Jeffrey J. Ackroyd

1.5k total citations
12 papers, 269 citations indexed

About

Jeffrey J. Ackroyd is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Jeffrey J. Ackroyd has authored 12 papers receiving a total of 269 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Oncology and 4 papers in Cancer Research. Recurrent topics in Jeffrey J. Ackroyd's work include DNA Repair Mechanisms (3 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Signaling Pathways in Disease (2 papers). Jeffrey J. Ackroyd is often cited by papers focused on DNA Repair Mechanisms (3 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Signaling Pathways in Disease (2 papers). Jeffrey J. Ackroyd collaborates with scholars based in United States, Russia and Australia. Jeffrey J. Ackroyd's co-authors include Donna S. Shewach, Pingping Hou, Xingdi Ma, Zhengdao Lan, Raghu Kalluri, Chang‐Jiun Wu, Qiang Zhang, Ronald A. DePinho, Joseph A. Wawrzyniak and Mikhail A. Nikiforov and has published in prestigious journals such as SHILAP Revista de lepidopterología, Cancer Research and Oncogene.

In The Last Decade

Jeffrey J. Ackroyd

11 papers receiving 266 citations

Peers

Jeffrey J. Ackroyd

ONCOL

IMMUN

PRM

Kristen S. Hill United States

Aurora Chinnici Italy

Jane Ho Chun Loong China

Jakub Faktor Czechia

Ambikai Gajan United States

Annie Wai Yeeng Chai Malaysia

Meng Lou China

Joël P. Charles Germany

Enilze M.S.F. Ribeiro Brazil

Bryan C. Nikolai United States

Kristen S. Hill United States

Jeffrey J. Ackroyd

228 ×1.3

110 ×1.1

ONCOL

68 ×0.9

58 ×1.3

IMMUN

30 ×1.2

PRM

Citations per year, relative to Jeffrey J. Ackroyd Jeffrey J. Ackroyd (= 1×) peers Kristen S. Hill

Countries citing papers authored by Jeffrey J. Ackroyd

Since Specialization

Citations

This map shows the geographic impact of Jeffrey J. Ackroyd's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jeffrey J. Ackroyd with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jeffrey J. Ackroyd more than expected).

Fields of papers citing papers by Jeffrey J. Ackroyd

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jeffrey J. Ackroyd. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jeffrey J. Ackroyd. The network helps show where Jeffrey J. Ackroyd may publish in the future.

Co-authorship network of co-authors of Jeffrey J. Ackroyd

This figure shows the co-authorship network connecting the top 25 collaborators of Jeffrey J. Ackroyd. A scholar is included among the top collaborators of Jeffrey J. Ackroyd based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jeffrey J. Ackroyd. Jeffrey J. Ackroyd is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Han, Jenny, Jeffrey J. Ackroyd, Jonathan S. Oakhill, et al.. (2022). Systemic Ablation of Camkk2 Impairs Metastatic Colonization and Improves Insulin Sensitivity in TRAMP Mice: Evidence for Cancer Cell-Extrinsic CAMKK2 Functions in Prostate Cancer. Cells. 11(12). 1890–1890. 7 indexed citations

Barekatain, Yasaman, Jeffrey J. Ackroyd, Victoria C. Yan, et al.. (2022). Abstract 2395: Homozygous MTAP deletion in primary human glioblastoma is not associated with elevation of methylthioadenosine. Cancer Research. 82(12_Supplement). 2395–2395.

Khadka, Sunada, Yasaman Barekatain, Jeffrey J. Ackroyd, et al.. (2021). Impaired anaplerosis is a major contributor to glycolysis inhibitor toxicity in glioma. SHILAP Revista de lepidopterología. 9(1). 27–27. 13 indexed citations

Hou, Pingping, Avnish Kapoor, Qiang Zhang, et al.. (2020). Tumor Microenvironment Remodeling Enables Bypass of Oncogenic KRAS Dependency in Pancreatic Cancer. Cancer Discovery. 10(7). 1058–1077. 99 indexed citations

Barekatain, Yasaman, Victoria C. Yan, Jeffrey J. Ackroyd, et al.. (2020). Robust detection of oncometabolic aberrations by 1H–13C heteronuclear single quantum correlation in intact biological specimens. Communications Biology. 3(1). 328–328. 2 indexed citations

Gammon, Seth T., Federica Pisaneschi, Madhavi Bandi, et al.. (2019). Mechanism-Specific Pharmacodynamics of a Novel Complex-I Inhibitor Quantified by Imaging Reversal of Consumptive Hypoxia with [18F]FAZA PET In Vivo. Cells. 8(12). 1487–1487. 20 indexed citations

Im, Michael M., et al.. (2016). Late DNA Damage Mediated by Homologous Recombination Repair Results in Radiosensitization with Gemcitabine. Radiation Research. 186(5). 466–477. 5 indexed citations

Bianchi, Anna, Archis Bagati, Emily E. Fink, et al.. (2016). Microphthalmia-associated transcription factor suppresses invasion by reducing intracellular GTP pools. Oncogene. 36(1). 84–96. 27 indexed citations

Im, Michael M., et al.. (2015). Drug Metabolism and Homologous Recombination Repair in Radiosensitization with Gemcitabine. Radiation Research. 183(1). 114–123. 7 indexed citations

10.

Bianchi, Anna, Joseph A. Wawrzyniak, Archis Bagati, et al.. (2015). Pharmacological targeting of guanosine monophosphate synthase suppresses melanoma cell invasion and tumorigenicity. Cell Death and Differentiation. 22(11). 1858–1864. 35 indexed citations

11.

Wawrzyniak, Joseph A., Anna Bianchi, Wiam Bshara, et al.. (2013). A Purine Nucleotide Biosynthesis Enzyme Guanosine Monophosphate Reductase Is a Suppressor of Melanoma Invasion. Cell Reports. 5(2). 493–507. 41 indexed citations

12.

Ladd, Brendon, et al.. (2013). Inhibition of homologous recombination with vorinostat synergistically enhances ganciclovir cytotoxicity. DNA repair. 12(12). 1114–1121. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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