Jin-li Bai

401 total citations
27 papers, 289 citations indexed

About

Jin-li Bai is a scholar working on Genetics, Molecular Biology and Surgery. According to data from OpenAlex, Jin-li Bai has authored 27 papers receiving a total of 289 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Genetics, 21 papers in Molecular Biology and 8 papers in Surgery. Recurrent topics in Jin-li Bai's work include Neurogenetic and Muscular Disorders Research (21 papers), RNA modifications and cancer (20 papers) and RNA Research and Splicing (9 papers). Jin-li Bai is often cited by papers focused on Neurogenetic and Muscular Disorders Research (21 papers), RNA modifications and cancer (20 papers) and RNA Research and Splicing (9 papers). Jin-li Bai collaborates with scholars based in China and South Korea. Jin-li Bai's co-authors include Fang Song, Yu-jin Qu, Yuwei Jin, Hong Wang, Wenhui Zhang, Yanyu Lü, Sung Won Han, Seoung Bum Kim, Yan Li and Juan Du and has published in prestigious journals such as Human Molecular Genetics, Gene and Clinica Chimica Acta.

In The Last Decade

Jin-li Bai

27 papers receiving 287 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jin-li Bai China	10	223	217	107	23	11	27	289
S. K. Ng Hong Kong	8	28 0.1×	170 0.8×	92 0.9×	38 1.7×	16 1.5×	16	330
Javier Sanz Switzerland	7	29 0.1×	83 0.4×	17 0.2×	29 1.3×	4 0.4×	16	263
Damian Gola Germany	10	26 0.1×	93 0.4×	19 0.2×	113 4.9×	4 0.4×	14	268
Lisandra Vila Ellis United States	5	11 0.0×	130 0.6×	129 1.2×	7 0.3×	11 1.0×	8	285
Fuqing Sun China	8	13 0.1×	82 0.4×	69 0.6×	14 0.6×	7 0.6×	11	287
Pouria Mashouri Canada	6	16 0.1×	118 0.5×	15 0.1×	85 3.7×	5 0.5×	10	201
Panayiota Demosthenous Cyprus	6	20 0.1×	46 0.2×	14 0.1×	8 0.3×	4 0.4×	13	189
Lidia Ziółkowska Poland	11	18 0.1×	138 0.6×	48 0.4×	25 1.1×	8 0.7×	33	362
M. Yegitbasi Germany	3	78 0.3×	175 0.8×	64 0.6×	10 0.4×	1 0.1×	3	361
Brian C. Weiford United States	3	92 0.4×	126 0.6×	107 1.0×	9 0.4×		6	396

Countries citing papers authored by Jin-li Bai

Since Specialization

Citations

This map shows the geographic impact of Jin-li Bai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jin-li Bai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jin-li Bai more than expected).

Fields of papers citing papers by Jin-li Bai

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jin-li Bai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jin-li Bai. The network helps show where Jin-li Bai may publish in the future.

Co-authorship network of co-authors of Jin-li Bai

This figure shows the co-authorship network connecting the top 25 collaborators of Jin-li Bai. A scholar is included among the top collaborators of Jin-li Bai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jin-li Bai. Jin-li Bai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Qu, Yu-jin, Jin-li Bai, Hong Qi, et al.. (2024). Variants located in intron 6 of SMN1 lead to misdiagnosis in genetic detection and screening for SMA. Heliyon. 10(6). e28015–e28015. 1 indexed citations

Bai, Jin-li, Yu-jin Qu, Wen‐Chen Huang, et al.. (2023). A high-fidelity long-read sequencing-based approach enables accurate and effective genetic diagnosis of spinal muscular atrophy. Clinica Chimica Acta. 553. 117743–117743. 6 indexed citations

Bai, Jin-li, Yu-jin Qu, Jingjing Li, et al.. (2023). Novel Alu-mediated deletions of the SMN1 gene were identified by ultra-long read sequencing technology in patients with spinal muscular atrophy. Neuromuscular Disorders. 33(5). 382–390. 6 indexed citations

Bai, Jin-li, et al.. (2022). Paleogeographic and sedimentary evolution of Meso–Neoproterozoic strata in the Ordos Basin, western North China Craton. Journal of Petroleum Science and Engineering. 215. 110600–110600. 9 indexed citations

