Jo C. Tsai

957 total citations
12 papers, 790 citations indexed

About

Jo C. Tsai is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Jo C. Tsai has authored 12 papers receiving a total of 790 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Oncology and 2 papers in Immunology. Recurrent topics in Jo C. Tsai's work include Protein Kinase Regulation and GTPase Signaling (4 papers), Angiogenesis and VEGF in Cancer (4 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Jo C. Tsai is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (4 papers), Angiogenesis and VEGF in Cancer (4 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Jo C. Tsai collaborates with scholars based in United States, Israel and France. Jo C. Tsai's co-authors include William C. Aird, Gary H. Perdew, William P. Long, Marilyn G. Pray-Grant, Robert Rosenberg, Katherine Spokes, M. Ruhul Abid, Kaikobad Irani, Frank W. Sellke and Mark J. Post and has published in prestigious journals such as Nature Medicine, Blood and Biochemistry.

In The Last Decade

Jo C. Tsai

12 papers receiving 766 citations

Peers

Jo C. Tsai

MB

CR

IMMUN

MICRO

HTM

Christelle Pigeon France

Zhesheng Wen China

Susan Kadner United States

June Giddings United Kingdom

Dikaia Xenaki Australia

Johan Lund Sweden

Yuan Gu United States

Yoshitaka Murata Japan

Karin Kilian Germany

Christelle Pigeon France

Jo C. Tsai

283 ×0.6

MB

123 ×0.6

CR

81 ×0.4

IMMUN

32 ×0.3

MICRO

45 ×0.5

HTM

Citations per year, relative to Jo C. Tsai Jo C. Tsai (= 1×) peers Christelle Pigeon

Countries citing papers authored by Jo C. Tsai

Since Specialization

Citations

This map shows the geographic impact of Jo C. Tsai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jo C. Tsai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jo C. Tsai more than expected).

Fields of papers citing papers by Jo C. Tsai

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jo C. Tsai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jo C. Tsai. The network helps show where Jo C. Tsai may publish in the future.

Co-authorship network of co-authors of Jo C. Tsai

This figure shows the co-authorship network connecting the top 25 collaborators of Jo C. Tsai. A scholar is included among the top collaborators of Jo C. Tsai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jo C. Tsai. Jo C. Tsai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

1.

Tsai, Jo C., Jie Zhang, Takashi Minami, et al.. (2002). Cloning and Characterization of the Human Lung Endothelial-Cell-Specific Molecule-1 Promoter. Journal of Vascular Research. 39(2). 148–159. 39 indexed citations

2.

Minami, Takashi, Diana Donovan, Jo C. Tsai, Robert Rosenberg, & William C. Aird. (2002). Differential regulation of the von Willebrand factor and Flt-1 promoters in the endothelium of hypoxanthine phosphoribosyltransferase–targeted mice. Blood. 100(12). 4019–4025. 28 indexed citations

3.

Abid, M. Ruhul, et al.. (2001). Vascular endothelial growth factor induces manganese‐superoxide dismutase expression in endothelial cells by a Racl‐regulated NADPH oxidase‐dependent mechanism. The FASEB Journal. 15(13). 2548–2550. 132 indexed citations

4.

Tsai, Jo C., Lixin Liu, Jie Zhang, et al.. (2001). Epidermal growth factor induces Egr-1 promoter activity in hepatocytes in vitro and in vivo. American Journal of Physiology-Gastrointestinal and Liver Physiology. 281(5). G1271–G1278. 17 indexed citations

5.

Li, Jian, Mark J. Post, Youhe Gao, et al.. (2000). PR39, a peptide regulator of angiogenesis. Nature Medicine. 6(1). 49–55. 303 indexed citations

6.

Tsai, Jo C., et al.. (2000). The egr‐1 promoter contains information for constitutive and inducible expression in transgenic mice. The FASEB Journal. 14(13). 1870–1872. 25 indexed citations

7.

Tsai, Jo C., Lixin Liu, Jiazhen Guan, & William C. Aird. (2000). The Egr-1 gene is induced by epidermal growth factor in ECV304 cells and primary endothelial cells. American Journal of Physiology-Cell Physiology. 279(5). C1414–C1424. 25 indexed citations

8.

Liu, Lixin, Jo C. Tsai, & William C. Aird. (2000). Egr-1 gene is induced by the systemic administration of the vascular endothelial growth factor and the epidermal growth factor. Blood. 96(5). 1772–1781. 3 indexed citations

9.

Liu, Lixin, Jo C. Tsai, & William C. Aird. (2000). Egr-1 gene is induced by the systemic administration of the vascular endothelial growth factor and the epidermal growth factor. Blood. 96(5). 1772–1781. 53 indexed citations

10.

Long, William P., Marilyn G. Pray-Grant, Jo C. Tsai, & Gary H. Perdew. (1998). Protein Kinase C Activity Is Required for Aryl Hydrocarbon Receptor Pathway-Mediated Signal Transduction. Molecular Pharmacology. 53(4). 691–700. 147 indexed citations

11.

Tsai, Jo C. & Gary H. Perdew. (1997). Ah Receptor Nuclear Translocator Protein Heterogeneity Is Altered after Heterodimerization with the Ah Receptor. Biochemistry. 36(29). 9066–9072. 16 indexed citations

12.

Tsai, Jo C.. (1997). Regulation of aryl hydrocarbon receptor nuclear translocator (ARNT) and AH receptor function. Purdue e-Pubs (Purdue University System). 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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