Jinfeng Teng
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- Neuroscience and Neuropharmacology Research 11
- Sensory Systems top 2%
- Ion Channels and Receptors 9
- Biological Psychiatry top 10%
- Molecular Biology top 5%
- Ion channel regulation and function 14
- Nicotinic Acetylcholine Receptors Study 9
- Receptor Mechanisms and Signaling 5
- Lipid Membrane Structure and Behavior 4
- Structural Biology top 10%
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- Plant Stress Responses and Tolerance 4
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- Cardiac electrophysiology and arrhythmias 4
Jinfeng Teng
27 papers receiving 1.8k citations
Hit Papers
Peers
Comparison fields: 5 of 113
- Cellular and Molecular Neuroscience 589
- Sensory Systems 155
- Biological Psychiatry 50
- Molecular Biology 1.3k
- Structural Biology 22
Countries citing papers authored by Jinfeng Teng
This map shows the geographic impact of Jinfeng Teng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jinfeng Teng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jinfeng Teng more than expected).
Fields of papers citing papers by Jinfeng Teng
This network shows the impact of papers produced by Jinfeng Teng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jinfeng Teng. The network helps show where Jinfeng Teng may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Jinfeng Teng, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 7 | |
| 2 | 2025 | 7 | |
| 3 | 2024 | 3 | |
| 4 | 2024 | 13 | |
| 5 | 2023 | 6 | |
| 6 | 2023 | 41 | |
| 7 | 2022 | 46 | |
| 8 | 2022 | 58 | |
| 9 | 2022 | 43 | |
| 10 | 2020 | 238 | |
| 11 | 2020 | 142 | |
| 12 | 2019 | 135 | |
| 13 | Structure of a human synaptic GABAA receptorbreakdown → | 2018 | 394 |
| 14 | 2018 | 168 | |
| 15 | 2017 | 10 | |
| 16 | 2016 | 17 | |
| 17 | Mechanosensitive Ion Channels in Cardiovascular Physiology. | 2014 | 8 |
| 18 | 2014 | 135 | |
| 19 | 2013 | 11 | |
| 20 | 2007 | 16 |
About Jinfeng Teng
Jinfeng Teng is a scholar working on Sensory Systems, Cellular and Molecular Neuroscience and Molecular Biology, having authored 27 papers that have together received 1.9k indexed citations. Recurring topics across this work include Ion channel regulation and function (14 papers), Neuroscience and Neuropharmacology Research (11 papers), Nicotinic Acetylcholine Receptors Study (9 papers), Ion Channels and Receptors (9 papers), Receptor Mechanisms and Signaling (5 papers), Plant Stress Responses and Tolerance (4 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Lipid Membrane Structure and Behavior (4 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (589 citations), Sensory Systems (155 citations) and Biological Psychiatry (50 citations). Jinfeng Teng has collaborated with scholars based in United States, Sweden and Japan. Frequent co-authors include Ryan Hibbs, Colleen Noviello, Richard M. Walsh, Ching Kung, Stephen H. Loukin, Andriy Anishkin, Shaotong Zhu, Jeong Joo Kim, Anant Gharpure and Rebecca J. Howard. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences, Nature Communications, Microbiology and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.