Jiangjuan Shao

6.2k total citations · 3 hit papers
114 papers, 4.8k citations indexed

About

Jiangjuan Shao is a scholar working on Epidemiology, Molecular Biology and Hepatology. According to data from OpenAlex, Jiangjuan Shao has authored 114 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Epidemiology, 43 papers in Molecular Biology and 42 papers in Hepatology. Recurrent topics in Jiangjuan Shao's work include Liver Disease Diagnosis and Treatment (45 papers), Liver physiology and pathology (36 papers) and RNA modifications and cancer (13 papers). Jiangjuan Shao is often cited by papers focused on Liver Disease Diagnosis and Treatment (45 papers), Liver physiology and pathology (36 papers) and RNA modifications and cancer (13 papers). Jiangjuan Shao collaborates with scholars based in China, United States and Australia. Jiangjuan Shao's co-authors include Shizhong Zheng, Zili Zhang, Anping Chen, Feng Zhang, Huanhuan Jin, Shanzhong Tan, Li Wu, Anping Chen, Feixia Wang and Zhen Yao and has published in prestigious journals such as Journal of the American Chemical Society, SHILAP Revista de lepidopterología and Hepatology.

In The Last Decade

Jiangjuan Shao

110 papers receiving 4.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Activation of ferritinophagy is required for the RNA-binding protein ELAVL1/HuR to regulate ferroptosis in hepatic stellate cells

2018 374 citations Zili Zhang, Zhen Yao et al. profile →
RNA-binding protein ZFP36/TTP protects against ferroptosis by regulating autophagy signaling pathway in hepatic stellate cells

2019 345 citations Zili Zhang, Mei Guo et al. profile →
Canonical Wnt signaling promotes HSC glycolysis and liver fibrosis through an LDH-A/HIF-1α transcriptional complex

2023 75 citations Feixia Wang, Desong Kong et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jiangjuan Shao China	41	2.2k	1.6k	1.1k	1.1k	969	114	4.8k
Zili Zhang China	41	2.5k 1.1×	1.4k 0.9×	1.2k 1.1×	1.2k 1.1×	734 0.8×	132	5.1k
Jiao Feng China	40	3.0k 1.3×	1.0k 0.7×	1.8k 1.6×	511 0.5×	597 0.6×	153	5.6k
Tinghong Ye China	38	1.9k 0.9×	590 0.4×	445 0.4×	548 0.5×	410 0.4×	126	4.1k
Yingqun Zhou China	37	1.4k 0.6×	919 0.6×	615 0.5×	165 0.2×	675 0.7×	92	3.3k
Valentina M. Factor United States	55	5.1k 2.3×	1.2k 0.8×	2.2k 2.0×	475 0.4×	2.7k 2.8×	109	8.7k
Lei Zhao China	33	1.7k 0.8×	519 0.3×	895 0.8×	1.0k 1.0×	232 0.2×	120	3.9k
Ling Xu China	33	1.5k 0.7×	620 0.4×	759 0.7×	183 0.2×	414 0.4×	92	2.8k
Na Lu China	50	4.3k 1.9×	601 0.4×	1.4k 1.3×	725 0.7×	139 0.1×	199	7.7k
Hui‐Ling Chiou Taiwan	36	1.9k 0.8×	545 0.3×	701 0.6×	351 0.3×	147 0.2×	94	3.8k
Taotao Ma China	33	1.6k 0.7×	507 0.3×	691 0.6×	235 0.2×	351 0.4×	79	2.9k

Countries citing papers authored by Jiangjuan Shao

Since Specialization

Citations

This map shows the geographic impact of Jiangjuan Shao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jiangjuan Shao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jiangjuan Shao more than expected).

Fields of papers citing papers by Jiangjuan Shao

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jiangjuan Shao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jiangjuan Shao. The network helps show where Jiangjuan Shao may publish in the future.

Co-authorship network of co-authors of Jiangjuan Shao

This figure shows the co-authorship network connecting the top 25 collaborators of Jiangjuan Shao. A scholar is included among the top collaborators of Jiangjuan Shao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jiangjuan Shao. Jiangjuan Shao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Li, Mengran, et al.. (2025). Artemether relieves liver fibrosis by triggering ferroptosis in hepatic stellate cells via DHHC12-mediated S-palmitoylation of the BECN1 protein. Free Radical Biology and Medicine. 231. 120–135. 5 indexed citations

Chen, Xiaolei, et al.. (2024). Network Pharmacology, Molecular Docking Analysis and Molecular DynamicsSimulation of Scutellaria baicalensis in the Treatment of Liver Fibrosis. Current Pharmaceutical Design. 30(17). 1326–1340. 4 indexed citations

Tian, Haoyuan, Shuqi Wang, Feng Zhang, et al.. (2024). Oxidative stress induces ferroptosis in tendon stem cells by regulating mitophagy through cGAS-STING pathway. International Immunopharmacology. 138. 112652–112652. 13 indexed citations

