Jie Leng
- Immunology and Allergy top 0.5%
- Cell Adhesion Molecules Research 5
- Cell Biology top 1%
- Cellular Mechanics and Interactions 3
- Molecular Biology top 5%
- Protein Kinase Regulation and GTPase Signaling 4
- Angiogenesis and VEGF in Cancer 2
- Melanoma and MAPK Pathways 2
- Cancer Research top 5%
- Oncology top 10%
- HER2/EGFR in Cancer Research 1
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- Coagulation, Bradykinin, Polyphosphates, and Angioedema 1
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- Cardiomyopathy and Myosin Studies 1
- Co-authors
- David A. ChereshRichard KlemkeLeo PriceGary BokochMartin A. SchwartzBrian P. EliceiriRobert PaulPamela L. Schwartzberg
- Journals
- Journal of Biological Chemistry (1 paper)The Journal of Cell Biology (3 papers)Molecular Cell (1 paper)
- Partner nations
- United StatesSwitzerlandChina
In The Last Decade
Jie Leng
8 papers receiving 2.3k citations
Hit Papers
Peers
Comparison fields: 5 of 89
- Immunology and Allergy 898
- Cell Biology 911
- Molecular Biology 1.5k
- Cancer Research 309
- Oncology 390
Countries citing papers authored by Jie Leng
This map shows the geographic impact of Jie Leng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jie Leng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jie Leng more than expected).
Fields of papers citing papers by Jie Leng
This network shows the impact of papers produced by Jie Leng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jie Leng. The network helps show where Jie Leng may publish in the future.
Co-authorship network
The 20 scholars most cited alongside Jie Leng, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 21 | |
| 2 | 2008 | 71 | |
| 3 | 2000 | 151 | |
| 4 | Selective Requirement for Src Kinases during VEGF-Induced Angiogenesis and Vascular Permeabilitybreakdown → | 1999 | 646 |
| 5 | 1999 | 45 | |
| 6 | 1999 | 229 | |
| 7 | Activation of Rac and Cdc42 by Integrins Mediates Cell Spreadingbreakdown → | 1998 | 535 |
| 8 | CAS/Crk Coupling Serves as a “Molecular Switch” for Induction of Cell Migrationbreakdown → | 1998 | 596 |
About Jie Leng
Jie Leng is a scholar working on Immunology and Allergy, Cell Biology and Genetics, having authored 8 papers that have together received 2.3k indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (5 papers), Protein Kinase Regulation and GTPase Signaling (4 papers), Cellular Mechanics and Interactions (3 papers), Angiogenesis and VEGF in Cancer (2 papers), Melanoma and MAPK Pathways (2 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (1 paper), HER2/EGFR in Cancer Research (1 paper) and Cardiomyopathy and Myosin Studies (1 paper). The work is most often cited by research in Immunology and Allergy (898 citations), Cell Biology (911 citations) and Molecular Biology (1.5k citations). Jie Leng has collaborated with scholars based in United States, Switzerland and China. Frequent co-authors include David A. Cheresh, Richard Klemke, Leo Price, Gary Bokoch, Martin A. Schwartz, Brian P. Eliceiri, Robert Paul, Pamela L. Schwartzberg, John Hood and Peter C. Brooks. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Cell Biology and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.