Jing‐biao Wu

813 total citations
17 papers, 494 citations indexed

About

Jing‐biao Wu is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Jing‐biao Wu has authored 17 papers receiving a total of 494 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Cancer Research and 6 papers in Immunology. Recurrent topics in Jing‐biao Wu's work include RNA modifications and cancer (4 papers), Cancer-related molecular mechanisms research (4 papers) and Cancer, Lipids, and Metabolism (3 papers). Jing‐biao Wu is often cited by papers focused on RNA modifications and cancer (4 papers), Cancer-related molecular mechanisms research (4 papers) and Cancer, Lipids, and Metabolism (3 papers). Jing‐biao Wu collaborates with scholars based in China and Hong Kong. Jing‐biao Wu's co-authors include Xin‐hua Liang, Ya‐ling Tang, Shasha Wang, Xin Pang, Jia‐shun Wu, Ya‐Jie Tang, Min Zheng, Xianghua Yu, Mei Zhang and Xiao Yang and has published in prestigious journals such as PLoS ONE, Optics Letters and Frontiers in Microbiology.

In The Last Decade

Jing‐biao Wu

17 papers receiving 490 citations

Peers

Countries citing papers authored by Jing‐biao Wu

Since Specialization

Citations

This map shows the geographic impact of Jing‐biao Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jing‐biao Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jing‐biao Wu more than expected).

Fields of papers citing papers by Jing‐biao Wu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jing‐biao Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jing‐biao Wu. The network helps show where Jing‐biao Wu may publish in the future.

Co-authorship network of co-authors of Jing‐biao Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Jing‐biao Wu. A scholar is included among the top collaborators of Jing‐biao Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jing‐biao Wu. Jing‐biao Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