Cao, Yanyan, Yu-jin Qu, Jin-li Bai, et al.. (2021). Factors associated with delayed diagnosis of spinal muscular atrophy in China and changes in diagnostic delay. Neuromuscular Disorders. 31(6). 519–527. 8 indexed citations

Wang, Jia, Jin-li Bai, Hong Wang, et al.. (2021). Antisense oligonucleotides targeting the SMN2 promoter region enhance SMN2 expression in spinal muscular atrophy cell lines and mouse model. Human Molecular Genetics. 31(10). 1635–1650. 8 indexed citations

Bai, Jin-li, et al.. (2020). Dual Mechanism of a New SMN1 Variant (c.835G>C, p.Gly279Arg) by Interrupting Exon 7 Skipping and YG Oligomerization in Causation of Spinal Muscular Atrophy. Journal of Molecular Neuroscience. 71(1). 112–121. 1 indexed citations

Cao, Yanyan, Yu-jin Qu, Jin-li Bai, et al.. (2020). Transmission characteristics of SMN from 227 spinal muscular atrophy core families in China. Journal of Human Genetics. 65(5). 469–473. 2 indexed citations

Cao, Yanyan, Wenhui Zhang, Yu-jin Qu, et al.. (2018). Diagnosis of Spinal Muscular Atrophy. Chinese Medical Journal. 131(24). 2921–2929. 11 indexed citations

10.

Bai, Jin-li, Yu-jin Qu, Lan Yang, et al.. (2017). The SMN1 common variant c.22 dupA in Chinese patients causes spinal muscular atrophy by nonsense-mediated mRNA decay in humans. Gene. 644. 49–55. 11 indexed citations

11.

Qu, Yu-jin, Ge Lin, Jin-li Bai, et al.. (2017). p.Val19Glyfs*21 and p.Leu228* variants in the survival of motor neuron 1 trigger nonsense-mediated mRNA decay causing the SMN1 PTC+ transcripts degradation. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 806. 31–38. 4 indexed citations

12.

Bai, Jin-li, et al.. (2017). Mosaicism of Tetrasomy 18p. Chinese Medical Journal. 130(6). 744–746. 3 indexed citations

13.

Qu, Yu-jin, Jin-li Bai, Hong Wang, et al.. (2016). Mutation Spectrum of the Survival of Motor Neuron 1 and Functional Analysis of Variants in Chinese Spinal Muscular Atrophy. Journal of Molecular Diagnostics. 18(5). 741–752. 42 indexed citations

14.

Qu, Yu-jin, Yan Li, Jin-li Bai, et al.. (2016). Association between SMN2 methylation and disease severity in Chinese children with spinal muscular atrophy. Journal of Zhejiang University SCIENCE B. 17(1). 76–82. 16 indexed citations

15.

Qu, Yu-jin, Jin-li Bai, Wenhui Zhang, et al.. (2015). A rare variant (c.863G>T) in exon 7 of SMN1 disrupts mRNA splicing and is responsible for spinal muscular atrophy. European Journal of Human Genetics. 24(6). 864–870. 10 indexed citations

16.

Qu, Yu-jin, Jin-li Bai, Liwen Wang, et al.. (2014). Association of Copy Numbers of Survival Motor Neuron Gene 2 and Neuronal Apoptosis Inhibitory Protein Gene With the Natural History in a Chinese Spinal Muscular Atrophy Cohort. Journal of Child Neurology. 30(4). 429–436. 29 indexed citations

17.

Qu, Yu-jin, et al.. (2013). Correlation of PLS3 expression with disease severity in children with spinal muscular atrophy. Journal of Human Genetics. 59(1). 24–27. 33 indexed citations

18.

Qu, Yu-jin, Juan Du, Erzhen Li, et al.. (2012). Subtle mutations in the SMN1 gene in Chinese patients with SMA: p.Arg288Met mutation causing SMN1 transcript exclusion of exon7. BMC Medical Genetics. 13(1). 86–86. 23 indexed citations

19.

Bai, Jin-li, et al.. (2011). A novel missense mutation of the ubiquitin protein ligase E3A gene in a patient with Angelman syndrome.. PubMed. 124(1). 84–8. 8 indexed citations

20.

Du, Juan, et al.. (2011). [Mutation analysis of SMN1 gene in patients with spinal muscular atrophy].. PubMed. 49(6). 411–5. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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