Chen, Li, Siwei Xia, Feixia Wang, et al.. (2023). m6A methylation-induced NR1D1 ablation disrupts the HSC circadian clock and promotes hepatic fibrosis. Pharmacological Research. 189. 106704–106704. 18 indexed citations

Wang, Yingqian, Min Shen, Yujia Li, et al.. (2023). COVID‐19‐associated liver injury: Adding fuel to the flame. Cell Biochemistry and Function. 41(8). 1076–1092. 1 indexed citations

Wang, Yingqian, Yujia Li, Min Shen, et al.. (2023). Artesunate Induces Ferroptosis in Hepatic Stellate Cells and Alleviates Liver Fibrosis via the ROCK1/ATF3 Axis. Journal of Clinical and Translational Hepatology. 12(1). 36–51. 10 indexed citations

Sun, Sumin, Zhanghao Li, Jun Kai, et al.. (2022). Modification of lysine deacetylation regulates curcumol‐induced necroptosis through autophagy in hepatic stellate cells. Phytotherapy Research. 36(6). 2660–2676. 14 indexed citations

Sun, Sumin, Zhanghao Li, Ying Su, et al.. (2022). Curcumol alleviates liver fibrosis by inducing endoplasmic reticulum stress-mediated necroptosis of hepatic stellate cells through Sirt1/NICD pathway. PeerJ. 10. e13376–e13376. 28 indexed citations

Xia, Siwei, Zhimin Wang, Li Chen, et al.. (2021). Dihydroartemisinin regulates lipid droplet metabolism in hepatic stellate cells by inhibiting lncRNA-H19-induced AMPK signal. Biochemical Pharmacology. 192. 114730–114730. 17 indexed citations

10.

Su, Ying, Chun Jin, Zhanghao Li, et al.. (2021). Dihydroartemisinin Induces Ferroptosis in HCC by Promoting the Formation of PEBP1/15‐LO. Oxidative Medicine and Cellular Longevity. 2021(1). 3456725–3456725. 42 indexed citations

11.

Zhang, Zili, Xian Wang, Zilong Wang, et al.. (2021). Dihydroartemisinin alleviates hepatic fibrosis through inducing ferroptosis in hepatic stellate cells. BioFactors. 47(5). 801–818. 38 indexed citations

12.

Jia, Yan, Liyuan Gao, Yang Xiang, et al.. (2020). Blockade of periostin-dependent migration and adhesion by curcumol via inhibition of nuclear factor kappa B signaling in hepatic stellate cells. Toxicology. 440. 152475–152475. 14 indexed citations

13.

Jia, Yan, Huanhuan Jin, Liyuan Gao, et al.. (2020). A novel lncRNA PLK4 up‐regulated by talazoparib represses hepatocellular carcinoma progression by promoting YAP‐mediated cell senescence. Journal of Cellular and Molecular Medicine. 24(9). 5304–5316. 18 indexed citations

14.

Li, Mengmeng, Jiangjuan Shao, Zijian Guo, et al.. (2020). Novel mitochondrion‐targeting copper(II) complex induces HK2 malfunction and inhibits glycolysis via Drp1‐mediating mitophagy in HCC. Journal of Cellular and Molecular Medicine. 24(5). 3091–3107. 41 indexed citations

15.

Wang, Zhimin, Siwei Xia, Tian Zhang, et al.. (2020). LncRNA-H19 induces hepatic stellate cell activation via upregulating alcohol dehydrogenase III-mediated retinoic acid signals. International Immunopharmacology. 84. 106470–106470. 10 indexed citations

16.

Bian, Mianli, Jianlin He, Huanhuan Jin, et al.. (2019). Oroxylin A induces apoptosis of activated hepatic stellate cells through endoplasmic reticulum stress. APOPTOSIS. 24(11-12). 905–920. 25 indexed citations

17.

Shao, Jiangjuan, Mengmeng Li, Zijian Guo, et al.. (2019). TPP-related mitochondrial targeting copper (II) complex induces p53-dependent apoptosis in hepatoma cells through ROS-mediated activation of Drp1. Cell Communication and Signaling. 17(1). 149–149. 45 indexed citations

18.

Jin, Huanhuan, Yan Jia, Zhen Yao, et al.. (2017). Hepatic stellate cell interferes with NK cell regulation of fibrogenesis via curcumin induced senescence of hepatic stellate cell. Cellular Signalling. 33. 79–85. 47 indexed citations

19.

Jin, Huanhuan, Qianming Chen, Chunfeng Lu, et al.. (2016). Activation of PPARγ/P53 signaling is required for curcumin to induce hepatic stellate cell senescence. Cell Death and Disease. 7(4). e2189–e2189. 128 indexed citations

20.

Zhang, Feng, Li Chen, Huanhuan Jin, et al.. (2015). Activation of Fas death receptor pathway and Bid in hepatocytes is involved in saikosaponin D induction of hepatotoxicity. Environmental Toxicology and Pharmacology. 41. 8–13. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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