#	Work	Indexed citations
1	Integrative multi-omics analysis and experimental validation identify molecular subtypes, prognostic signature, and CA9 as a therapeutic target in oral squamous cell carcinoma 2025 ·Frontiers in Cell and Developmental Biology ·Yun Zhao, (unknown), Yamei Jiang, Jing‐biao Wu	1
2	Radiation Pattern Decoupled Patch Antennas Based on TM₀₁ and TM₁₁ Modes 2023 ·IEEE Antennas and Wireless Propagation Letters ·Jing‐biao Wu, (unknown), Nan Yang, Kwok Wa Leung, Kai Lu, Peng Fei Hu	10
3	OAM mode generation in helically twisted hollow-core antiresonant fiber 2023 ·Optics Letters ·Jiajing Tu, Jing‐biao Wu, (unknown), Jie Zhang, Shecheng Gao, Weiping Liu, Zhaohui Li	8
4	Artemisinin suppressed tumour growth and induced vascular normalisation in oral squamous cell carcinoma via inhibition of macrophage migration inhibitory factor 2022 ·Oral Diseases ·Xianghua Yu, Jing‐biao Wu, Hua‐yang Fan, (unknown), (unknown), (unknown), (unknown), (unknown), Xin Pang, Mei Zhang, Xin‐hua Liang, Ya‐ling Tang	7
5	Myeloid derived suppressor cells contribute to the malignant progression of oral squamous cell carcinoma 2020 ·PLoS ONE ·Xin Pang, Hua‐yang Fan, Ya‐ling Tang, Shasha Wang, Ming‐xin Cao, Haofan Wang, (unknown), Ke Wang, Xianghua Yu, Jing‐biao Wu, (unknown), (unknown), Xin‐hua Liang	52
6	Non-coding RNAs derailed: The many influences on the fatty acid reprogramming of cancer 2019 ·Life Sciences ·Xianghua Yu, Haofan Wang, Jing‐biao Wu, Shasha Wang, Ya‐Jie Tang, Ya‐ling Tang, Xin‐hua Liang	11
7	PRRX1 Regulates Cellular Phenotype Plasticity and Dormancy of Head and Neck Squamous Cell Carcinoma Through miR-642b-3p 2019 ·Neoplasia ·Jian Jiang, Min Zheng, Mei Zhang, Xiao Yang, Li Li, Shasha Wang, Jia‐shun Wu, Xianghua Yu, Jing‐biao Wu, Xin Pang, Ya‐Jie Tang, Ya‐ling Tang, Xin‐hua Liang	33
8	MIF promotes perineural invasion through EMT in salivary adenoid cystic carcinoma 2019 ·Molecular Carcinogenesis ·Mei Zhang, (unknown), Haofan Wang, Shasha Wang, Xianghua Yu, Jing‐biao Wu, Xin Pang, Jia‐shun Wu, Xiao Yang, Ya‐Jie Tang, (unknown), Li Li, Xin‐hua Liang, Min Zheng, Ya‐ling Tang	22
9	Cathepsin B defines leader cells during the collective invasion of salivary adenoid cystic carcinoma 2019 ·International Journal of Oncology ·Jia‐shun Wu, (unknown), Haofan Wang, Xianghua Yu, Xin Pang, Jing‐biao Wu, Shasha Wang, Mei Zhang, Xiao Yang, Ming‐xin Cao, Ya‐Jie Tang, Xin‐hua Liang, Min Zheng, Ya‐ling Tang	31
10	Hypoxia promotes vasculogenic mimicry formation by vascular endothelial growth factor A mediating epithelial‐mesenchymal transition in salivary adenoid cystic carcinoma 2019 ·Cell Proliferation ·Haofan Wang, Shasha Wang, Min Zheng, (unknown), Ke Wang, Xiaolei Gao, Ming‐xin Cao, Xianghua Yu, Xin Pang, Mei Zhang, Jing‐biao Wu, Jia‐shun Wu, Xiao Yang, Ya‐Jie Tang, Yu Chen, Ya‐ling Tang, Xin‐hua Liang	63
11	Macrophage migration inhibitory factor promotes the invasion and metastasis of oral squamous cell carcinoma through matrix metalloprotein‐2/9 2019 ·Molecular Carcinogenesis ·Shasha Wang, Min Zheng, Xin Pang, Mei Zhang, Xianghua Yu, Jing‐biao Wu, Xiaolei Gao, Jia‐shun Wu, Xiao Yang, Ya‐Jie Tang, Ya‐ling Tang, Xin‐hua Liang	20
12	PRRX1‐induced epithelial‐to‐mesenchymal transition in salivary adenoid cystic carcinoma activates the metabolic reprogramming of free fatty acids to promote invasion and metastasis 2019 ·Cell Proliferation ·Yaping Jiang, Ya‐ling Tang, Shasha Wang, Jia‐shun Wu, Mei Zhang, Xin Pang, Jing‐biao Wu, Yu Chen, Ya‐Jie Tang, Xin‐hua Liang	16
13	Overexpression Cathepsin D Contributes to Perineural Invasion of Salivary Adenoid Cystic Carcinoma 2018 ·Frontiers in Oncology ·Mei Zhang, Jia‐shun Wu, (unknown), Xin Pang, Li Li, Shasha Wang, Jing‐biao Wu, Ya‐Jie Tang, Xin‐hua Liang, Min Zheng, Ya‐ling Tang	15
14	Targeting VEGF pathway to normalize the vasculature: an emerging insight in cancer therapy 2018 ·OncoTargets and Therapy ·Jing‐biao Wu, Ya‐ling Tang, Xin‐hua Liang	84
15	Porphyromonas gingivalis Promotes 4-Nitroquinoline-1-Oxide-Induced Oral Carcinogenesis With an Alteration of Fatty Acid Metabolism 2018 ·Frontiers in Microbiology ·Jia‐shun Wu, Min Zheng, Mei Zhang, Xin Pang, Li Li, Shasha Wang, Xiao Yang, Jing‐biao Wu, Ya‐Jie Tang, (unknown), Ya‐ling Tang, Xin‐hua Liang	55
16	Autophagy is positively associated with the accumulation of myeloid‑derived suppressor cells in 4‑nitroquinoline‑1‑oxide‑induced oral cancer 2018 ·Oncology Reports ·Jia‐shun Wu, Li Li, Shasha Wang, Xin Pang, Jing‐biao Wu, (unknown), Ya‐Jie Tang, Ya‐ling Tang, Min Zheng, Xin‐hua Liang	25
17	LncRNAs as an intermediate in HPV16 promoting myeloid-derived suppressor cell recruitment of head and neck squamous cell carcinoma 2017 ·Oncotarget ·Xiangrui Ma, (unknown), Jing‐biao Wu, Yaping Jiang, Xiaolei Gao, Xiao Cen, Jia‐shun Wu, Shasha Wang, Ya‐Jie Tang, Ya‐ling Tang, Xin‐hua Liang	41

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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