Jens Weise

3.2k total citations · 1 hit paper
42 papers, 2.1k citations indexed

About

Jens Weise is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Developmental Neuroscience. According to data from OpenAlex, Jens Weise has authored 42 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 15 papers in Cellular and Molecular Neuroscience and 12 papers in Developmental Neuroscience. Recurrent topics in Jens Weise's work include Neurogenesis and neuroplasticity mechanisms (12 papers), Nerve injury and regeneration (7 papers) and Prion Diseases and Protein Misfolding (6 papers). Jens Weise is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (12 papers), Nerve injury and regeneration (7 papers) and Prion Diseases and Protein Misfolding (6 papers). Jens Weise collaborates with scholars based in Germany, Denmark and United States. Jens Weise's co-authors include Mathias Bähr, Carsten O. Daub, Jürgen Kurths, Ralf Steuer, Joachim Selbig, Sönke Schwarting, Walter Schulz‐Schaeffer, Inga Zerr, Thorsten R. Doeppner and Olaf Crome and has published in prestigious journals such as Journal of Neuroscience, Bioinformatics and Stroke.

In The Last Decade

Jens Weise

42 papers receiving 2.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The mutual information: Detecting and evaluating dependencies betweenvariables

2002 565 citations Jens Weise et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jens Weise Germany	22	1.1k	455	431	291	169	42	2.1k
Nobuo Nagai Japan	27	683 0.6×	449 1.0×	289 0.7×	97 0.3×	222 1.3×	149	2.3k
Heike Siebert Germany	17	642 0.6×	301 0.7×	368 0.9×	150 0.5×	145 0.9×	45	1.3k
Zhandong Liu United States	37	2.6k 2.4×	371 0.8×	639 1.5×	89 0.3×	252 1.5×	121	4.7k
Mohamed Akil France	30	364 0.3×	573 1.3×	287 0.7×	70 0.2×	182 1.1×	114	3.4k
Gang Lin United States	15	1.2k 1.1×	399 0.9×	242 0.6×	672 2.3×	48 0.3×	30	2.4k
Shinji Fukui Japan	20	493 0.5×	172 0.4×	165 0.4×	105 0.4×	328 1.9×	129	1.5k
Jingwei Zheng China	27	517 0.5×	152 0.3×	379 0.9×	68 0.2×	424 2.5×	62	2.1k
Yasuhiro Watanabe Japan	31	2.2k 2.1×	853 1.9×	88 0.2×	127 0.4×	49 0.3×	185	4.2k
Guangyu Wu United States	41	4.2k 4.0×	1.1k 2.4×	179 0.4×	69 0.2×	122 0.7×	119	5.7k
Shahin Mohammadi United States	13	1.3k 1.2×	279 0.6×	793 1.8×	130 0.4×	123 0.7×	20	2.2k

Countries citing papers authored by Jens Weise

Since Specialization

Citations

This map shows the geographic impact of Jens Weise's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jens Weise with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jens Weise more than expected).

Fields of papers citing papers by Jens Weise

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jens Weise. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jens Weise. The network helps show where Jens Weise may publish in the future.

Co-authorship network of co-authors of Jens Weise

This figure shows the co-authorship network connecting the top 25 collaborators of Jens Weise. A scholar is included among the top collaborators of Jens Weise based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jens Weise. Jens Weise is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pötter, Stephan, et al.. (2023). Spatiotemporal model for the evolution of a mega-yardang system in the foreland of the Russian Altai. Aeolian Research. 61. 100866–100866. 3 indexed citations

Weise, Jens, et al.. (2021). Meeting Demands for Mass Customization: A Hybrid Organic Computing Approach. 2021 IEEE Symposium Series on Computational Intelligence (SSCI). 7326. 1–8. 2 indexed citations

Weise, Jens, et al.. (2020). MOSAIK: A Formal Model for Self-Organizing Manufacturing Systems. IEEE Pervasive Computing. 20(1). 9–18. 6 indexed citations

Doeppner, Thorsten R., Britta Kaltwasser, Jana Schlechter, et al.. (2015). Cellular prion protein promotes post-ischemic neuronal survival, angioneurogenesis and enhances neural progenitor cell homing via proteasome inhibition. Cell Death and Disease. 6(12). e2024–e2024. 42 indexed citations

Williams, Sarah K., Richard Fairless, Jens Weise, et al.. (2011). Neuroprotective Effects of the Cellular Prion Protein in Autoimmune Optic Neuritis. American Journal Of Pathology. 178(6). 2823–2831. 12 indexed citations

