. Lomas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joanne L. Lomas. Joanne L. Lomas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Fas-Ligand Mediated Apoptosis in Severe Sepsis and Shock 2003 ·Scandinavian Journal of Infectious Diseases ·Alfred Ayala, Joanne L. Lomas, Patricia S. Grutkoski, Chun‐Shiang Chung	24
2	Inhibition of CD1d activation suppresses septic mortality: a role for NK-T cells in septic immune dysfunction 2003 ·Journal of Surgical Research ·(unknown), (unknown), Joanne L. Lomas, (unknown), Laurent Brossay, William G. Cioffi, Alfred Ayala	34
3	Differential Effects of Macrophage Inflammatory Chemokine-2 and Keratinocyte-Derived Chemokine on Hemorrhage-Induced Neutrophil Priming for Lung Inflammation: Assessment by Adoptive Cells Transfer in Mice 2003 ·Shock ·Joanne L. Lomas, Chun‐Shiang Chung, Patricia S. Grutkoski, Brian W. LeBlanc, Liz M. Lavigne, Jonathan S. Reichner, Stephen H. Gregory, Lesley Doughty, William G. Cioffi, Alfred Ayala	63
4	Inhibition of Fas/Fas ligand signaling improves septic survival: differential effects on macrophage apoptotic and functional capacity 2003 ·Journal of Leukocyte Biology ·Chun‐Shiang Chung, Grace Y. Song, Joanne L. Lomas, H. Hank Simms, Irshad H. Chaudry, Alfred Ayala	113
5	NF-??B ACTIVATION HAS TISSUE-SPECIFIC EFFECTS ON IMMUNE CELL APOPTOSIS DURING POLYMICROBIAL SEPSIS 2002 ·Shock ·Amit Joshi, Chun‐Shiang Chung, Grace Y. Song, Joanne L. Lomas, (unknown), Alfred Ayala	20
6	Pathological aspects of apoptosis in severe sepsis and shock? 2002 ·The International Journal of Biochemistry & Cell Biology ·Alfred Ayala, Joanne L. Lomas, Patricia S. Grutkoski, Chun‐Shiang Chung	49
7	Shock-Induced Neutrophil Mediated Priming for Acute Lung Injury in Mice 2002 ·American Journal Of Pathology ·Alfred Ayala, Chun‐Shiang Chung, Joanne L. Lomas, Grace Y. Song, Lesley Doughty, Stephen H. Gregory, William G. Cioffi, Brian W. LeBlanc, Jonathan S. Reichner, H. Hank Simms, Patricia S. Grutkoski	129
8	dsRNA-ACTIVATED PROTEIN KINASE (PKR) MEDIATES VIRAL PRIMING OF LETHALITY IN POLYMICROBIAL SEPSIS, 2002 ·Shock ·(unknown), Chun‐Shiang Chung, (unknown), (unknown), Joanne L. Lomas, Alfred Ayala	1
9	Inhibition of Fas signaling prevents hepatic injury and improves organ blood flow during sepsis 2001 ·Surgery ·Chun‐Shiang Chung, Shaolong Yang, Grace Y. Song, Joanne L. Lomas, Ping Wang, H. Hank Simms, Irshad H. Chaudry, Alfred Ayala	47
10	A MOUSE MODEL OF PRIMING FOR ACUTE LUNG INJURY FOLLOWING SHOCK. 2001 ·Shock ·Alfred Ayala, Chun‐Shiang Chung, Joanne L. Lomas, (unknown), Lesley Doughty, H. Hank Simms	4
11	ROLE OF MIP-2 IN SUPPRESSION OF NEUTROPHIL APOPTOSIS. 2001 ·Shock ·Joanne L. Lomas, Chun‐Shiang Chung, Guisheng Song, (unknown), (unknown), H. Hank Simms, Alfred Ayala	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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