Jeffrey L. Barr
- Cellular and Molecular Neuroscience top 5%
- Molecular Biology
- Behavioral Neuroscience top 5%
- Cognitive Neuroscience
- Epidemiology
- Co-authors
- Gina L. ForsterEllen M. UnterwaldKenneth J. RennerG. Cristina BrailoiuEugen BrailoiuDaniel R. DaviesMary E. AboodPasquale Manzerra
- Topics
- Neuroscience and Neuropharmacology Research (13 papers)Receptor Mechanisms and Signaling (8 papers)Neurotransmitter Receptor Influence on Behavior (8 papers)
- Partner nations
- United StatesCanadaUnited Kingdom
In The Last Decade
Jeffrey L. Barr
28 papers receiving 552 citations
Peers
Comparison fields: 5 of 72
- Cellular and Molecular Neuroscience 277
- Molecular Biology 203
- Behavioral Neuroscience 83
- Cognitive Neuroscience 82
- Epidemiology 64
Countries citing papers authored by Jeffrey L. Barr
This map shows the geographic impact of Jeffrey L. Barr's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jeffrey L. Barr with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jeffrey L. Barr more than expected).
Fields of papers citing papers by Jeffrey L. Barr
This network shows the impact of papers produced by Jeffrey L. Barr. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jeffrey L. Barr. The network helps show where Jeffrey L. Barr may publish in the future.
Co-authorship network of co-authors of Jeffrey L. Barr
This figure shows the co-authorship network connecting the top 25 collaborators of Jeffrey L. Barr. A scholar is included among the top collaborators of Jeffrey L. Barr based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jeffrey L. Barr. Jeffrey L. Barr is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 9 | |
| 3 | 0 | |
| 4 | 13 | |
| 5 | 16 | |
| 6 | 5 | |
| 7 | 20 | |
| 8 | 56 | |
| 9 | 16 | |
| 10 | 8 | |
| 11 | 13 | |
| 12 | 20 | |
| 13 | 9 | |
| 14 | 39 | |
| 15 | 46 | |
| 16 | 72 | |
| 17 | 18 | |
| 18 | 29 | |
| 19 | 51 | |
| 20 | 14 |
About Jeffrey L. Barr
Jeffrey L. Barr is a scholar working on Cellular and Molecular Neuroscience, Behavioral Neuroscience and Neurology, having authored 29 papers that have together received 556 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (13 papers), Receptor Mechanisms and Signaling (8 papers) and Neurotransmitter Receptor Influence on Behavior (8 papers). The work is most often cited by research in Behavioral Neuroscience (83 citations), Biological Psychiatry (46 citations) and Cellular and Molecular Neuroscience (277 citations). Jeffrey L. Barr has collaborated with scholars based in United States, Canada and United Kingdom. Frequent co-authors include Gina L. Forster, Ellen M. Unterwald, Kenneth J. Renner, G. Cristina Brailoiu, Eugen Brailoiu, Daniel R. Davies, Mary E. Abood, Pasquale Manzerra, Jamie L. Scholl and Pingwei Zhao. Their work appears in journals such as PLoS ONE, International Journal of Molecular Sciences and Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.