Doeppner, Thorsten R., et al.. (2010). Transplantation of TAT-Bcl-xL-transduced neural precursor cells: Long-term neuroprotection after stroke. Neurobiology of Disease. 40(1). 265–276. 32 indexed citations

Doeppner, Thorsten R., Gunnar P.H. Dietz, Jens Weise, & Mathias Bähr. (2010). Protection of hippocampal neurogenesis by TAT-Bcl-xL after cerebral ischemia in mice. Experimental Neurology. 223(2). 548–556. 12 indexed citations

Doeppner, Thorsten R., Gunnar P.H. Dietz, Joachim Gerber, et al.. (2009). TAT-Bcl-xL improves survival of neuronal precursor cells in the lesioned striatum after focal cerebral ischemia. Neurobiology of Disease. 34(1). 87–94. 32 indexed citations

Weise, Jens, Thorsten R. Doeppner, Arne Wrede, et al.. (2008). Overexpression of cellular prion protein alters postischemic Erk1/2 phosphorylation but not Akt phosphorylation and protects against focal cerebral ischemia. Restorative Neurology and Neuroscience. 26(1). 57–64. 35 indexed citations

10.

Crome, Olaf, Thorsten R. Doeppner, Sönke Schwarting, et al.. (2007). Enhanced poly(ADP‐ribose) polymerase‐1 activation contributes to recombinant tissue plasminogen activator‐induced aggravation of ischemic brain injury in vivo. Journal of Neuroscience Research. 85(8). 1734–1743. 11 indexed citations

11.

Weise, Jens, et al.. (2005). Expression time course and spatial distribution of activated caspase-3 after experimental status epilepticus: Contribution of delayed neuronal cell death to seizure-induced neuronal injury. Neurobiology of Disease. 18(3). 582–590. 66 indexed citations

12.

Kreutz, Michael R., Jens Weise, Daniela C. Dieterich, et al.. (2004). Rearrangement of the retino‐collicular projection after partial optic nerve crush in the adult rat. European Journal of Neuroscience. 19(2). 247–257. 18 indexed citations

13.

Weise, Jens, et al.. (2003). Detection of acute thalamo-mesencephalic infarction: diffusion abnormality precedes T2 hyperintensity. Acta Neurologica Scandinavica. 108(1). 52–54. 1 indexed citations

14.

Küker, Wilhelm, Jens Weise, Hilmar Krapf, et al.. (2002). MRI characteristics of acute and subacute brainstem and thalamic infarctions: value of T2- and diffusion-weighted sequences. Journal of Neurology. 249(1). 33–42. 55 indexed citations

15.

Weise, Jens, Stefan Isenmann, & Mathias Bähr. (2001). Increased expression and activation of poly(ADP-ribose) polymerase (PARP) contribute to retinal ganglion cell death following rat optic nerve transection. Cell Death and Differentiation. 8(8). 801–807. 41 indexed citations

16.

Weise, Jens, Stefan Isenmann, Nikolaj Klöcker, et al.. (2000). Adenovirus-Mediated Expression of Ciliary Neurotrophic Factor (CNTF) Rescues Axotomized Rat Retinal Ganglion Cells But Does Not Support Axonal Regeneration in Vivo. Neurobiology of Disease. 7(3). 212–223. 101 indexed citations

17.

Weise, Jens, et al.. (2000). Degeneration and regeneration of ganglion cell axons. Microscopy Research and Technique. 48(2). 55–62. 11 indexed citations

18.

Kreutz, Michael R., Tobias M. Böckers, Jürgen Bockmann, et al.. (1998). Axonal Injury Alters Alternative Splicing of the Retinal NR1 Receptor: the Preferential Expression of the NR1b Isoforms Is Crucial for Retinal Ganglion Cell Survival. Journal of Neuroscience. 18(20). 8278–8291. 53 indexed citations

19.

Speliotes, Elizabeth K., et al.. (1996). Increased expression of basic fibroblast growth factor (bFGF) following focal cerebral infarction in the rat. Molecular Brain Research. 39(1-2). 31–42. 70 indexed citations

20.

Henshaw, Ross, Jens Weise, Vladimir S. Trubetskoy, et al.. (1995). Basic Fibroblast Growth Factor Protects against Excitotoxicity and Chemical Hypoxia in Both Neonatal and Adult Rats. Journal of Cerebral Blood Flow & Metabolism. 15(4). 619–623. 81 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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