Jim Talbot

729 citations
3 papers · 593 · 1 hit paper · h-index 2

Impact in

Papers in

Jim Talbot

2 papers receiving 576 citations

Hit Papers

The Duchenne muscular dystrophy gene product is localized in sarcolemma of human skeletal muscle 1988 · 568 citations
5680+12+25Years since publication100200300400500

Peers

Jim Talbot
Comparison fields: 5 of 62
  • Rehabilitation 79
  • Molecular Biology 553
  • Genetics 66
  • Biomaterials 79
  • Cardiology and Cardiovascular Medicine 137
Replace Chris A. Feener with:
Chris A. Feener United States
Keith Davison United Kingdom
J.Rafael M. Gorospe United States
L. Specht United States
G K Suthers Australia
J LEGER France
Christiana DelloRusso United States
Christian Pastoret France
Ora Fuchs Israel
Kowashi Yoshioka Japan
Jim Talbot relative to Chris A. Feener United States Chris A. Feener's profile →
Citations per field
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Chris A. Feener · 1×
Citations per year

Countries citing papers authored by Jim Talbot

Since Specialization
Citations

This map shows the geographic impact of Jim Talbot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jim Talbot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jim Talbot more than expected).

Fields of papers citing papers by Jim Talbot

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jim Talbot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jim Talbot. The network helps show where Jim Talbot may publish in the future.

Co-authors

The 11 scholars most cited alongside Jim Talbot, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jim Talbot Line = papers co-authored together Jim Talbot links everyone, so they are left out of the graph.

All Works

3 of 3 papers shown
#Work
1
The Duchenne muscular dystrophy gene product is localized in sarcolemma of human skeletal muscle
Hit paper breakdown →
1988568
2
Landmarks in linguistic thought II: The Western tradition in the twentieth century
200024
3 19851

About Jim Talbot

Jim Talbot is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Language and Linguistics, Cell Biology and Geography, Planning and Development, having authored 3 papers that have together received 593 indexed citations. Recurring topics across this work include Biotin and Related Studies (1 paper), Cardiomyopathy and Myosin Studies (1 paper), Muscle Physiology and Disorders (1 paper), Historical Linguistics and Language Studies (1 paper) and Geography Education and Pedagogy (1 paper). The work is most often cited by research in Rehabilitation (79 citations), Molecular Biology (553 citations), Genetics (66 citations), Biomaterials (79 citations) and Cardiology and Cardiovascular Medicine (137 citations). Jim Talbot has collaborated with scholars based in Canada and United States. Frequent co-authors include Peter N. Ray, Henry J. Klamut, Elizabeth E. Zubrzycka‐Gaarn, Robert S. Hodges, Ronald G. Worton, Dennis E. Bulman, George Karpati, Arthur H.M. Burghes, Nigel Love and John E. Joseph. Their work appears in journals such as Nature and Journal of Geological Education.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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