Jens E. V. Petersen

1.5k total citations · 1 hit paper
13 papers, 872 citations indexed

About

Jens E. V. Petersen is a scholar working on Public Health, Environmental and Occupational Health, Hematology and Immunology. According to data from OpenAlex, Jens E. V. Petersen has authored 13 papers receiving a total of 872 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Public Health, Environmental and Occupational Health, 4 papers in Hematology and 4 papers in Immunology. Recurrent topics in Jens E. V. Petersen's work include Malaria Research and Control (10 papers), Mosquito-borne diseases and control (7 papers) and Blood Coagulation and Thrombosis Mechanisms (4 papers). Jens E. V. Petersen is often cited by papers focused on Malaria Research and Control (10 papers), Mosquito-borne diseases and control (7 papers) and Blood Coagulation and Thrombosis Mechanisms (4 papers). Jens E. V. Petersen collaborates with scholars based in Denmark, United States and Tanzania. Jens E. V. Petersen's co-authors include Thomas Lavstsen, Louise Turner, Christian W. Wang, Thor G. Theander, John Lusingu, Line Jee Hartmann Rasmussen, Jesper Eugen‐Olsen, Jakob S. Jespersen, Matthew K. Higgins and Morten A. Nielsen and has published in prestigious journals such as Nature, Blood and PLoS ONE.

In The Last Decade

Jens E. V. Petersen

12 papers receiving 864 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Severe malaria is associated with parasite binding to endothelial protein C receptor

2013 412 citations Louise Turner, Thomas Lavstsen et al. Nature profile →

Peers

Jens E. V. Petersen

PHEOH

IMMUN

HEMAT

ONCOL

Pascal Pointaire Switzerland

Sarah J. Higgins United States

Valentina Barrera United Kingdom

Kejia Kan Germany

Kannan Venugopal Australia

Samuel Kampondeni Malawi

Vinicius Albuquerque Sortica Brazil

Lolita Bebris United States

Ângela C. Crespo United States

Enrica Alteri Switzerland

Pascal Pointaire Switzerland

Jens E. V. Petersen

482 ×0.8

PHEOH

457 ×1.3

IMMUN

131 ×1.2

31 ×0.4

HEMAT

68 ×0.9

ONCOL

Citations per year, relative to Jens E. V. Petersen Jens E. V. Petersen (= 1×) peers Pascal Pointaire

Countries citing papers authored by Jens E. V. Petersen

Since Specialization

Citations

This map shows the geographic impact of Jens E. V. Petersen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jens E. V. Petersen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jens E. V. Petersen more than expected).

Fields of papers citing papers by Jens E. V. Petersen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jens E. V. Petersen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jens E. V. Petersen. The network helps show where Jens E. V. Petersen may publish in the future.

Co-authorship network of co-authors of Jens E. V. Petersen

This figure shows the co-authorship network connecting the top 25 collaborators of Jens E. V. Petersen. A scholar is included among the top collaborators of Jens E. V. Petersen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jens E. V. Petersen. Jens E. V. Petersen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Markwalter, Christine F., Jens E. V. Petersen, Kelsey M. Sumner, et al.. (2023). Symptomatic malaria enhances protection from reinfection with homologous Plasmodium falciparum parasites. PLoS Pathogens. 19(6). e1011442–e1011442. 1 indexed citations

Saelens, Joseph W., Jens E. V. Petersen, Elizabeth Freedman, et al.. (2021). Impact of Sickle Cell Trait Hemoglobin on the Intraerythrocytic Transcriptional Program of Plasmodium falciparum. mSphere. 6(5). e0075521–e0075521. 4 indexed citations

Petersen, Jens E. V., Joseph W. Saelens, Elizabeth Freedman, et al.. (2021). Sickle-trait hemoglobin reduces adhesion to both CD36 and EPCR by Plasmodium falciparum-infected erythrocytes. PLoS Pathogens. 17(6). e1009659–e1009659. 8 indexed citations

Rasmussen, Line Jee Hartmann, Jens E. V. Petersen, & Jesper Eugen‐Olsen. (2021). Soluble Urokinase Plasminogen Activator Receptor (suPAR) as a Biomarker of Systemic Chronic Inflammation. Frontiers in Immunology. 12. 780641–780641. 140 indexed citations

Petersen, Jens E. V., Thomas Kallemose, Karen D. Barton, Avshalom Caspi, & Line Jee Hartmann Rasmussen. (2020). Soluble urokinase plasminogen activator receptor (suPAR) as a prognostic marker of mortality in healthy, general and patient populations: protocol for a systematic review and meta-analysis. BMJ Open. 10(7). e036125–e036125. 7 indexed citations

Petersen, Jens E. V., Alphaxard Manjurano, Reginald A. Kavishe, et al.. (2016). Plasma Ang2 and ADAM17 levels are elevated during clinical malaria; Ang2 level correlates with severity and expression of EPCR-binding PfEMP1. Scientific Reports. 6(1). 35950–35950. 12 indexed citations

Jespersen, Jakob S., Christian W. Wang, Bent Petersen, et al.. (2016). Plasmodium falciparum var genes expressed in children with severe malaria encode CIDR α1 domains. EMBO Molecular Medicine. 8(8). 839–850. 64 indexed citations

Subramani, Ramesh, Casper Hempel, Jens E. V. Petersen, et al.. (2015). Plasmodium falciparum-Infected Erythrocyte Knob Density Is Linked to the PfEMP1 Variant Expressed. mBio. 6(5). e01456–15. 24 indexed citations

Tamayo, Ibai, Louise Turner, Christian W. Wang, et al.. (2015). Protein C system defects inflicted by the malaria parasite protein PfEMP1 can be overcome by a soluble EPCR variant. Thrombosis and Haemostasis. 114(11). 1038–1048. 40 indexed citations

10.

Lau, Clinton K., Louise Turner, Jakob S. Jespersen, et al.. (2014). Structural Conservation Despite Huge Sequence Diversity Allows EPCR Binding by the PfEMP1 Family Implicated in Severe Childhood Malaria. Cell Host & Microbe. 17(1). 118–129. 130 indexed citations

11.

Petersen, Jens E. V., Eveline A. M. Bouwens, Ibai Tamayo, et al.. (2014). Disruption of EPCR-Dependent Functions By Plasmodium Falciparum Erythrocyte Membrane Protein 1 (PfEMP1) Can be Rescued By a Soluble EPCR Variant. Blood. 124(21). 577–577. 1 indexed citations

12.

Berger, Sanne Schou, Louise Turner, Christian W. Wang, et al.. (2013). Plasmodium falciparum Expressing Domain Cassette 5 Type PfEMP1 (DC5-PfEMP1) Bind PECAM1. PLoS ONE. 8(7). e69117–e69117. 29 indexed citations

13.

Turner, Louise, Thomas Lavstsen, Sanne Schou Berger, et al.. (2013). Severe malaria is associated with parasite binding to endothelial protein C receptor. Nature. 498(7455). 502–505. 412 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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